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Clinical Trials/NCT05456048
NCT05456048
Completed
Not Applicable

Influence of Molecular Abnormalities on Treatment Response of the Regimen of Venetoclax Plus Azacytidine Combined With Homoharringtonine Versus Venetoclax Plus Hypomethylating Agents (HMA) in Relapsed/Refractory Acute Myeloid Leukemia

Nanfang Hospital, Southern Medical University1 site in 1 country231 target enrollmentDecember 3, 2018

Overview

Phase
Not Applicable
Intervention
VAH regimen
Conditions
Relapsed Acute Myeloid Leukemia
Sponsor
Nanfang Hospital, Southern Medical University
Enrollment
231
Locations
1
Primary Endpoint
CR/CRi
Status
Completed
Last Updated
3 years ago

Overview

Brief Summary

The aim of this study is to reveal the influence of gene mutations on the treatment response of the regimen of HHT combined with Venetoclax plus AZA versus venetoclax plus HMA in the salvage therapy of RR-AML.

Detailed Description

Venetoclax-based regimens have heen used in the salvage therapy of relapsed/resfractory (RR) acute myeloid leukemia (AML). More and more studies have shown that molecular abnormalities and venetoclax combined regimens significantly impact the response of venetoclax-based therapy. Our exploratory study revealed that venetoclax plus azacytidine combined with homoharringtonine (VAH) had remarkably higher response than venetoclax plus hypomethylating agents (HMA) in RR-AML. Yet the influence of molecular abnormalities on the response of VAH regimen remains unknown.

Registry
clinicaltrials.gov
Start Date
December 3, 2018
End Date
May 31, 2022
Last Updated
3 years ago
Study Type
Observational
Sex
All

Investigators

Responsible Party
Principal Investigator
Principal Investigator

Qifa Liu

Professor

Nanfang Hospital, Southern Medical University

Eligibility Criteria

Inclusion Criteria

  • Patients must have been treated for at least one cycle of VEN-based regimen and finished outcome assessment.

Exclusion Criteria

  • Acute promyelocytic leukemia (AML subtype M3)
  • Previous exposure to the treatment of VEN-based regimen
  • Cardiac dysfunction (particularly congestive heart failure, unstable coronary artery disease and serious cardiac ventricular arrhythmias requiring antiarrhythmic therapy)
  • Respiratory failure (PaO2 ≤60mmHg)
  • Hepatic abnormalities (total bilirubin ≥2 times the upper limit of normal \[ULN\], alanine aminotransferase (ALT) or aspartate aminotransferase (AST) ≥2 times the ULN)
  • Renal dysfunction (creatinine ≥2 times the ULN or creatinine clearance rate \< 30 mL/min)
  • ECOG performance status 3, 4 or 5
  • Substantial history of neurological, psychiatric, endocrine, metabolic, immunological, or any other medical condition not suitable for the trial (investigators' decision)
  • Active acute or chronic graft-versus-host disease (GVHD). Active acute GVHD or chronic GVHD is defined as GVHD requiring either at least 1 mg/kg per day of prednisone (or equivalent) or treatment beyond systemic corticosteroids.
  • Patients with pregnancy

Arms & Interventions

VAH group

Patients assigned to this group received one to two cycles of VAH regimen as salvage therapy of RR-AML.

Intervention: VAH regimen

VEN+HMA group

Patients assigned to this group received one to two cycles of venetoclax plus HMA regimen as salvage therapy of RR-AML.

Intervention: VEN+HMA regimen

Outcomes

Primary Outcomes

CR/CRi

Time Frame: At the end of Cycle 2 (each cycle is 28 days)

Complete remission and CR with incomplete count recovery

Secondary Outcomes

  • MRD negative(At the end of Cycle 2 (each cycle is 28 days))
  • Overall response(At the end of Cycle 2 (each cycle is 28 days))
  • Overall survival(2 years)
  • Event-free survival(2 years)

Study Sites (1)

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