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The Effect of Ramipril in Suppressing ST2 Expression in Rheumatic Mitral Stenosis Patients

Phase 3
Conditions
Rheumatic Mitral Stenosis
Rheumatic Heart Disease
Fibrosis; Heart
ACE Inhibitor
Mitral Stenosis
Interventions
Registration Number
NCT03991910
Lead Sponsor
Indonesia University
Brief Summary

Objective propose: to investigate the effect of Ramipril in suppressing ST2 (suppression of tumorigenicity 2) in the cardiac mitral valve in patients with Rheumatic Heart Disease. We hypothesized that we hypothesized that ramipril will improve rheumatic mitral valve fibrosis through the downregulation of ST2.

Detailed Description

The efficacy of secondary prevention is limited in the prevention of RHD progression. For this reason, new strategies and therapies are needed to prevent the progression of RHD. Neutralizing inflammatory cytokines or antagonizing their receptor function has been considered as a useful therapeutic strategy to treat autoimmune diseases. In this respect, new therapies targeting ST 2 and their receptors as studied in some autoimmune diseases may promise a new approach for patients with RHD. Angiotensin II induces the upregulation of Transforming growth factor β (TGF-β) and latter the binding of IL-33 to sST2 and not to the natural ligand (ST2L). The binding of IL-33 to sST2 will cause fibrogenesis even more. Thus, ACEI is hypothesized to attenuate this vicious cycle through the inhibition of Angiotensin II and consequently increase Bradykinin that furtherly inhibits fibrosis through the negative regulation of angiotensin II activity in Mitogen Activator Protein Kinase (MAPK) pathways through the suppression of the Ca2+ response and the Na+ transportACE inhibitor were agents with anti-fibrosis effects. The investigators keen to investigate the effect of Ramipril in suppressing ST2 expression as biomarkers of fibrosis in cardiac mitral valve in patients with Rheumatic Heart Disease in the National Cardiac Center Harapan Kita hospital Jakarta Indonesia. This study was designed as a randomized clinical trial. Patients with mitral stenosis valvular dysfunction due to rheumatic process planned for cardiac valve replacement surgery were given Ramipril or placebo for a minimum of 12 weeks (3 months). ST2 expression will be analyzed as the fibrosis biomarker in the mitral valve. This study will be conducted in the Department of Cardiology and Vascular Medicine, University Indonesia, National Cardiac Center Harapan Kita Hospital, Jakarta, Indonesia from June 2019

Recruitment & Eligibility

Status
UNKNOWN
Sex
All
Target Recruitment
66
Inclusion Criteria
  • Patients with mitral valve stenosis or a combination
  • aged more than 18 years
  • undergo cardiac valve replacement operation with or without a tricuspid valve repair,
  • patients with systolic blood pressure (SBP) ≥ 100 mmHg and diastolic blood pressure (DBP) ≥ 60 mmHg
  • passed in medication phase without side effect minimum 4 weeks until operation schedule
Exclusion Criteria
  1. Patients with congenital heart disease
  2. patients with non-mitral valve surgery
  3. patients with coronary artery bypass surgery
  4. patients who refuse to join this study.
  5. adults aged over 65 years or older
  6. pregnant women
  7. patients with autoimmune disease.
  8. Patients with persistent hypotension (systolic blood pressure (BP) < 100 mm Hg)
  9. severe aortic stenosis (aortic valve orifice < 0.75 cm2 )
  10. chronic renal dysfunction with serum creatinine > 2.5 mg/ dL,
  11. known ACEI intolerance.

Study & Design

Study Type
INTERVENTIONAL
Study Design
PARALLEL
Arm && Interventions
GroupInterventionDescription
controlPlaceboscontrol patients will be given a placebo
treatmentRamipril 5Mg Oral CapsuleRamipril 5 mg treatment group
Primary Outcome Measures
NameTimeMethod
ST2 expression in mitral valve tissue and papillary musclea year

expression of ST2 in mitral valve tissue, using immunohistochemistry method

Secondary Outcome Measures
NameTimeMethod
TAPSE (tricuspid annular plane systolic excursion)1 year

echocardiography parameter to asses right ventricular function

End systolic dimension1 year

The diameter across a ventricle at the end of systole, if not else specified then usually referring to the transverse (left-to-right) internal (luminal) distance, excluding thickness of walls, although it can also be measured as the external distance.

Tricuspid regurgitation severity1 year

TRicuspid regurgitation severity is classified ad mild, moderate, and severe, according to European Association of Echocardiography measurement year 2010 for Tricuspid Valve regusrgitation severity.

Ejection fraction1 year

echocardiographic parameter to asses ventricular function

Mitral valve gradient1 year

mitralvalve graient is a echocardiographic parameters of the pressure gradient in the mitral valve

NT-proBNP concentration (pg/ml)a year

concentration of NT-proBNP, plasma markers for cardiac dysfunction.

NYHA classa year

related symptoms will be graded in class I to IV according to NYHA.

All-cause mortality1 year

Study participants will be followed up until 1 year after the surgery for mortality of any cause.

End diastolic dimension1 year

The diameter across a ventricle at the end of diastole, if not else specified then usually referring to the transverse (left-to-right) internal (luminal) distance, excluding thickness of walls, although it can also be measured as the external distance.

ST2 Plasma concentrationa year

plasma level of ST2 measured by ELISA

Tricuspid maximal velocity (Vmax)1 year

Tricuspid maximal velocity (Vmax) is the echocardiographic parameters of the maximal velocity in tricuspid valve annulus

cardiovascular mortality1 year

Study participants will be followed up until 1 year after the surgery for any mortality that is caused by progression of the cardiac disease

Mitral valve area1 year

mitral valve area is the area of mitral valve, measured by the Gorlin formula MVA (cm2) = (CO ÷ DFP) ÷ (38.0 x MPG) where MVA is the mitral valve area, CO is cardiac output, DFP is the diastolic flow period, 38.0 is the constant and MPG is pressure gradient.

Trial Locations

Locations (1)

Ade Meidian Ambari

🇮🇩

Jakarta, DKI Jakarta, Indonesia

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