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Clinical Trials/NCT04411862
NCT04411862
Completed
Phase 3

Efficacy of Phosphatidylcholine in Addition to Behavior Therapy by Clinical Pharmacist in the Management of Non Alcoholic Fatty Liver (NAFLD)

Nehal Abou Seada0 sites100 target enrollmentJanuary 2, 2016

Overview

Phase
Phase 3
Intervention
Not specified
Conditions
Non-Alcoholic Fatty Liver Disease
Sponsor
Nehal Abou Seada
Enrollment
100
Primary Endpoint
change from baseline Body Mass Index (BMI) at 3 and 6 month
Status
Completed
Last Updated
5 years ago

Overview

Brief Summary

This study evaluates efficacy of Phosphatidylcholine in addition to life style modification and patient health education by clinical Pharmacist in the Management of Non Alcoholic Fatty Liver NAFLD. All participants with NAFLD will receive life style intervention and half of them will receive additionally Phosphatidylcholine.

Detailed Description

As a result of increasing rates of obesity Non Alcoholic Fatty Liver (NAFLD) is the most common liver disorder affecting 17-46% of adults and parallels the prevalence of Metabolic Syndrome (MetS) and its components which also increases the risk of more advanced disease both in adults and in children. Its pathogenesis is complex and multifactorial, mainly involving genetic, environmental and metabolic factors. New concepts are constantly appearing in the literature, promising new diagnostic and therapeutic tools. Further studies are needed to better characterize not only NAFLD development but overall NAFLD progression, in order to better identify NAFLD patients at higher risk of metabolic, cardiovascular and neoplastic complications. Pharmacological treatments aimed primarily at improving liver disease should generally be limited to those with biopsy-proven Nonalcoholic steatohepatitis (NASH) and liver fibrosis. Not much therapeutic options for NAFLD are accepted until today besides correction of obesity with hypocaloric diets and physical exercise and controlling hyperglycemia with diet, insulin, or oral hypoglycemic agents. Weight loss generally reduces hepatic steatosis.Essential phospholipid (EPL) as a nutritional supplement is one of the drugs under discussion with significant positive effects as antioxidative, antifibrotic effects and high biocompatibility on NAFLD.

Registry
clinicaltrials.gov
Start Date
January 2, 2016
End Date
July 1, 2019
Last Updated
5 years ago
Study Type
Interventional
Study Design
Parallel
Sex
All

Investigators

Sponsor
Nehal Abou Seada
Responsible Party
Sponsor Investigator
Principal Investigator

Nehal Abou Seada

Principal Investigator

Ain Shams University

Eligibility Criteria

Inclusion Criteria

  • fatty liver upon Ultrasonography (US) /Computed Tomography (CT) /Magnetic Resonance Imaging (MRI) with either incidental increased Alanine Aminotransferase (ALT)
  • the presence of risk factors related to NAFLD + increased ALT
  • symptomatic liver disease +/- hepatomegaly, +/- increased ALT
  • homeostasis model assessment-insulin resistance HOMA IR score \> 3
  • presence of liver steatosis or stiffness measured by transient elastography
  • eligible patients had at least one of the following metabolic comorbidities: hypertension, Type 2 Diabetes Mellitus, overweight/obesity (BMI\>27 kg/m2) serum cholesterol of \> 200 mg/d
  • Patients were excluded from the study if showing evidence :

Exclusion Criteria

  • if showing evidence of alcoholic or chronic liver disease
  • Hepatocellular Carcinoma, autoimmune hepatitis
  • end stage liver disease
  • treatment with other hepatoprotectants
  • other concomitant EPL within 30 days of study initiation
  • pregnancy or lactation

Outcomes

Primary Outcomes

change from baseline Body Mass Index (BMI) at 3 and 6 month

Time Frame: baseline, at 3 and 6 month

person's weight in kilograms divided by the square of the person's height in metres (kg/m2).

change from baseline liver stiffness at 3 and 6 month

Time Frame: baseline , at 3 and 6 month

Liver Stiffness and fibrosis score measured by Transient elastography (Fibroscan) F0 = no fibrosis F1 = portal fibrosis without septa F2 = portal fibrosis with few septa F3 = numerous septa without cirrhosis F4 = cirrhosis

change from baseline Lipid Profile

Time Frame: baseline , at 3 and 6 month

Total cholesterol ,Triglyceride ,Low Density Lipoprotein ,High Density Lipoprotein

change from baseline Oxidative stress markers

Time Frame: baseline , at 3 and 6 month

malonaldehyde (MDA) as an index of lipid peroxidation by colorimetric assay

change from baseline NAFLD score at 3 and 6 month

Time Frame: baseline , at 3 and 6 month

NAFLD Fibrosis Score is based on six readily available variables (age, BMI, hyperglycemia, albumin, platelet count, AST/ALT ratio) and it is calculated using published formula (http: //naflds- core.com) . A low cutpoint (score \< -1.455) signified the absence of advanced fibrosis, whereas a high cutpoint (score\> 0.676) identified advanced fibrosis.

change from baseline homeostasis model assessment Insulin resistance HOMA IR scores at 3 and 6 month

Time Frame: baseline , at 3 and 6 month

HOMA IR scores \<3 normal HOMA IR scores \>5 severe insulin resistance 3 to 5 moderate insulin resistance

Secondary Outcomes

  • change from baseline Complete Blood Picture at 3 and 6 month(baseline , at 3 and 6 month)

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