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Acute Effects of Wine Consumption on Healthy Volunteers

Not Applicable
Conditions
Healthy, Postprandial
Registration Number
NCT01627912
Lead Sponsor
Harokopio University
Brief Summary

The purpose of this study is to investigate whether red and white wine consumption has acute effects on postprandial biochemical markers related to platelet aggregation, inflammation and oxidative stress compared to water or 12.5% ethanol aqueous solution consumption.

Detailed Description

The last few years, epidemiologic studies indicate that regular moderate consumption of alcohol is associated with lower risk of coronary heart disease and heart attack, as well as with lower mortality. More specific, a J or U-shaped association between alcohol consumption and the incidence of coronary heart disease have been suggested, which means that there was lower disease risk in moderate alcohol consumers than in abstainers or heavy drinkers.

The scientific interest was focused on wine after the term "French paradox" was introduced, in order to describe the epidemiological observation that the French suffer a relatively low incidence of coronary heart disease, despite having a diet relatively rich in saturated fats. The paradox was attributed to the moderate consumption of red wine by French. Even though many clinical studies have occurred since then, only few of them report the postprandial effect of wine, mainly focusing on the study of oxidative stress markers and endothelium dysfunction. Also, a limited number of publications refer to the postprandial wine effect upon platelet aggregation, which is an indicative marker for inflammation / thrombosis and atherosclerosis.

The limited clinical evidence prompted us to investigate the postprandial effect of wine consumption upon platelet aggregation, inflammation and oxidation markers, by undertaking a clinical study of crossover design. The subjects randomly consumed 4ml of drink \[Robola or Cabernet Sauvignon or 12.5% ethanol or water\]/kg of individual, parallel with a standardized meal, which consisted of 30.8% carbohydrates, 12.0% proteins and 53.1% fat. The meal total energy was 787.2 kcal.

Recruitment & Eligibility

Status
UNKNOWN
Sex
Male
Target Recruitment
10
Inclusion Criteria
  • healthy
  • non-obese
Exclusion Criteria
  • smokers
  • those who reported slimming or any other dietary regime
  • abstainers from alcohol consumption
  • heavy drinkers
  • athletes
  • subjects who were on medication, such as aspirin, that may have an impact on platelet aggregation or surgical events that may have affected the study outcomes
  • participants with a known diagnosis of either hypertension or diabetes
  • subjects on medication

Study & Design

Study Type
INTERVENTIONAL
Study Design
CROSSOVER
Primary Outcome Measures
NameTimeMethod
platelet aggregation300 min after standardized meal plus tested drink consumption.

In each time point platelet rich plasma (PRP) was isolated from the blood of volunteers and platelet aggregation upon Platelet activating factor (PAF) was measured in CHRONO-LOG aggregometer.

markers of inflammation360 min after standardized meal plus tested drink consumption
markers of oxidative stress360 min after standardized meal plus tested drink consumption

TBARS, ex vivo serum oxidation etc

PAF metabolism360 min after standardized meal plus tested drink consumption

Measurement of PAF biosynthetic / catabolic enzymes in leucocytes and LpPLA2 in serum

Secondary Outcome Measures
NameTimeMethod
Glucose levels360 min after standardized meal plus tested drink consumption
Insulin levels120 min after standardized meal plus tested drink consumption
lipids360 min after standardized meal plus tested drink consumption

HDL-cholesterol, LDL-cholesterol, total chlesterol, triglycerides

Trial Locations

Locations (1)

Department of Nutrition-Dietetics, Harokopio University

🇬🇷

Athens, Greece

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