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Influence of Central Nervous Insulin Sensitivity on Insulin Secretion

Not Applicable
Completed
Conditions
Insulin Secretion
Interventions
Registration Number
NCT02870361
Lead Sponsor
University Hospital Tuebingen
Brief Summary

Insulin resistance is a central pathophysiological component of type 2 diabetes and is associated with a high risk of cardiovascular disease. The tissue in which it manifests are mainly muscle, liver, and adipose tissue. Since the transport of glucose to the brain is independent of insulin, this organ has traditionally not been studied in this regard. In animal experiments, however, knockout of the insulin receptor in the brain leads to obesity and peripheral insulin resistance. This finding of insulin action in the brain could also be confirmed in human studies.

The investigators intend to investigate whether central nervous insulin action affects insulin secretion in humans. For this purpose, nasal insulin and placebo are administered 15 minutes before a hyperglycemic hyperinsulinemic clamps, which stimulate insulin secretion. Insulin sensitivity of the brain is measured by a an established protocol with functional magnetic resonance imaging before and after nasal insulin administration.

Detailed Description

Not available

Recruitment & Eligibility

Status
COMPLETED
Sex
Male
Target Recruitment
15
Inclusion Criteria
  • HbA1c ≤6.0%
  • normal glucose tolerance during 75g oral glucose tolerance test (OGTT)
Exclusion Criteria
  • Not removable metal parts in or on the body
  • manifest cardiovascular disease
  • claustrophobia
  • recent surgery (less than 3 months)
  • Simultaneous participation in other studies
  • Acute disease or infection within the last 4 weeks
  • neurological and psychiatric disorders
  • treatment with centrally acting drugs
  • hemoglobin Hb <13g / dl
  • Hypersensitivity to any of the substances used

Study & Design

Study Type
INTERVENTIONAL
Study Design
CROSSOVER
Arm && Interventions
GroupInterventionDescription
Insulin nasal sprayintranasal insulin160 Units of human insulin as nasal spray
Placebo nasal sprayPlaceboNasal spray containing placebo solution
Primary Outcome Measures
NameTimeMethod
Insulin secretion assessed as serum C-peptide levels during a hyperglycemic clamp0-10 min during hyperglycemic clamp and 10-90 min during hyperglycemic clamp
Secondary Outcome Measures
NameTimeMethod
Correlation with autonomous nervous system activity10 - 150 minutes post nasal spray

Correlation of the change in pancreatic insulin secretion by central insulin action with the simultaneous change of the autonomous nervous system (measured by heart rate variability).

Peripheral insulin sensitivity10-90 min and 70-90 min during hyperglycemic clamp

Effect of nasal insulin versus placebo on peripheral insulin sensitivity assessed by hyperglycemic hyperinsulinemic clamp.

Correlation with brain insulin sensitivity15-30 minutes post insulin nasal spray

Correlation of regional brain insulin sensitivity with the change of pancreatic insulin secretion due to central action of insulin. Changes in regional cerebral blood flow from before to after intranasal insulin administration will be assessed by functional magnetresonance imaging (fMRI)

Differential effects in lean and overweight0-10 min during hyperglycemic clamp and 10-90 min during hyperglycemic clamp

Differences in the effect of nasal insulin versus placebo on C-peptide levels during a hyperglycemic clamp between lean and overweight men will be assessed.

Trial Locations

Locations (1)

University of Tuebingen, Department of Internal Medicine IV

🇩🇪

Tübingen, Germany

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