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Clinical Trials/NCT00395200
NCT00395200
Completed
Phase 1

Autologous Adult Human Mesenchymal Stem Cells: a Neuroprotective Therapy for Multiple Sclerosis

University of Cambridge2 sites in 1 country10 target enrollmentJuly 2008

Overview

Phase
Phase 1
Intervention
Not specified
Conditions
Multiple Sclerosis
Sponsor
University of Cambridge
Enrollment
10
Locations
2
Primary Endpoint
Adverse events
Status
Completed
Last Updated
14 years ago

Overview

Brief Summary

Hypothesis: Intravenous administration of bone marrow-derived autologous adult human mesenchymal stem cells is a safe novel therapeutic approach for patients with multiple sclerosis.

Mesenchymal Stem Cells in Multiple Sclerosis (MSCIMS) is a phase I/IIA trial designed to establish the safety of intravenous administration of bone marrow-derived autologous adult human mesenchymal stem cells to patients with multiple sclerosis.

Detailed Description

Disease under investigation: Multiple Sclerosis Phase: I/IIA Number of patients: 10 Design: 18 month cross over, single treatment at 6 months Intervention: Administration of bone marrow-derived autologous mesenchymal stem cells Route of administration: Intravenous Dose: Up to 2,000,000 Mesenchymal Stem Cells per kilogram Source of patients: Referrals accepted from Neurologists in East Anglia and North London, UK Referral Criteria: (all 3 required) 1. Clinically definite multiple sclerosis 2. Expanded Kurtzke Disability Status Score 2.0 - 6.5 (inclusive) 3. Evidence of optic nerve damage by * history of optic neuritis, or * relative afferent pupillary defect, or * optic atrophy on fundoscopy, or * abnormal visual evoked potential from either or both eyes suggestive of demyelination Primary Objective: Establish the safety of intravenously administered bone marrow-derived autologous mesenchymal stem cells at a dose of up to 2,000,000 cells/kg over 12 months by monitoring adverse reactions. Secondary Objectives: Explore the efficacy of intravenously administered bone marrow-derived autologous mesenchymal stem cells at a dose of up to 2,000,000 cells/kg over 12 months on visual function by clinical, neurophysiological, and imaging assessments. Outcome Measures: 1. Primary * Adverse events 2. Secondary * Visual function (acuity and colour) * Visual evoked potential latency * Optic nerve Magnetisation Transfer Ratio * Retinal nerve fibre layer thickness (by optical coherence tomography) * Brain lesion Magnetisation Transfer Ratio * MRI brain T1 hypointensity load * T cell response suppression 3. Tertiary * Multiple Sclerosis Functional Composite Score * Expanded Kurtzke Disability Status Score

Registry
clinicaltrials.gov
Start Date
July 2008
End Date
December 2010
Last Updated
14 years ago
Study Type
Interventional
Study Design
Single Group
Sex
All

Investigators

Responsible Party
Principal Investigator
Principal Investigator

Peter Connick

Research Associate

University of Cambridge

Eligibility Criteria

Inclusion Criteria

  • Clinically definite multiple sclerosis
  • Expanded Kurtzke Disability Status Score 2.0 - 6.5 (inclusive)
  • Evidence of optic nerve damage by:
  • history of optic neuritis, or
  • relative afferent pupillary defect, or
  • optic atrophy on fundoscopy, or
  • abnormal visual evoked potential from either or both eyes suggestive of demyelination
  • Prolonged visual evoked potential P100 latency with preserved waveform
  • T2 lesion on MRI optic nerve
  • Retinal nerve fibre layer thickness on optical coherence tomography \> 40 microns

Exclusion Criteria

  • Age \< 18 years
  • Age \> 65 years
  • Patient lacks capacity to give informed consent
  • Presence of a severe bleeding disorder
  • Planning a pregnancy during the trial period
  • Current MS disease modifying therapy

Outcomes

Primary Outcomes

Adverse events

Time Frame: 0,1,2,3,4,12 and 52 weeks post treatment

Secondary Outcomes

  • Visual function (acuity and colour)(12 and 52 weeks post treatment)
  • Visual evoked potential latency(12 and 52 weeks post treatment)
  • Optic nerve Magnetisation Transfer Ratio(12 and 52 weeks post treatment)
  • Retinal nerve fibre layer thickness (by optical coherence tomography)(12 and 52 weeks post treatment)
  • Brain lesion Magnetisation Transfer Ratio(12 and 52 weeks post treatment)
  • MRI brain T1 hypointensity load(12 and 52 weeks post treatment)
  • Multiple Sclerosis Functional Composite Score(12 and 52 weeks post treatment)
  • Expanded Kurtzke Disability Status Score(12 and 52 weeks post treatment)

Study Sites (2)

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