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The Farnesoid X Receptor (FXR) Ligand Obeticholic Acid in NASH Treatment Trial(FLINT)

Phase 2
Completed
Conditions
Nonalcoholic Fatty Liver Disease (NAFLD)
Nonalcoholic Steatohepatitis (NASH)
Interventions
Registration Number
NCT01265498
Lead Sponsor
National Institute of Diabetes and Digestive and Kidney Diseases (NIDDK)
Brief Summary

Administration of the farnesoid X receptor (FXR) ligand obeticholic acid (OCA) for 72 weeks to subjects with biopsy evidence of nonalcoholic steatohepatitis (NASH) will result in improvement in their liver disease as measured by changes in the nonalcoholic fatty liver disease (NAFLD) activity score (NAS).

Detailed Description

To evaluate whether treatment with obeticholic acid, 25 mg daily for 72 weeks compared to treatment with placebo, improves the severity of nonalcoholic fatty liver disease (NAFLD) as determined from hepatic histology.

Recruitment & Eligibility

Status
COMPLETED
Sex
All
Target Recruitment
283
Inclusion Criteria
  • 18 years of age or older as of the initial screening interview and provision of consent
  • Histologic evidence of definite or probable nonalcoholic steatohepatitis (NASH) based upon a liver biopsy obtained no more than 90 days prior to randomization and a nonalcoholic fatty liver disease activity score (NAS) of 4 or greater with at least 1 in each component of the NAS score (steatosis scored 0-3, ballooning degeneration scored 0-2, and lobular inflammation scored 0-3).
Exclusion Criteria
  • Current or history of significant alcohol consumption for a period of more than 3 consecutive months within 1 year prior to screening (significant alcohol consumption is defined as more than 20 grams per day in females and more than 30 grams per day in males, on average)

  • Inability to reliably quantify alcohol consumption based upon local study physician judgment

  • Use of drugs historically associated with nonalcoholic fatty liver disease (NAFLD) (amiodarone, methotrexate, systemic glucocorticoids, tetracyclines, tamoxifen, estrogens at doses greater than those used for hormone replacement, anabolic steroids, valproic acid, and other known hepatotoxins) for more than 2 weeks in the year prior to randomization

  • Prior or planned (during the study period) bariatric surgery (eg, gastroplasty, roux-en-Y gastric bypass)

  • Uncontrolled diabetes defined as Hemoglobin A1c 9.5% or higher within 60 days prior to enrollment

  • Presence of cirrhosis on liver biopsy

  • A platelet count below 100,000/mm3

  • Clinical evidence of hepatic decompensation as defined by the presence of any of the following abnormalities:

    • Serum albumin less than 3.2 grams/deciliter (g/dL)
    • International Normalized Ratio(INR)greater than 1.3
    • Direct bilirubin greater than 1.3 milligrams per deciliter (mg/dL)
    • History of esophageal varices, ascites or hepatic encephalopathy
  • Evidence of other forms of chronic liver disease:

    • Hepatitis B as defined by presence of hepatitis B surface antigen (HBsAg)
    • Hepatitis C as defined by presence of hepatitis C virus (HCV) ribonucleic acid (RNA) or positive hepatitis C antibody (anti-HCV)
    • Evidence of ongoing autoimmune liver disease as defined by compatible liver histology
    • Primary biliary cirrhosis as defined by the presence of at least 2 of these criteria (i) Biochemical evidence of cholestasis based mainly on alkaline phosphatase elevation (ii)Presence of anti-mitochondrial antibody (AMA) (iii)Histologic evidence of nonsuppurative destructive cholangitis and destruction of interlobular bile ducts
    • Primary sclerosing cholangitis
    • Wilson's disease as defined by ceruloplasmin below the limits of normal and compatible liver histology
    • Alpha-1-antitrypsin(A1AT) deficiency as defined by diagnostic features in liver histology (confirmed by alpha-1 antitrypsin level less than normal; exclusion at the discretion of the study physician)
    • History of hemochromatosis or iron overload as defined by presence of 3+ or 4+ stainable iron on liver biopsy
    • Drug-induced liver disease as defined on the basis of typical exposure and history
    • Known bile duct obstruction
    • Suspected or proven liver cancer
    • Any other type of liver disease other than nonalcoholic steatohepatitis (NASH)
  • Serum alanine aminotransferase (ALT) greater than 300 units per liter (U/L)

  • Serum creatinine of 2.0 mg/dL or greater

  • Use of ursodeoxycholic acid (Ursodiol, Urso) within 90 days prior to enrollment

  • Inability to safely obtain a liver biopsy

  • History of biliary diversion

  • Known positivity for Human Immunodeficiency Virus (HIV) infection

  • Active, serious medical disease with likely life expectancy less than 5 years

  • Active substance abuse including inhaled or injection drugs in the year prior to screening

  • Pregnancy, planned pregnancy, potential for pregnancy and unwillingness to use effective birth control during the trial, breast feeding

  • Participation in an investigational new drug (IND) trial in the 30 days before randomization

  • Any other condition which, in the opinion of the investigator, would impede compliance or hinder completion of the study

  • Failure to give informed consent

Study & Design

Study Type
INTERVENTIONAL
Study Design
PARALLEL
Arm && Interventions
GroupInterventionDescription
Obeticholic acidobeticholic acidobeticholic acid
PlaceboplaceboPlacebo
Primary Outcome Measures
NameTimeMethod
Hepatic Histological Improvement in Nonalcoholic Fatty Liver Disease (NAFLD) Activity Score (NAS)baseline to 72 weeks

Centrally scored histological improvement in nonalcoholic fatty liver disease (NAFLD) from baseline to the end of 72 weeks of treatment, where improvement is defined as:

1. No worsening in fibrosis; and

2. A decrease in NAFLD Activity Score (NAS) of at least 2 points

Secondary Outcome Measures
NameTimeMethod
Change in Bicarbonatebaseline to 72 weeks
Change in Calciumbaseline to 72 weeks
Change in Phosphatebaseline to 72 weeks
Change in Creatininebaseline to 72 weeks
Change in Uric Acidbaseline to 72 weeks
Change in Albuminbaseline to 72 weeks
Change in Total Proteinbaseline to 72 weeks
Change in Prothrombin Timebaseline to 72 weeks
Change in International Normalised Ratiobaseline to 72 weeks
Change in Fasting Serum Glucosebaseline to 72 weeks
Change in Insulinbaseline to 72 weeks
Change in HOMA-IRbaseline to 72 weeks
Change in Glycated Haemoglobin A1cbaseline to 72 weeks
Change in Weightbaseline to 72 weeks
Change in Body-mass Indexbaseline to 72 weeks
Change in Waist Circumferencebaseline to 72 weeks
Change in Waist-to-hip Ratiobaseline to 72 weeks
Change in Systolic Blood Pressurebaseline to 72 weeks
Change in Diastolic Blood Pressurebaseline to 72 weeks
Change in Haematocritbaseline to 72 weeks
Change in Mean Corpuscular Volumebaseline to 72 weeks
Change in White Blood Cell Countbaseline to 72 weeks
Change in Platelet Countbaseline to 72 weeks
Resolution of NASH Diagnosisbaseline to 72 weeks

Resolution of definite nonalcoholic steatohepatitis. Resolution defined as either not NAFLD, or NAFLD but not non-alcoholic steatohepatitis on week 72 biopsy

Fibrosis: Patient With Improvementbaseline to 72 weeks

Patients with improvement in fibrosis score. Fibrosis was assessed on a scale of 0-4, with higher scores showing more severe fibrosis.

Fibrosis: Change in Scorebaseline to 72 weeks

Change in fibrosis score. Fibrosis was assessed on a scale of 0-4, with higher scores showing more severe fibrosis.

Total NAFLD Activity Score: Change in Scorebaseline to 72 weeks

NAFLD activity score was assessed on a scale of 0-8, with higher scores showing more severe disease (the components of this measure are steatosis \[assessed on a scale of 0-3\], lobular inflammation \[assessed on a scale of 0-3\], and hepatocellular ballooning \[assessed on a scale of 0-2\]).

Hepatocellular Ballooning: Patients With Improvementbaseline to 72 weeks

Patients with improvement in hepatocellular ballooning score. Hepatocellular ballooning was assessed on a scale of 0-2, with higher scores showing more severe ballooning.

Hepatocellular Ballooning: Change in Scorebaseline to 72 weeks

Change in hepatocellular ballooning score. Hepatocellular ballooning was assessed on a scale of 0-2, with higher scores showing more severe ballooning.

Steatosis: Patients With Improvementbaseline to 72 weeks

Patients with improvement in steatosis score. Steatosis was assessed on a scale of 0-3, with higher scores showing more severe steatosis.

Steatosis: Change in Scorebaseline to 72 weeks

Change in steatosis score. Steatosis was assessed on a scale of 0-3, with higher scores showing more severe steatosis.

Lobular Inflammation: Patients With Improvementbaseline to 72 weeks

Patients with improvement in lobular inflammation score. Lobular inflammation was assessed on a scale of 0-3, with higher scores showing more severe lobular inflammation.

Lobular Inflammation: Change in Scorebaseline to 72 weeks

Change in lobular inflammation score. Lobular inflammation was assessed on a scale of 0-3, with higher scores showing more severe lobular inflammation.

Portal Inflammation: Patients With Improvementbaseline to 72 weeks

Patients with improvement in portal inflammation score. Portal inflammation was assessed on a scale of 0-2, with higher scores showing more severe portal inflammation.

Portal Inflammation: Change in Scorebaseline to 72 weeks

Change in portal inflammation score. Portal inflammation was assessed on a scale of 0-3, with higher scores showing more severe portal inflammation.

Change in Alanine Aminotransferasebaseline to 72 weeks
Change in Asparate Aminotransferasebaseline to 72 weeks
Change in Alkaline Phosphatasebaseline to 72 weeks
Change in γ-glutamyl Transpeptidasebaseline to 72 weeks
Change in Total Bilirubinbaseline to 72 weeks
Change in Total Cholesterolbaseline to 72 weeks
Change in HDL Cholesterolbaseline to 72 weeks
Change in LDL Cholesterolbaseline to 72 weeks
Change in Triglyceridesbaseline to 72 weeks
Change in Haemoglobinbaseline to 72 weeks
Change in SF-36 Quality of Life Physical Component Summarybaseline to 72 weeks

Short Form (36) Health Survey The SF-36 evaluates health-related quality of life in 8 domains consisting of two components: physical and mental. The score for each domain ranges from 0 to 100. Norm based scoring (based on the general US population) is used with a mean of 50 and standard deviation of 10. Higher values represent a better outcome.

Change in SF-36 Quality of Life Mental Component Summarybaseline to 72 weeks

Short Form (36) Health Survey The SF-36 evaluates health-related quality of life in 8 domains consisting of two components: physical and mental. The score for each domain ranges from 0 to 100. Norm based scoring (based on the general US population) is used with a mean of 50 and standard deviation of 10. Higher values represent a better outcome.

Trial Locations

Locations (9)

Duke University Medical Center

🇺🇸

Durham, North Carolina, United States

Cleveland Clinic Foundation

🇺🇸

Cleveland, Ohio, United States

University of California, San Diego

🇺🇸

San Diego, California, United States

Indiana University

🇺🇸

Indianapolis, Indiana, United States

Case Western Reserve University

🇺🇸

Cleveland, Ohio, United States

University of California, San Francisco

🇺🇸

San Francisco, California, United States

St. Louis University

🇺🇸

Saint Louis, Missouri, United States

Virginia Commonwealth University

🇺🇸

Richmond, Virginia, United States

Virginia Mason Medical Center

🇺🇸

Seattle, Washington, United States

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