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Clinical Trials/NCT00276302
NCT00276302
Completed
Phase 1

A Phase 1, Safety Assessment and Pharmacokinetic Study of IPI-504 in Patients With Either Metastatic and/or Unresectable Gastrointestinal Stromal Tumors (GIST) or Advanced or Metastatic Soft Tissue Sarcomas (STS)

Infinity Pharmaceuticals, Inc.4 sites in 2 countries63 target enrollmentDecember 2005

Overview

Phase
Phase 1
Intervention
IPI-504
Conditions
Gastrointestinal Stromal Tumors
Sponsor
Infinity Pharmaceuticals, Inc.
Enrollment
63
Locations
4
Primary Endpoint
To recommend a dose for subsequent studies of IPI-504
Status
Completed
Last Updated
15 years ago

Overview

Brief Summary

The primary objectives of the study are:

  • Determine the safety and maximum tolerated dose (MTD) of IPI-504 in GIST and STS patients who have failed prior therapies
  • Recommend a dose for subsequent studies of IPI-504

Detailed Description

IPI-504 is a novel, water-soluble analog of 17-AAG and a potent inhibitor of Hsp90. Hsp90's role in the cell is to control the proper folding, function, and viability of various "client" proteins. Many of these client proteins (such as AKT, Her-2, Bcr-Abl, PDGFR-α, and c-Kit) are oncoproteins or important cell signaling proteins. In patients with GIST, mutations in the tyrosine kinase receptor Kit play a critical role in the pathogenesis of this disease. Inhibition of Kit signaling with the tyrosine kinase inhibitor Imatinib (IM) is a very effective treatment for GIST patients. However, new mutations arise in Kit conferring resistance to IM treatment which results in disease progression. Kit is a client protein of Hsp90 and is sensitive to IPI-504. In Soft Tissue Sarcomas, there may be genetic abnormalities that lead to the expression of certain proteins that drive the growth of cancer. These cancer-driving proteins may be stimulated by HSP90. This provides a scientific rationale for Phase 1 clinical testing of IPI-504 in patients with advanced GIST and STS who have failed prior therapies.

Registry
clinicaltrials.gov
Start Date
December 2005
End Date
November 2010
Last Updated
15 years ago
Study Type
Interventional
Study Design
Parallel
Sex
All

Investigators

Eligibility Criteria

Inclusion Criteria

  • Pathologically confirmed diagnosis of GIST or STS
  • Failed prior therapies
  • ECOG performance status of 0-2
  • Ability to adhere to the study visit schedule and all protocol requirements

Exclusion Criteria

  • Previous treatment with 17-AAG, DMAG, or other known Hsp90 inhibitor
  • Participation in any investigational drug study or treatment with any other kinase inhibitor therapy within 2 weeks preceding start of treatment
  • Concurrent radiation therapy is not permitted
  • Concurrent treatment with any agent that alters CYP3A activity
  • Concurrent treatment with any agent that may prolong the QTc interval
  • Myocardial infarction or active ischemic heart disease within 6 months
  • History of arrhythmia
  • Baseline QTc \>450
  • Grade 3 or greater peripheral neuropathy
  • Renal insufficiency, serum creatinine \>1.5 x ULN

Arms & Interventions

1

Schedule A: Doses occur on Days 1, 4, 8, and 11 followed by 10 days with no study drug administration.

Intervention: IPI-504

2

Schedule B: Doses occur on Days 1, 4, 8, 11, 15, and 18 (twice weekly for 3 weeks continuously).

Intervention: IPI-504

Outcomes

Primary Outcomes

To recommend a dose for subsequent studies of IPI-504

Time Frame: 18 months

To determine the safety and maximum tolerated dose (MTD) of IPI-504 in GIST and STS patients who have failed prior therapies

Time Frame: 18 months

Secondary Outcomes

  • To examine the pharmacokinetic (PK) parameters of IPI-504 in GIST and STS patients(18 months)
  • To assess in a preliminary way the potential anti-tumor activity of IPI-504 in GIST and STS.(18 months)
  • To explore potential pharmacodynamic (PD) markers of biologic activity of IPI-504 in GIST and STS.(18 months)

Study Sites (4)

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