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A Study in Japan of the Safety and Antiviral Activity With Chronic Hepatitis B Infection

Phase 2
Completed
Conditions
Chronic Hepatitis B
Interventions
Registration Number
NCT01020565
Lead Sponsor
Bristol-Myers Squibb
Brief Summary

The objectives of this study are to demonstrate that entecavir has antiviral activity with undetectable at Week 48, and to assess the safety and the pharmacokinetic in Japanese patients given entecavir at each dose of 0.1 and 0.5 mg for 52 weeks

Detailed Description

Not available

Recruitment & Eligibility

Status
COMPLETED
Sex
All
Target Recruitment
60
Inclusion Criteria
  • Documentation of chronic hepatitis B infection by ALL of the following:

    1. Positive for HBsAg OR, negative for IgM core antibody and confirmation of chronic hepatitis B on liver biopsy
    2. Positive for HBeAg OR negative for HBeAg
    3. Documented HBV Viremia on 2 or more occasions and at screening visit: Viremia on sample drawn AND HBV DNA of ≥ 10*5* copies/mL by PCR assay at the screening visit
  • ALT in the range of 1.3 to 10 x ULN

Exclusion Criteria

Not provided

Study & Design

Study Type
INTERVENTIONAL
Study Design
PARALLEL
Arm && Interventions
GroupInterventionDescription
Entecavir (0.1 mg)Entecavir-
Entecavir (0.5 mg)Entecavir-
Primary Outcome Measures
NameTimeMethod
Incidence of clinical adverse events and discontinuations due to adverse events of entecavir at doses of 0.5 and 1 mgWeek 52 (end of dosing) plus 5 days
Incidence of laboratory abnormalities of entecavir at doses of 0.5 and 1 mg for 52 weeksWeek 52 (end of dosing) plus 5 days
Proportion of subjects with reduction in HBV DNA by ≥2 log10 or to undetectable level (<400 copies/mL) by PCR assayWeek 48
Secondary Outcome Measures
NameTimeMethod
Mean change from baseline in log10 HBV DNA measured by PCR assay for each entecavir dose (0.5 and 1 mg) at Week 48Baseline, Week 48
Proportion of subjects who achieve undetectable HBV DNA (<400 copies/mL) by PCR assay at Week 48Week 48
Proportion of subjects HBeAg-positive at baseline who have loss of HBeAg from serum at Week 48Week 48
Proportion of subjects HBeAg-positive at baseline who achieve seroconversion (loss of HBeAg and appearance of HBeAb) at Week 48Week 48
Proportion of subjects with abnormal ALT at baseline who achieve normalization of serum ALT (<1.25 x ULN) at Week 48Week 48
Proportion of subjects HBeAg-positive at baseline who have Complete Response [undetectable HBV DNA levels by PCR assay, negative for HBeAg and normal serum ALT] at Week 48Week 48
Proportion of subjects HBeAg-negative at baseline who have Complete Response [undetectable HBV DNA levels by PCR assay, remain negative for HBeAg and normal serum ALT] at Week 48Week 48
Proportion of subjects who achieve Complete Response, and remain Complete response for 24 weeks after stopping drugWeek 72
Proportion of subjects w/ histological improvement in liver (improvement in necroinflammatory score (≥2 points decrease, Knodell HAI3 score) & no worsening of fibrosis (≥1 point increase, Knodell fibrosis score) at Wk 48 liver biopsy compared to baselineBaseline, Week 48
Changes in liver histology as assessed by the New Inuyama Classification for histological assessment of chronic hepatitisWeek 52
Relationship between HBV isolates (genotypes A,B,C, etc.) at baseline and antiviral activityWeek 48, or at end of dosing (up to Week 52)
Incidence of resistance mutations of HBV isolates in subjects who have a rise in HBV DNA (by ≥1 log above the nadir for that subject) while on study drug.Week 48, or at end of dosing (up to Week 52)
Mutation of HBV DNA polymerase at Week 48 from baselineBaseline, Week 48
Plasma concentrations of entecavir at selected time points during the treatment periodpre-dosing, Week 2 or 4, Week 12, Week 24 and Week 36
Population pharmacokinetic assessment of entecavir developed from concentration-time data obtained from healthy subjectspre-dosing, Week 2 or 4, Week 12, Week 24 and Week 36
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