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Pregnancy, Arsenic and Immune Response

Completed
Conditions
Vaccine Response Impaired
Micronutrient Deficiency
Influenza
Arsenic--Toxicology
Immunologic Disorders Complicating Pregnancy
Registration Number
NCT03930017
Lead Sponsor
Johns Hopkins Bloomberg School of Public Health
Brief Summary

As the global availability of vaccines increases, and reaches areas disproportionately affected by arsenic and malnutrition, resolving questions about potential environmental and biologic barriers to maternal immunization has become increasingly urgent. It is not known whether arsenic, a known developmental toxicant, can alter maternal immune responses to vaccination and whether exposure to arsenic during pregnancy can impair the transfer of maternal vaccine-induced antibody to the newborn. Moreover, factors known to affect arsenic metabolism and toxicity outcomes, particularly micronutrients critical in one-carbon metabolism, have not been evaluated in studies of arsenic immunotoxicity and vaccine-induced protection in mothers and their newborns.

The objective in this study is to investigate whether maternal arsenic exposure and one-carbon metabolism micronutrient deficiencies alter maternal and newborn measures of vaccine-induced protection, respiratory morbidity, and systemic immune function following influenza vaccination during pregnancy.

Detailed Description

The objective in this study is to investigate whether maternal arsenic exposure and one-carbon metabolism micronutrient deficiencies alter maternal and newborn measures of vaccine-induced protection, respiratory morbidity, and systemic immune function following influenza vaccination during pregnancy. The hypothesis is that maternal arsenic exposure and one-carbon metabolism micronutrient deficiencies alter maternal and newborn influenza antibody titer and avidity, respiratory infection morbidity, and markers of systemic immune function following maternal influenza vaccination during pregnancy. This study leverages a comprehensive pregnancy surveillance system at the JiVitA Maternal and Child Health and Nutrition Research Project site in Bangladesh (hereafter JiVitA) to pursue the following three aims:

Aim 1. Establish whether arsenic exposure during pregnancy alters maternal and newborn influenza antibody titer and avidity following maternal influenza vaccination.

Aim 2. Determine whether markers of systemic immune function mediate the association between arsenic exposure and respiratory illness in pregnant women and their newborns.

Aim 3. Assess whether arsenic exposure and one-carbon metabolism micronutrient deficiencies during pregnancy have a joint effect on markers of systemic immune function and respiratory illness in mothers and their newborns.

This study will yield three expected outcomes. First, it will fill critical knowledge gaps about whether arsenic exposure and one-carbon metabolism micronutrient deficiencies alter immune responses to a vaccination with known benefits for mothers and their newborns. Second, it will increase understanding of arsenic-associated respiratory morbidity and specific immune function pathways between arsenic exposure and respiratory morbidity in mothers and their newborns. Finally, as the global availability of vaccines increases, improving knowledge of potential environmental and biologic barriers to maternal and newborn vaccine-induced protection could lead to improved vaccine regimens (targeted vaccination campaigns, higher vaccine doses, and/or additional booster immunizations) to restore vaccine-induced protection in arsenic-exposed and malnutrition-affected populations of pregnant women and newborns worldwide.

Recruitment & Eligibility

Status
COMPLETED
Sex
Female
Target Recruitment
784
Inclusion Criteria

Women who:

  • are within 13-16 weeks of gestational age (GA) of pregnancy;
  • are between 13 and 45 years of age;
  • are married;
  • provide informed consent for herself and assent for her unborn child;
  • agree to receive the seasonal influenza vaccine (VAXIGRIP® TETRA seasonal quadrivalent inactivated influenza vaccine, Sanofi Pasteur) upon study enrollment.
Exclusion Criteria

Women who:

  • have pre-existing immune-related health condition (e.g., immunodeficiency, lupus, chronic infection, or cancer);
  • previous or current use of immune-altering drug/therapy (e.g., steroids);
  • have already received influenza vaccination for the current season.

Study & Design

Study Type
OBSERVATIONAL
Study Design
Not specified
Primary Outcome Measures
NameTimeMethod
Change in geometric mean HI antibody titer (GMT)Comparing baseline to 28 days post vaccination, birth, and 3 months post-partum

GMT HI antibody titers will be transformed to binary logarithms, and original values will be divided by 4 (undetectable titer) to set the starting point of the log scale to zero prior to transformation. We will calculate average log2 GMT antibody titers.

Change in anti-influenza virus total immunoglobulin G (IgG) enzyme immunoassayComparing baseline to 28 days post vaccination, birth, and 3 months post-partum

Total IgG antibodies to influenza virus as measured in serum or plasma by enzyme immunoassay

Change in influenza virus neutralizing antibody titerComparing baseline to 28 days post vaccination, birth, and 3 months post-partum

Virus neutralization is measured as a titer calculated based on the highest serum dilution that eliminates virus.

Change in influenza hemagglutination-inhibition (HI) antibody titerComparing baseline to 28 days post vaccination, birth, and 3 months post-partum

Influenza hemagglutination-inhibition (HI) antibody titer will be measured in participant's serum.

Mean percent influenza virus antibody avidityMeasured at baseline, 28 days post vaccination, birth, and 3 months post-partum

The accumulated strength of multiple affinities of individual non-covalent binding interactions of influenza-specific antibodies, including avidity of antibodies to seasonal inactivated influenza virus (IIV) strains included in the formulation in Sanofi Pasteur's 2018-2019 seasonal VAXIGRIP® TETRA vaccine.

Geometric mean ratio of infant:mother HI titerBirth and 3 months post-partum

Ratio of infant to mother HI titer as a measure of transplacental transfer of influenza antibody.

Seroconversion rateDefined as a post-vaccination HI titer of ≥40 given a pre-vaccination titer ≤10 or, alternatively, a ≥4-fold increase in HI titer between pre-vaccination and post-vaccination sera if the pre-vaccination titer was >10.

The proportion of pregnant women demonstrating seroconversion

Secondary Outcome Measures
NameTimeMethod
Laboratory-confirmed influenza (LCI)From date of enrollment visit until date of 3 months postpartum visit, assessed at weekly intervals

Influenza A and/or B virus real-time (RT)-quantitative polymerase chain reaction (qPCR) positive nasal swab from a participant reporting ILI at a weekly mobile phone positive follow-up.

Maternal influenza-like illness (ILI)From date of enrollment visit until date of 3 months postpartum visit, assessed at weekly intervals

Defined as at least one symptom-free day prior to onset of fever \>37.8°C and cough or sore throat.

Infant influenza-like illness (ILI)From date of birth visit until date of 3 months postpartum visit, assessed at weekly intervals

Defined as at least one symptom-free day prior to onset of fever \>37.8°C and cough.

Acute respiratory illness (ARI)From date of enrollment visit until date of 3 months postpartum visit, assessed at weekly intervals

Defined as: cough; rapid breathing or grunting or wheezing, excluding asthma; blood in sputum; ear discharge; low fever; and/or headache. A stand-alone outcome of ARI plus fever will be defined as the above symptoms plus high fever \>37.8°celsius (C).

Trial Locations

Locations (1)

JiVitA Maternal and Child Health and Nutrition Research Program

🇧🇩

Gaibandha, Bangladesh

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