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The Effect of Addition of Metformin to SGLT2 In Diabetic Patients With Heart Failure With Preserved Ejection Fraction

Phase 2
Not yet recruiting
Conditions
Heart Failure With Preserved Ejection Fraction
Diabete Type 2
Interventions
Registration Number
NCT06080802
Lead Sponsor
October University for Modern Sciences and Arts
Brief Summary

a prospective open-label, randomized controlled study to evaluate the efficacy of the addition of metformin to SGLT2 in diabetic patient with preserved ejection fraction

Detailed Description

Regardless of the benefits noted with SGLT2is, metformin is recommended as first-line therapy for glycemic control in individuals with T2DM and HF, including HFpEF, with estimated glomerular filtration rates (eGFRs) ≥30 mL/min/1.73 m2. This is based on the demonstrated experience with long-term use; its safety, low cost, and low side effect profile; as well as observational (not clinical trial) data suggesting a 20% relative risk reduction in mortality in individuals with HF, including HFpEF.

Nevertheless, it is worth mentioning that Metformin is a common anti-diabetic drug with both systemic and cardioprotective benefits in addition to its hypoglycaemic effect. At the cellular level metformin activates adenosine monophosphate-activated protein kinase (AMPK) an important regulator of several metabolic pathways resulting in enhanced glucose utilisation, reduction of protein synthesis and improvement of mitochondrial function. Furthermore, metformin has been shown to reduce collagen accumulation and potentially reduce LV hypertrophy and improve diastolic function in the diabetic myocardium. The cardio protection afforded by metformin treatment seems to result from interference with TGF-beta signaling pathway and activation of the AMP-kinase signaling cascade. A recent systematic review and meta regression analysis have shown that metformin treatment was associated with a reduction in mortality in patients with HFpEF. In addition, treatment with metformin of non-diabetic metabolic syndrome patients with diastolic dysfunction, on top of lifestyle counseling, was associated with improved diastolic function.

Nevertheless, a recent met analysis showed that initial SGLT2 inhibitor/metformin combination therapy has glycaemic and weight benefits compared with either agent alone and appears relatively safe. High dose SGLT2 inhibitor/metformin combination therapy appears to have modest weight, but no glycaemic benefits compared with the low dose combination therapy.

based on that we our aim is to evaluate the efficacy of the addition of metformin to SGLT2 in diabetic patient with preserved ejection fraction

Recruitment & Eligibility

Status
NOT_YET_RECRUITING
Sex
All
Target Recruitment
80
Inclusion Criteria

Age of 40 years to 74 years. HFpEF (≥ 50%) Written informed consent of the subject to participate in the study. New York Heart Association functional class I-IV. Diabetic patients SGL-2 naive. Newly diagnosed heart failure of preserved ejection fraction

Exclusion Criteria

Patients with heart failure with reduced ejection fraction (< 40%) Age less than 40 and more than 74 GFR < 30 mL/min A1c > 9 Known allergy to metformin End- stage liver disease Cancer Pregnancy or lactation

Study & Design

Study Type
INTERVENTIONAL
Study Design
PARALLEL
Arm && Interventions
GroupInterventionDescription
interventionMetforminLifestyle counseling plus standard evidence-based therapy for HFpEF (SGLT2i/ARBs/MRA/ +/- diuretics)+ metformin
Primary Outcome Measures
NameTimeMethod
Hospitalization ratebaseline, 3 and 6 months

Hospitalization rate

HRQOL using Minnesota Living with Heart Failure Questionnaire for quality-of-life evaluation (MLFHQ)baseline, 3 and 6 months

HRQOL using Minnesota Living with Heart Failure Questionnaire for quality-of-life evaluation (MLFHQ)

Secondary Outcome Measures
NameTimeMethod
The change in the mean early diastolic mitral annular velocity (mean e'), at 3 and 6 monthsbaseline, 3 and 6 months

The change in the mean early diastolic mitral annular velocity (mean e'), at 3 and 6 months

adverse drug effectsbaseline, 3 and 6 months

adverse drugs effects

Neutrophil/lymphocyte ratio -AMPK pathwaybaseline, 3 and 6 months

Neutrophil/lymphocyte ratio

-AMPK pathway

Change in N-terminal pro-BNP (NT-proBNP)baseline, 3 and 6 months

Change in N-terminal pro-BNP (NT-proBNP)

Change in body weightbaseline, 3 , 6 months

Change in body weight

Inflammatory and oxidative stressbaseline, 3 and 6 months

Inflammatory and oxidative stress

Trial Locations

Locations (1)

clinical research uint- El-sheikh zayed specialized hospital SMC- Egyptian Ministry of health

🇪🇬

Cairo, Egypt

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