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Effects of Cetoleic Acid on Atherosclerosis (Ketolinsyre's Effekt på Aterosklerose)

Not Applicable
Recruiting
Conditions
Metabolic Syndrome
Interventions
Dietary Supplement: Control oil
Dietary Supplement: Cetoleic acid
Registration Number
NCT06172335
Lead Sponsor
Oslo University Hospital
Brief Summary

In this Randomized Controlled Trial (RCT) we want to study the effect of an oil with high concentrations of cetoelic acid (C22:n1-11) (intervention) compared to supplements with a low concentration of cetoleic acid (control), but with equivalent content of EPA og DHA, on plasma levels of epa and dha as well as atherosclerotic markers, glucose, c-peptide and triglycerides in a patient group with a metabolically unfavorable phenotype.

Our primary endpoints are changes in the concentration of EPA and DHA in plasma.

Detailed Description

This is a randomized double-blinded controlled trial (randomized 1:1). Study population: men and women 20-70 years with a metabolically unfavorable phenotype defined as: triglycerides \> 1.7 mmol/L and waist measurement \> 80 cm (women) and \> 94 cm (men).

Study design:

* 3 weeks run-in-period where all participants consume control capsules every morning.

* Randomization intervention: control (1:1). All participants consume their capsules (control or intervention) for 4 weeks.

The intervention oil is consists of an oil high in cetoleic acid and the control oil is low in cetoleic acid. Both the intervention oil and the control oil are a mix of different oils; fish oils, olive oil, "high-oleic sunflower oil" and rapeseed oil so that the content of EPA, DHA and ALA is similar in the two oils.

Power calculation and sample size:

It was expected a difference of 15% in n-3 between the groups after the intervention (Østbye et al. 2019, doi:10.1017/S0007114519001478).

The level of significance was set to 5% (two-sided) and the power to 80%. A total of thirty-eight subjects were required to participate in the study, but a high dropout rate was expected (20%) and it was considered necessary to include a total of seventy (n=70) subjects (thirty-five per arm).

Recruitment & Eligibility

Status
RECRUITING
Sex
All
Target Recruitment
70
Inclusion Criteria
  • Triglycerides > 1.7 mmol/L
  • Waist measurement > 80 cm (women) and > 94 cm (men)
Exclusion Criteria
  • Chronic disease (liver/kidney/metabolism)
  • Ongoing active cancer treatment
  • Excessive alcohol consumption (>40g/day)
  • Pregnant/breastfeeding or planned pregnancy during the intervention
  • High intake of fish (>3 weekly meals)
  • Level of free thyroxine (T4) and triiodothyronine (T3) outside reference ranges.
  • Hypertension (≥ 160/ 100 mmHg)
  • Total cholesterol > 7.8 mmol/L
  • Blood donation during the intervention period
  • Difficulty following the protocol
  • Smoking or sniffing
  • Regular use (> 1 day/week) of anti-inflammatory drugs
  • Regular use of omega-3 supplements/cod liver oil
  • Drug use other than stable use of statins, hypertension drugs (Ca antagonists, diuretics and beta blockers).
  • Hormonal treatment excluding stable use of thyroxine and birth control pills/contraceptive rod

Study & Design

Study Type
INTERVENTIONAL
Study Design
PARALLEL
Arm && Interventions
GroupInterventionDescription
Control oilControl oil6 x capsules control oil (oil= mix of fish oils, olive oil, high-oleic sunflower oil and rapeseed oil) with low content of cetoleic acid (35 mg/day, estimated 0.58%) every morning for 4 weeks.
Intervention (Cetoleic acid)Cetoleic acid6 x capsules intervention oil (oil = mix of fish oils, olive oil, high-oleic sunflower oil and rapeseed oil) with high content of cetoleic acid (1780 mg/day, estimated: 29,76%) every morning for 4 weeks.
Primary Outcome Measures
NameTimeMethod
EPA and DHA in plasma4 weeks intervention

EPA and DHA concentration in plasma measured at baseline and after 4 week intervention (and as a "control measurement" at the screening visit)

Secondary Outcome Measures
NameTimeMethod
Inflammatory markers4 weeks intervention

The concentration of circulating levels of inflammatory markers

Resolvin4 weeks intervention

Blood levels of resolvin (omega- 3 derivates) concentration

Lipid profile4 weeks intervention

Plasma levels of triglycerides, total cholesterol, LDL cholesterol, HDL cholesterol, apolipoproteins (apoB and apoA), and Lp(a)

Glucose4 weeks intervention

Serum levels of glucose

C-peptid4 weeks intervention

serum levels of C-peptid

Gene expression, metabolome and lipidome4 weeks intervention

Changes in PBMC (peripheral blood mononuclear cell) gene expression profile. Changes in plasma metabolome and lipidome profile. Changes in PBMC epitranscriptome, as a regulator of the gene expression profile

Trial Locations

Locations (1)

Oslo University Hospital

🇳🇴

Oslo, Norway

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