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Ansa Cervicalis and Hypoglossal Nerve Stimulation in OSA

Not Applicable
Recruiting
Conditions
Obstructive Sleep Apnea
Interventions
Device: Grass S88 Muscle Stimulator
Registration Number
NCT05501236
Lead Sponsor
Vanderbilt University Medical Center
Brief Summary

Polysomnography (PSG) and drug-induced sleep endoscopy (DISE) are widely used diagnostic studies for assessing obstructive sleep apnea (OSA) severity and collapse patterns of the upper airway anatomy during sleep. Hypoglossal nerve stimulation (HNS) therapy for obstructive sleep apnea suffers from variable response at the level of the soft palate. The Investigators propose a study examining the physiologic effect of ansa cervicalis stimulation (ACS) alone and in combination with HNS during PSG and DISE.

Detailed Description

Obstructive Sleep Apnea (OSA) is a common disorder characterized by repetitive upper airway collapse during inspiration caused, in part, by a loss of neuromotor tone in specific upper airway muscles, with multiple associated health sequelae impacting millions of Americans. Patient adherence to the reference treatment, positive airway pressure (PAP), remains problematic. Despite the recent promising development of hypoglossal nerve stimulation (HNS) as a surgical therapy, its indications are limited and a proportion of eligible patients do not achieve sufficient response, leaving a critical unmet need for effective therapeutic alternatives to PAP.

This project challenges the long-held concept that the genioglossus muscle is primarily responsible for the maintenance of pharyngeal patency during sleep and proposes a novel therapeutic mechanism. It is built upon strong evidence that caudal pharyngeal traction from the trachea has a marked impact on pharyngeal patency primarily mediated through changes in lung volume. Contraction of the sternothyroid muscle, an infrahyoid cervical strap muscle that inserts onto the thyroid cartilage, also generates caudal pharyngeal traction. Our data suggest that ansa cervicalis stimulation (ACS) of the sternothyroid muscle unfolds and stretches the lateral pharyngeal walls and tensions the distal edge of the soft palate caudally, increasing airway patency.

The major hypothesis of the Investigators is that ACS overcomes specific anatomic and neuromuscular defects of upper airway control that restore pharyngeal patency in patients with OSA. This hypothesis is supported by published and preliminary data demonstrating that: (1) the degree of end-expiratory lung volume decrease in sleep correlates with observed increases in pharyngeal collapsibility, and (2) unilateral ACS increases maximum inspiratory airflow and velopharyngeal cross-sectional area during flow-limited breathing in sedated humans. These findings suggest that (3) tracheal traction, as mediated by end-expiratory lung volume (EELV), is a major contributor to airway patency in sleep. In this project, the Investigators will elucidate specific mechanisms for control of pharyngeal patency with caudal traction during drug-induced sleep endoscopy (DISE) and natural sleep (PSG). The Investigators will address these aims by characterizing (1) the effects of ACS of the sternothyroid muscle(s) on upper airway pressure-area and pressure-flow relationships, and (2) determine how subject anatomic, physiologic, and polysomnographic characteristics modulate these responses.

Recruitment & Eligibility

Status
RECRUITING
Sex
All
Target Recruitment
100
Inclusion Criteria
  1. Consenting adults with BMI≥ 25 and ≤ 40 kg/m2
  2. Obstructive sleep apnea with an AHI between 20 and 80 events/hr (with hypopneas defined by 4% oxyhemoglobin desaturations); ≥80% obstructive events.
Exclusion Criteria
  1. Chronic use of opiate medications, illicit drug use, or alcohol dependency
  2. Other known concomitant sleep disorder (e.g., central sleep apnea, periodic limb movements, narcolepsy)
  3. Clinical history or evidence of cardiopulmonary disease (or oxygen use), liver, renal, immunodeficiency, neurodegenerative diseases, or previous adverse reactions to anesthesia.
  4. Prior upper airway reconstructive surgery excluding tonsillectomy (e.g., cleft palate repair, uvulopalatopharyngoplasty)
  5. Indwelling neurostimulation device (e.g. cardiac pacemaker, spinal, vagal, or hypoglossal nerve stimulator)

Study & Design

Study Type
INTERVENTIONAL
Study Design
SINGLE_GROUP
Arm && Interventions
GroupInterventionDescription
Muscle stimulationGrass S88 Muscle StimulatorConsented participants who meet eligibility will have a drug induced sleep endoscopy (DISE) and second sleep study and the Grass S88 (or comparable) muscle stimulator.
Primary Outcome Measures
NameTimeMethod
Basic physiologic measurements during Drug Induced Sleep Endoscopy (DISE) - Airway cross sectional diameterDuring DISE, approximately 15 minutes

Airway cross-sectional diameter (mm\^2) will be measured throughout the operative procedure via flexible fiberoptic nasopharyngoscopy.

Basic physiologic measurements during Polysomnography (PSG) - Airflow dataDuring sleep study exam (PSG), approximately 8 hours

Airflow data (L/min) will be measured during the sleep study via a pneumotachometer applied to the nose.

Basic physiologic measurements during Polysomnography (PSG) - Electroocoulogram (EOG)During sleep study exam (PSG), approximately 8 hours

EOG (mV) will be collected during the sleep study via skin surface electrodes.

Basic physiologic measurements during Polysomnography (PSG) - Respiratory effort dataDuring sleep study exam (PSG), approximately 8 hours

Respiratory effort data (mV) will be collected during the sleep study via respiratory inductance plethysmography.

Basic physiologic measurements during Drug Induced Sleep Endoscopy (DISE) - Airflow dataDuring DISE, approximately 15 minutes

Airflow data (L/min) will be measured throughout the operative procedure via a pneumotachometer applied to the nose.

Basic physiologic measurements during Polysomnography (PSG) - Electroencephalogram (EEG)During sleep study exam (PSG), approximately 8 hours

EEG (mV) will be collected during the sleep study via skin surface electrodes.

Basic physiologic measurements during Polysomnography (PSG) - Video dataDuring sleep study exam (PSG), approximately 8 hours

Video data will be collected during the sleep study via in-room camera.

Basic physiologic measurements during Drug Induced Sleep Endoscopy (DISE) - Upper airway pressure changesDuring DISE, approximately 15 minutes

Upper airway pressure changes (cmH20) will be measured throughout the operative procedure via a pneumotachometer applied to the nose.

Basic physiologic measurements during Drug Induced Sleep Endoscopy (DISE) - Respiratory effort dataDuring DISE, approximately 15 minutes

Respiratory effort data (mV) will be measured throughout the operative procedure via two respiratory inductance plethysmography belts.

Basic physiologic measurements during Polysomnography (PSG) - Electrocardiogram (EKG)During sleep study exam (PSG), approximately 8 hours

EKG (mV) will be collected during the sleep study via skin surface electrodes.

Basic physiologic measurements during Polysomnography (PSG) - Electromyography (EMG)During sleep study exam (PSG), approximately 8 hours

EMG data (mV) will be collected during the sleep study via skin surface electrodes.

Secondary Outcome Measures
NameTimeMethod
Amount of current needed for adequate stimulationCollected during operative and sleep study procedures, taking about 15 minutes.

Obtain preliminary data regarding including the amount of current needed to adequately stimulate the ansa cervicalis stimulation (ACS) alone and in combination with hypoglossal nerve stimulation (HNS) during PSG and DISE via a neurostimulator connected to percutaneous electrodes.

Trial Locations

Locations (1)

Vanderbilt University Medical Center

🇺🇸

Nashville, Tennessee, United States

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