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Lurasidone Vs Olanzapine on Neurotrophic Biomarkers and Cardiometabolic Parameters in Unmedicated Schizophrenia

Phase 4
Completed
Conditions
Schizophrenia
Interventions
Registration Number
NCT03304457
Lead Sponsor
All India Institute of Medical Sciences, Bhubaneswar
Brief Summary

Schizophrenia (SCZ) is a chronic, severe and disabling mental disorder with unclear etiology and pathophysiology concerned with neuro-developmental,neurodegenerative abnormalities and cognitive impairmentslinked to behavioural changes.According to neurotrophic hypothesis, the changes result due to the abnormal regulation of neurotrophic factor, especially the decreased serum brain derived neurotrophic factor (BDNF) validated by several meta-analyses. However, the regulation of nerve growth factor (NGF) in SCZ remains unclear because of the inconsistent findings from the previous clinical studies.

Lurasidone is a novel antipsychotic drug approved for adult SCZ and for affective symptomatology \& cognitive deficits. Principal advantages over some other second-generation antipsychotics are its highly favourable metabolic profile and once daily dosing regimen. Some of the studies indicate that risperidone, olanzapine, clozapine \& aripiprazole might not alter BDNF levels, at least within 8 weeks of treatment.While other two studies with olanzapine suggest that BDNF might influence the response to monotherapy in SCZ patients.All these previous studies are non-conclusive \& contradictory to each other which draw our attention for doing the research further to reach a conclusive result about the effect of olanzapine and lurasidone on neurotrophic biomarkers in SCZ.

Detailed Description

Schizophrenia (SCZ) is a chronic, severe and disabling mental disorder with unclear aetiology and pathophysiology concerned with neuro-developmental,neurodegenerative abnormalities and cognitive impairments linked to behavioural changes.According to neurotrophic hypothesis, the changes result due to the abnormal regulation of neurotrophic factor, especially the decreased serum brain derived neurotrophic factor (BDNF) validated by several meta-analyses. However, the regulation of nerve growth factor (NGF) in SCZ remains unclear because of the inconsistent findings from the previous clinical studies.

Lurasidone is a novel antipsychotic drug approved for adult SCZ and for affective symptomatology \& cognitive deficits. Principal advantages over some other second-generation antipsychotics are its highly favourable metabolic profile and once daily dosing regimen. Some of the studies indicate that risperidone, olanzapine, clozapine \& aripiprazole might not alter BDNF levels, at least within 8 weeks of treatment.While other two studies with olanzapine suggest that BDNF might influence the response to monotherapy in SCZ patients.All these previous studies are non-conclusive \& contradictory to each other which draw our attention for doing the research further to reach a conclusive result about the effect of olanzapine and lurasidone on neurotrophic biomarkers in SCZ.

Most of the antipsychotic drugs prescribed for SCZ are based on the dopamine hypothesis. In recent times, neurotrophic hypothesis gained importance in the pathophysiology of SCZ. So, our study may enable psychiatrist to choose a better antipsychotic drug having effect on both dopamine as well as neurotrophic factors. Previously there were no studies on effect of lurasidone on neurotrophic factors in SCZ \& also there was no head-on comparison of lurasidone and olanzapine

Recruitment & Eligibility

Status
COMPLETED
Sex
All
Target Recruitment
100
Inclusion Criteria
  • All treatment naive patients clinically diagnosed first episode of SCZ according to ICD-10
  • Patients of either sex with age range 18-45 years
  • Treatment naïve patients
Exclusion Criteria
  • Other Psychotic spectrum disorders (F21- F29)
  • Highly agitated/ violent/ suicidal patients who need immediate treatment
  • Patients with comorbid substance abuse except Nicotine use or history of organicity
  • Patients with known history of diabetes mellitus, hypertension or any long standing significant medical illness/ significant neurological impairment/ clinical observable mental retardation
  • Pregnant and nursing women

Study & Design

Study Type
INTERVENTIONAL
Study Design
PARALLEL
Arm && Interventions
GroupInterventionDescription
OlanzapineOlanzapineOlanzapine will be prescribed at a dose of 10mg once daily orally for 6 weeks
LurasidoneLurasidoneLurasidone will be prescribed at a dose of 80mg/day once daily orally for 6 weeks
Primary Outcome Measures
NameTimeMethod
Serum brain derived neurotrophic factor (BDNF)6 weeks

the change in serum level of BDNF from baseline after treatment with lurasidone or olanzapine

Secondary Outcome Measures
NameTimeMethod
serum nerve growth factor (NGF)6 weeks

the change in serum level of NGF from baseline after treatment with lurasidone or olanzapine

Social and occupational functioning assessment scale (SOFAS)6 weeks

To determine the association (if any) between change in serum neurotrophic factor and SOFAS (Social and occupational functioning assessment scale) score

PANSS score6 weeks

To determine the association (if any) between change in serum neurotrophic factor and PANSS score

High Density Lipoprotein (HDL)6 week

to assess dyslipidemia

Serum Triglyceride6 weeks

to assess dyslipidemia

Glycosylated Hemoglobin (HbA1c)6 weeks

to assess dysglycemia

Very Low Density Lipoprotein (VLDL)6 week

to assess dyslipidemia

Serum Neurotrophin 3 (NT3)6 weeks

the change in serum level of NT3 from baseline after treatment with lurasidone or olanzapine

Serum Insulin6 weeks

to assess insulin resistance

Fasting blood sugar6 weeks

to assess dysglycemia

Low Density Lipoprotein (LDL)6 week

to assess dyslipidemia

LDL/HDL ratio6 weeks

to assess dyslipidemia and cardiovascular risk

Serum hsCRP6 weeks

to assess cardiovascular risk in schizophrenia

Trial Locations

Locations (1)

AIIMS

🇮🇳

Bhubaneshwar, Odisha, India

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