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Mycose AdminiStration for HealIng Alzheimer NEuropathy (MASHIANE)

Phase 1
Conditions
Alzheimer Disease
Interventions
Registration Number
NCT04663854
Lead Sponsor
Mashhad University of Medical Sciences
Brief Summary

Alzheimer's disease (AD) is an irreversible, progressive brain disorder that slowly destroys memory and thinking skills, and, eventually, the ability to function independently. Despite the significant effort to understand the basic biology of the disease and pharmaceutical advances to develop drugs, there is no effective therapy available to treat AD or slow the disease progression.

β-amyloid accumulation outside brain cells and abnormal accumulations of tau protein inside neurons are taught to be two main changes in the brain that lead to AD. Progressive accumulation of β-amyloid interferes with the neuron-to-neuron communication at synapses, contributing to neural cell death. Also, tau tangles block the transport of nutrients and other essential molecules into the neurons. Many molecules have been shown to inhibit amyloid aggregation. The anti-amyloidogenic activity of trehalose was confirmed in both in vitro and in vivo studies and its inhibitory effects on β-amyloid formation in AD have also been demonstrated. Trehalose is a non-toxic disaccharide and no dose-dependent adverse effects were seen in any of the safety studies. It can act as a chemical chaperone and stabilizes the natively folded structure of protein and also trehalose has been identified as an autophagy inducer and promotes the clearance of aggregated proteins. Therefore, trehalose could be a valuable candidate for the treatment and prevention of amyloid-related disease. Based on the proposed hypothesis, this study aim to investigate the potential efficacy of trehalose administration in patients with AD.

Detailed Description

Purpose of the study: A randomized, triple-blind, pilot clinical trial has been designed to evaluate the effectiveness of trehalose on reducing the symptoms in AD patients. Study intervention: twenty patients with Alzheimer's disease were randomly divided into an intervention and a control group. Trehalose will be administrated intravenously (15 g/week) for 12 weeks in the intervention group and the control group will be received normal saline as a placebo.

Recruitment & Eligibility

Status
UNKNOWN
Sex
All
Target Recruitment
20
Inclusion Criteria
  • Mini-Mental State Examination (MMSE) score range from 10 to 23
  • Not having other cognitive disorders
Exclusion Criteria
  • MMSE score higher than 23 or lower than 10
  • The presence of other cognitive disorders which will be evaluated by clinical assessment and brain imaging
  • The presence of factors affecting cognitive impairment such as depression
  • Vascular dementia and Lewy body dementia
  • Previous history of head trauma
  • Use of alcohol and other drugs that affect cognitive functioning

Study & Design

Study Type
INTERVENTIONAL
Study Design
PARALLEL
Arm && Interventions
GroupInterventionDescription
PlaceboTrehaloseParticipants will be received equal volume of normal saline weekly for a period of 12 weeks.
TrehaloseTrehaloseTrehalose Participants will be received intravenous trehalose infusion weekly (15 g/week) for a period of 12 weeks.
Primary Outcome Measures
NameTimeMethod
Changes of Mini-Mental State Exam (MMSE)From baseline to 12 weeks

Global cognition will be assessed by MMSE test, which will be conducted at baseline and week 12.

Changes in Clinical Dementia Rating Scale (CDR)From baseline to 12 weeks

Clinical Dementia Rating Scale (CDR) has six different cognitive and behavioral domains such as memory, orientation, judgment and problem solving, community affairs, home and hobbies performance, and personal care, which will be conducted at baseline and week 12.

Secondary Outcome Measures
NameTimeMethod

Trial Locations

Locations (1)

Ghaem Educational, Research and Treatment Center

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Mashhad, Razavi Khorasan, Iran, Islamic Republic of

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