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Clinical Trials/NCT04914806
NCT04914806
Recruiting
Not Applicable

Association Between Premature Birth and Its Comorbidities With NO and the HPG Activation at Minipuberty

National and Kapodistrian University of Athens3 sites in 3 countries240 target enrollmentJune 1, 2021

Overview

Phase
Not Applicable
Intervention
Not specified
Conditions
Prematurity
Sponsor
National and Kapodistrian University of Athens
Enrollment
240
Locations
3
Primary Endpoint
Gonadotrophin levels
Status
Recruiting
Last Updated
3 years ago

Overview

Brief Summary

Case-control study of inhaled Nitric Oxide (iNO) treatment of full-term and preterm infants. The main objective of this study is to investigate the association between premature birth and its later comorbidities (neuroendocrine, metabolic, cognitive, etc) with iNO treatment and the maturation of the HPG axis during minipuberty.

Detailed Description

In preterm infants, inhaled NO (iNO) is routinely used to treat respiratory failure and pulmonary hypertension, while preclinical studies have shown that it markedly increases NO concentrations in the brain. Animal and human studies have shown that NO deficiency may jeopardize the establishment of a mature and functional HPG axis whereas it is also associated with a series of comorbidities affecting the overall brain development (e.g. sensory, fertility and cognitive functions). Prematurity has been associated with a series of non-communicable diseases of major importance in public health, including neurodevelopmental impairments, metabolic abnormalities (e.g. obesity, type 2 diabetes mellitus, impaired glucose tolerance) and cardiovascular disease. This study aims to evaluate the associations between altered minipuberty in preterm infants and the later development of multi-comorbidities (mental and non-mental disorders), and identify the possible implication of the NO pathway as a causative mechanism. Specific objectives : 1. Assess the efficiency of NO replenishment therapy (i.e. role of NO pathway) as a therapeutic against alterations of minipuberty resulting from preterm birth. 2. Assess the efficiency of NO replenishment therapy (i.e. role of NO pathway) as a therapeutic against the multi-comorbidities (mental and non-mental disorders) related to altered minipuberty as a result of preterm birth.

Registry
clinicaltrials.gov
Start Date
June 1, 2021
End Date
December 31, 2025
Last Updated
3 years ago
Study Type
Observational
Sex
All

Investigators

Responsible Party
Principal Investigator
Principal Investigator

Soultana (Tania) Siahanidou

Professor

National and Kapodistrian University of Athens

Eligibility Criteria

Inclusion Criteria

  • Preterm (GA\<37 weeks) or full-term (≥37 weeks of gestation) infants
  • Requiring respiratory support
  • Admitted to Neonatal Care Unit

Exclusion Criteria

  • Severe congenital anomalies
  • Suspected major chromosomal anomalies
  • Infants judged by the physician as nonviable

Outcomes

Primary Outcomes

Gonadotrophin levels

Time Frame: up to 9 months of age

Serial blood levels of gonadotrophins (FSH and LH)

Bayleys scale score

Time Frame: at 9 months of age

The Bayley Scales of Infant and Toddler Development will be used to assess neurodevelopment. Values \<85 indicate developmental delay.

Body fat and fat-free mass

Time Frame: up to 9 months of age

Serial fat and fat-free mass measurements by air displacement plethysmography

HOMA-IR

Time Frame: up to 9 months

Serial measurements of blood glucose/insulin (HOMA-IR)

Secondary Outcomes

  • Otoacoustic emissions(up to 9 months)
  • Fundoscopy(up to 9 months)
  • gusto-facial reflex(at 3 and 9 months of age)
  • Leptin levels(up to 9 months)

Study Sites (3)

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