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Clinical Trials/NCT02236247
NCT02236247
Unknown
Phase 1

Heart Rate Control as an Additional Therapeutic Strategy in Patients With Decompensated Heart Failure: a Prospective, Randomized, Double-blinded, Placebo-controlled Study.

University of Sao Paulo1 site in 1 country50 target enrollmentMay 2013

Overview

Phase
Phase 1
Intervention
ivabradine
Conditions
Decompensated Heart Failure
Sponsor
University of Sao Paulo
Enrollment
50
Locations
1
Primary Endpoint
Change from baseline heart rate
Last Updated
9 years ago

Overview

Brief Summary

Study aims to compare the I(f) inhibitor ivabradine with placebo as strategy of heart rate control in patients with decompensated heart failure (DHF).

Detailed Description

Sympathetic hyperactivity and consequent increase in heart rate (HR) are physiological responses to low cardiac output in patients with decompensated heart failure (DHF). However, elevated HR may become inappropriate in these patients, increasing myocardial oxygen demand and decreasing diastolic filling time and might lead to hemodynamic deterioration, ventricular dysfunction (tachycardiomyopathy) and clinical deterioration. Studies show the elevated HR is a predictor of poor prognosis in DHF. Subanalyses of large clinical trials using beta blockers (BBs) demonstrate the adequate control of HR correlates with a better outcome in patients with stable chronic heart failure (HF). However, use of BBs in patients with DHF is limited due to negative inotropic and hypotensive effects of these drugs. As alternative to BBs, ivabradine has shown to increase survival of patients with chronic stable systolic HF. Compared to BBs, ivabradine has the advantage of "pure" negative chronotropic effect, no effect on myocardial contractility or peripheral vascular resistance. Despite the inhibition of I (f) has been validated as a therapeutic option in patients with stable HF, there are no studies available on this strategy in patients with DHF. We hypothesized that HR control by ivabradine might improve clinical, hemodynamic and neurohormonal parameters in patients with DHF. The aim of this study was to evaluate the efficacy of HR control with ivabradine in patients with DHF.

Registry
clinicaltrials.gov
Start Date
May 2013
End Date
August 2017
Last Updated
9 years ago
Study Type
Interventional
Study Design
Parallel
Sex
All

Investigators

Responsible Party
Principal Investigator
Principal Investigator

Marco Stephan Lofrano Alves

MD

University of Sao Paulo

Eligibility Criteria

Inclusion Criteria

  • Sinus node rhythm
  • HR\> 80 bpm
  • Hospitalization for DHF
  • Ejection fraction ≤ 40%
  • Sign informed consent

Exclusion Criteria

  • Systolic blood pressure \<85 mmHg
  • Signs of hypoperfusion
  • Dobutamine\>15 mcg/Kg/min
  • Acute myocarditis
  • Primary valvular disease requiring surgery
  • Stroke in the last three months
  • Hypertrophic or restrictive cardiomyopathy
  • Sinus node disease
  • Atrial fibrillation or flutter
  • Second or third degree atrio-ventricular blockade

Arms & Interventions

ivabradine

I(f) inhibitor, heart rate controller

Intervention: ivabradine

placebo

placebo pill will be administered orally twice daily

Intervention: Placebo

Outcomes

Primary Outcomes

Change from baseline heart rate

Time Frame: Baseline, day 5 after intervention

Heart rate will be assessed at morning, after 30 minutes of rest, recorded by electrocardiogram.

Secondary Outcomes

  • Change from baseline blood pressure(Baseline, day 5 after intervention)
  • Change from baseline ejection fraction(Baseline, day 5 after intervention)
  • Change from baseline stroke volume(Baseline, day 5 after intervention)
  • Change from baseline creatinine(Baseline, day 5 after intervention)
  • Change from baseline brain natriuretic peptide(Baseline, day 5 after intervention)
  • Clinical(Up to 6 months)

Study Sites (1)

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