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Feasibility of Switching Fluoropyrimidine Due to Cardiotoxicity Study

Conditions
Solid Tumor
Registration Number
NCT04260269
Lead Sponsor
Helsinki University Central Hospital
Brief Summary

The purpose of the present study is to evaluate cardiotoxicity during re-challenge of a different modality of fluoropyrimidine (primary end-point S-1 and secondary any other fluoropyrimidine) after having perceived cardiotoxicity with a fluoropyrimidine based regimen previously. The patient population is being treated for solid tumors.

Detailed Description

Fluoropyrimidine chemotherapy agents, such as 5-fluorouracil and capecitabine, are occasionally associated with cardiotoxicity that may manifest as chest pain, ECG alterations, cardiac arrhythmia, and rarely myocardial infarction and sudden death. Clinical fluoropyrimidine cardiotoxicity is infrequent (1-8% of patients), but subclinical toxicity may be much more common (up to one third of patients). The underlying mechanisms are not well understood, but they may include abnormal coronary artery contractility or spasm, and myocardial toxicity. Cardiotoxicity may be less frequent with S-1 (a combination of tegafur, gimeracil and oteracil at a molar ratio of 1:0.4:1) as compared with 5-fluorouracil and capecitabine, but head-to-head comparisons are lacking.

Anecdotal evidence suggests that patients who have cardiotoxicity on other fluoropyrimidines may be successfully treated with S-1. The purpose of this retrospective study is to compare different 5-fluorouracil-based dosing modalities and S-1, and compare cardiotoxicity during these treatments.

The patient population was treated for solid tumors with a 5-fluorouracil based regimen and had a cardiac event grade 1-4. All patients were re-challenged with a different fluoropyrimidine or S-1 and assessed for cardiotoxicity during re-challenge.

Recruitment & Eligibility

Status
ENROLLING_BY_INVITATION
Sex
All
Target Recruitment
200
Inclusion Criteria
  • Solid tumor
  • Cardiotoxicity grade 1-4 during fluoropyrimidine-based treatment
  • Re-challenge with a different fluoropyrimidine-based therapy
Exclusion Criteria

• Participation in a trial with experimental drugs

Study & Design

Study Type
OBSERVATIONAL
Study Design
Not specified
Primary Outcome Measures
NameTimeMethod
Recurrence of fluoropyrimidine related cardiac toxicity after switch to S-1 based treatmentAfter switch to and during one line of S-1 based chemotherapy (average 6 months)

Cardiac tolerability according to NCI-CTCAE following cardiotoxicity initiated switch of fluoropyrimidine to S-1

Secondary Outcome Measures
NameTimeMethod
Recurrence of fluoropyrimidine related cardiac toxicity after switch to any fluoropyrimidineAfter switch to and during one line of another fluoropyrimidine regimen (average 6 months)

Cardiac tolerability according to NCI-CTCAE following cardiotoxicity initiated switch of fluoropyrimidine to another fluoropyrimidine chemotherapy

Cardiac symptoms during fluoropyrimidine chemotherapyDuring one line of fluoropyrimidine based chemotherapy (average 6 months)

Frequency and severity according to NCI-CTCAE of cardiac symptoms during different fluoropyrimidines and the correlation with other added cytotoxics or biologics

Diagnostic work-upDuring one line of fluoropyrimidine based chemotherapy (average 6 months)

Diagnostic work-up for cardiotoxicity in real world data

Time-lines for cardiotoxicityDuring one line of fluoropyrimidine based chemotherapy (average 6 months)

Time-lines for appearance of cardiotoxicity during fluoropyrimidine-based chemotherapy

Dose-intensityDuring one cycle (average 3 weeks) of fluoropyrimidine-based chemotherapy causing cardiac toxicity

Dose-intensity of the therapy at the cycle causing cardiotoxicity

Alteration in cardiac functional parameters during fluoropyrimidine treatment induced cardiotoxicityDuring one cycle (average 3 weeks) of fluoropyrimidine-based chemotherapy causing cardiac toxicity

The alterations of (if evaluated), graded as normal, non-significant abnormalities or significant abnormalities.:

* ECG abnormalities

* Ejection fraction in %

* Coronary artery status on angiogram

* Cardiac arrhythmias in ECG, Holter or cardiac monitor registration

* Plasma troponin concentration and other cardiac enzymes and other laboratory tests as within reference range ro abnormal

* Serum alpha-fluoro-beta-alanine (FBAL) concentration

Trial Locations

Locations (13)

Department of Oncology

🇫🇮

Tampere, Pirkanmaa, Finland

Turku university hospital

🇫🇮

Turku, Finland

Academic Medical Center

🇳🇱

Amsterdam, Netherlands

Landspitali

🇮🇸

Reykjavík, Iceland

Sundsvall hospital

🇸🇪

Sundsvall, Sweden

Odense University Hospital

🇩🇰

Odense, Denmark

Karolinska University Hospital

🇸🇪

Stockholm, Sweden

Haukeland University Hospital

🇳🇴

Bergen, Norway

Skone university hospital

🇸🇪

Lund, Sweden

Helsinki University Central Hospital

🇫🇮

Helsinki, Uusimaa, Finland

Oulu university hospital

🇫🇮

Oulu, Finland

St. Vincents University Hospital

🇮🇪

Dublin, Ireland

Uppsala academic hospital

🇸🇪

Uppsala, Sweden

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