MedPath

Effects of COVID-19 on Endothelium in HIV-Positive Patients in Sub-Saharan Africa

Conditions
Hiv
ART
Covid19
Interventions
Biological: COVID-19
Biological: HIV
Drug: ART
Registration Number
NCT04709302
Lead Sponsor
Medical University of Graz
Brief Summary

Background: Coronavirus disease 2019 (COVID-19) has affected almost every country in the world, especially in terms of health system capacity and economic burden. People from sub-Saharan Africa (SSA) often face interaction between human immunodeficiency virus (HIV) infection and non-communicable diseases such as cardiovascular disease. Role of HIV infection and anti-retroviral treatment (ART) in altered cardiovascular risk is questionable and there is still need to further carry out research in this field. However, thus far it is unclear, what impact the COVID-19 co-infection in people living with HIV (PLHIV), with or without therapy will have. The ENDOCOVID project aims to investigate whether and how HIV-infection in COVID-19 patients modulates the time course of the disease, alters cardiovascular risk, and changes vascular endothelial function and coagulation parameters/ thrombosis risk.

Methods: In this long-term study, cardiovascular research on PLHIV with or without ART with COVID-19 and HIV-negative with COVID-19 will be carried out via clinical and biochemical measurements for cardiovascular risk factors and biomarkers of cardiovascular disease (CVD). Vascular and endothelial function will be measured by brachial artery flow-mediated dilatation (FMD), carotid intima-media thickness (IMT) assessments, and retinal blood vessel analyses, along with vascular endothelial biomarkers and coagualation markers. The correlation between HIV-infection in COVID-19 PLHIV with or without ART and its role in enhancement of cardiovascular risk and endothelial dysfunction will be assessed. Potential changes in these endpoints by COVID-19 will be followed for 4 weeks across the three groups (PLHIVwith or without ART and HIV negatives).

Impact of project: The ENDOCOVID project aims to evaluate in the long-term the cardiovascular risk and vascular endothelial function in PLHIV thus revealing an important transitional cardiovascular phenotype in COVID-19.

Detailed Description

Background:

SARS-CoV-2 has affected almost every country worldwide with regard to health system capacities and economic burden, so far. The typical clinical presentations of COVID-19 are fever, sore through, cough, dyspnea, abdominal pain and diarrhea. Approximately 15% of patients with COVID-19 must be hospitalized and 5% are critically ill, and often develop acute respiratory distress syndrome and need to be admitted to intensive care units. Risk of severe disease increases with age, male sex, and with co-morbidities such as chronic lung disease, CVDs, and diabetes. COVID-19 can also affect the nervous systems and cause loss of smell or taste sensation.

Populations in SSA are increasingly facing a double burden of disease involving the interaction between HIV-AIDS and non-communicable diseases such as CVDs. In general, vascular endothelial function can be regarded as a marker of the net harmful effects of cardiovascular risk factors on the vascular wall. HIV- infected patients have premature atherosclerosis and increased risk of ischemic heart disease. The cardiovascular risk represented by HIV-infection is moreover affected by anti-retroviral treatment (ART). ART may, among other effects, prompt dyslipidaemic and metabolic changes in patients over to systemic immune activation, stimulation of endothelial inflammation and atherosclerosis. However, a recent meta-analysis showed that evidence for - and against - a role for ART in the development of CVDs remains unconvincing and that more research is necessary.

With the current COVID-19 pandemic, which has also spread across Africa, another burden is added. However, thus far it is unclear, what effect the additional COVID-19 co-infection in people with HIV (PLHIV) will have as there are no data yet on how HIV/AIDS a prevalent diseases SSA, will affect COVID-19 infection rates or outcomes. Currently, there is no proof for a higher COVID -19 infection rate in PLHIV compared to HIV-negatives. However, there is a possibility that HIV infection may have an influence on the course of infection. PLHIV who are on ART with a normal CD4 T-cell count and suppressed viral load should not be at an increased risk of severe disease. On the other hand, this theoretically positive situation could be counterbalanced by recent evidence that ART could also have effects on COVID-19. Furthermore, HIV-infected patients may additionally suffer from co-morbidities, which could pose a greater risk of COVID-19 infection. Currently, there is no data available about COVID-19 infection in PLHIV in SSA reflecting who in particular is at high risk for infections, complications and fatal outcomes. As COVID-19 associated multisystem inflammation, and the way organ damage caused by COVID-19 occurring in patients with COVID-19 is still incompletely understood, and current treatment options are limited. There is therefore an urgent need to better understand the risk of severe and fatal COVID-19 outcomes, especially in PLHIV (+/- ART).

A severe course of COVID-19 can cause the development of COVID-19-associated coagulopathy, with features of both disseminated intravascular coagulation and thrombotic microangiopathy, resulting in widespread microvascular thrombosis that may involve consumption of coagulation factors and the liver. There seems to be a causal relationship with the inflammatory and reparative processes involving diffuse alveolar damage (DAD), because thrombi are frequently detected in small pulmonary arteries, most presumed secondary to endothelial damage. The damage of endothelium could be due to the direct viral infection of the endothelial cells, which express ACE-2 receptors, or to a host response. Besides, the alveolar fibrin deposition in DAD may affect the local homeostasis between fibrinolysis and coagulation. Alveolar and endothelial damage of smaller vessels may be followed by microvascular pulmonary thrombosis, which could then extend to larger vessels. Additionally, elevated D-dimer has been observed in patients with COVID-19. It is known that inceased D-dimer is associated with acute pulmonary emboli (APE), deep venous thrombosis (DVT), cancer, peripheral vascular disease, inflammatory diseases and pregnancy. It has been found that patients with COVID-19, including those not on respirators but bedconfined, develop DVT and APE much earlier than expected. Even with usage of prophylactic anticoagulation, autopsy has shown that deaths in COVID-19 may be caused by the thrombosis in segmental and subsegmental pulmonary arterial vessels. ENDOCOVID project aims to evaluate the degree to which COVID-19 induced inflammation contributes to endothelial function and coagulation changes and cardiometabolic risk in PLHIV (+/-ART). Endothelial function will be evaluated non-invasively via FMD and blood asymmetric dimethyl arginine (ADMA).

Problem statement HIV infection is a burden for the cardiovascular system but coinfection of HIV and SARS-CoV-2 in SSA population could be a very big load and could lead to higher menace of cardiovascular health. However, the potential of risk in this population, including early vascular and endothelial changes, is yet unclear. Given the ongoing global pandemic, epidemiological studies to address this knowledge gap is necessary. The aim of this study is to evaluate cardiovascular risk in HIV and COVID-19 populations, which also would allow assessment of current - and future - cardiovascular risk, via vascular endothelial function measurements. The ENDOCOVID project has been designed to give an evaluation of cardiovascular risk in PLHIV and COVID-19 living in SAA. ENDOCOVID builds up on the EndoAfrica study, which is currently being carried out in SSA.

Aim and objectives The general aim of the ENDOCOVID project is to identify whether and how HIV-infection with or without ART in COVID-19 patients relates to the time course of the disease, alters cardiovascular risk, and changes vascular endothelial structure and function in adults living in the SSA.

Three groups of participants will take part in this project:

1. PLHIV without ART but with COVID-19

2. PLHIV with ART and COVID-19

3. HIV-negative patients with COVID-19, sex- /age matched

The following objectives will be investigated:

1. Evaluation of the prevalence of COVID-19 in PLHIV (with or without ART) admitted to clinics in South Africa and Nigeria because of COVID-19.

2. Assessment of the incidence of acute respiratory distress syndrome, admission to ICU, fatal course of disease (30-days mortality), and thromboembolic events in hospitalized patients included in the study.

3. Evaluation of the influence of ART on severity of COVID-19 in PLHIV (with or without ART) as well as clinical presentation, changes of endothelial/vascular function and biomarkers.

4. Evaluation of the effects of COVID-19 on endothelial/vascular function, thrombotic factors, and oro-naso sensory alterations over infection and after recovery.

5. Correlation of hospitalization and mortality of COVID-19 infected PLHIV(with or without ART) and HIV negatives with co-morbidity incidence;

6. Assessment of the incidence of obesity and insulin resistance in the 3 groups.

Recruitment & Eligibility

Status
ENROLLING_BY_INVITATION
Sex
All
Target Recruitment
342
Inclusion Criteria

Not provided

Exclusion Criteria

Not provided

Study & Design

Study Type
OBSERVATIONAL
Study Design
Not specified
Arm && Interventions
GroupInterventionDescription
COVID-19 positive, HIV-positive with ARTCOVID-19Patients tested positive for SARS-CoV-2 Patients tested positive for HIV who are on ART
COVID-19 positive, HIV-positive with ARTARTPatients tested positive for SARS-CoV-2 Patients tested positive for HIV who are on ART
COVID-19 positive, HIV-positive without ARTCOVID-19Patients tested positive for SARS-CoV-2 Patients tested positive for HIV ART naive group
COVID-19 positive, HIV-positive with ARTHIVPatients tested positive for SARS-CoV-2 Patients tested positive for HIV who are on ART
COVID-19 positive, HIV-positive without ARTHIVPatients tested positive for SARS-CoV-2 Patients tested positive for HIV ART naive group
COVID-19 positive, HIV-negativeCOVID-19Patients tested positive for SARS-CoV-2 Patients tested positive for HIV ART naive group
Primary Outcome Measures
NameTimeMethod
Number of patients developing Acute Respiratory Distress Syndromethrough study completion, up to 3 months

Comparison of all three groups, recorded for each patient

Number of ICU admissionsthrough study completion, up to 3 months

Comparison of all three groups, recorded for each patient

Number of Deathsthrough study completion, up to 3 months

Comparison of all three groups

Secondary Outcome Measures
NameTimeMethod
Endothelial function and vascular changes: PWVthrough study completion, an average of 3 months

PWV, a marker of aortic stiffness, is most commonly measured as the time it takes a pulse wave to travel from the carotid to the femoral arteries divided by the distance multiplied by 0.8. PWV can be measured by several devices. The non-invasive Vicorder device has been shown to have good reproducibility - even when the assessor has limited experience in its usage - and the results obtained reflect those obtained via invasive central blood pressure measurements and those of SphygmoCor device.

Coagulatory Parameters: CATthrough study completion, an average of 3 months

Calibrated automated thrombography as thrombosis marker (Thrombinoscope BV)

Cardiometabolic Status: hs-CRPthrough study completion, an average of 3 months

high sensitivity CRP as inflammatory marker, assessed via blood samples in a routine laboratory

Endothelial function and vascular changes: IMTthrough study completion, an average of 3 months

Intima media thickness is assessed via ultrasound in the carotid artery

Cardiometabolic Status: Cholesterolthrough study completion, an average of 3 months

To assess the lipid profile, cholesterol levels will be assessed via blood samples in a routine laboratory

Cardiometabolic Status: Triglyceridesthrough study completion, an average of 3 months

To assess the lipid profile, triglyceride concentrations will be assessed via blood samples in a routine laboratory

Coagulatory Parameters: D-dimerthrough study completion, an average of 3 months

increased D-Dimer as marker for pulmonary embolism in a routine laboratory

Cardiometabolic Status: ADMAthrough study completion, an average of 3 months

Asymmetric dimethylarginine (ADMA) as marker for endothelial function (from blood sample) measured via commercially available ELISA kits

Cardiometabolic Status: Glycated Hemoglobinthrough study completion, an average of 3 months

HbA1c as marker for diabetes, assessed via blood samples in a routine laboratory

Endothelial function and vascular changes: FMDthrough study completion, an average of 3 months

Assessed non-invasively using ultrasound in the brachial artery (flow mediated dilatation)

Endothelial function and vascular changes: Retinal microvasculature analysisthrough study completion, an average of 3 months

A non-mydriatic, hand-held, portable digital retinal camera (Optomed Aurora, Optomed Oy, Oulu, Finland) will be used for collecting retinal images. Dimensions of vessels and microvascular state will be analyzed offline with the semi-automated MONA REVA software (VITO, Belgium).

Cardiometabolic Status: HDL cholesterolthrough study completion, an average of 3 months

To assess the lipid profile, HDL cholesterol concentrations will be assessed via blood samples in a routine laboratory

Cardiometabolic Status: LDL cholesterolthrough study completion, an average of 3 months

To assess the lipid profile, LDL cholesterol concentrations will be assessed via blood samples in a routine laboratory

Cardiometabolic Status: Glucosethrough study completion, an average of 3 months

Assessed via blood samples in a routine laboratory

Coagulatory Parameters: TFthrough study completion, an average of 3 months

Tissue factor (TF) measured via triggered thromboelastometry (TEM) coagulation analyzer (ROTEM05)

Trial Locations

Locations (3)

Lagos Stae University Teaching Hospital Ikeja

🇳🇬

Lagos, Nigeria

Walter Sisulu University

🇿🇦

Mthatha, South Africa

University of Ilorin Teaching Hospital

🇳🇬

Ilorin, Kwara State, Nigeria

© Copyright 2025. All Rights Reserved by MedPath