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The Effect of Low Pressure Pneumoperitoneum During Laparoscopic Colorectal Surgery on Innate Immune Homeostasis.

Phase 4
Completed
Conditions
Pneumoperitoneum
Surgery
Immune Suppression
Neuromuscular Blockade
Interventions
Procedure: Low pressure pneumoperitoneum
Registration Number
NCT03572413
Lead Sponsor
Radboud University Medical Center
Brief Summary

Substudy of the RECOVER trial (a randomised controlled trial comparing the effect of low pressure pneumoperitoneum with deep neuromuscular block versus normal pressure pneumoperitoneum with moderate neuromuscular block during laparoscopic colorectal surgery on early quality of recovery) investigating innate immune homeostasis after laparoscopic colorectal surgery.

Detailed Description

Rationale: increased intra-abdominal pressure can cause peritoneal mesothelial cell injury either directly or by compression of the capillary vessels, causing a variable degree of ischemia reperfusion injury. The immune system can identify damage to host cells by recognising Danger-Associated Molecular Patterns (DAMPs) that are released upon cell death in an uncontrolled fashion, such as during surgical trauma. DAMPs elicit an immune response similar to the response to invading pathogens and induce an anti-inflammatory immune response strongly related to postoperative recovery, infectious complications and mortality. Low pressure PNP is associated with lower levels of serum pro- and anti-inflammatory cytokines and better preservation of innate immune function.

Objective: to establish the relationship between the use of low pressure pneumoperitoneum with deep neuromuscular blockade and innate immune function after laparoscopic colorectal surgery.

Study design: a multi-center, blinded, randomized controlled clinical trial.

Study population: adult individuals scheduled for laparoscopic colorectal surgery with a primary colonic anastomosis.

Intervention: participants will be randomly assigned in a 1:1 fashion to either the experimental group (group A): low pressure PNP (8 mmHg) with deep NMB (PTC 1-2) or the control group (group B): normal pressure PNP (12 mmHg) with moderate NMB (TOF count 1-2).

Primary endpoint: mononuclear cell responsiveness ex-vivo as reflected by TNFα release upon LPS stimulation.

Secondary endpoints: mononuclear cell responsiveness ex-vivo as reflected by IL-6, IL-10 and IL-1beta release upon LPS stimulation. Peritoneal mesothelial hypoxia as reflected by peritoneal HIF1α mRNA expression, histological peritoneal mesothelial cell injury and plasma levels of DAMPs and cytokines.

Recruitment & Eligibility

Status
COMPLETED
Sex
All
Target Recruitment
100
Inclusion Criteria
  • Scheduled for laparoscopic colorectal surgery with a primary anastomosis
  • Obtained informed consent
  • Age over 18 years
Exclusion Criteria
  • Insufficient control of the Dutch language to read the patient information and to fill out the questionnaires
  • Primary colostomy
  • Neo-adjuvant chemotherapy
  • Chronic use of analgesics or psychotropic drugs
  • Use of NSAIDs shorter than 5 days before surgery
  • Known or suspected allergy to rocuronium of sugammadex
  • Neuromuscular disease
  • Indication for rapid sequence induction
  • Severe liver- or renal disease (creatinine clearance <30ml/min)
  • BMI >35 kg/m²
  • Deficiency of vitamin K dependent clotting factors or coagulopathy

Study & Design

Study Type
INTERVENTIONAL
Study Design
PARALLEL
Arm && Interventions
GroupInterventionDescription
Low pressure PNP, deep NMBLow pressure pneumoperitoneumLow pressure pneumoperitoneum of 8 mmHg with deep neuromuscular block (post tetanic count of 1-2) reached by titration with continuous infusion of Rocuronium bromide.
Low pressure PNP, deep NMBRocuronium bromideLow pressure pneumoperitoneum of 8 mmHg with deep neuromuscular block (post tetanic count of 1-2) reached by titration with continuous infusion of Rocuronium bromide.
Normal pressure PNP, moderate NMBRocuronium bromideNormal pressure pneumoperitoneum of 12 mmHg with moderate neuromuscular block (TOF count of 1-2) reached by titration with bolus or continuous infusion of a low dose of Rocuronium bromide.
Primary Outcome Measures
NameTimeMethod
Mononuclear cell responsiveness ex vivoBefore surgery, 24 and 72 hours after surgery

TNFalpha release upon LPS stimulation

Secondary Outcome Measures
NameTimeMethod
Mononuclear cell responsiveness ex-vivoBefore surgery, 24 and 72 hours after surgery

IL-6, IL-10 and IL-1beta release upon LPS stimulation

HLA-DR mRNA expressionBefore surgery, 24 and 72 hours after surgery

MHC-II antigen presenting immune receptor

Serum DAMPs and cytokinesBefore surgery, at the end of surgery and 24 hours after surgery

Danger associated molecular patterns, TNFalpha, IL-6 and IL-10

Peritoneal histology and HIF1alpha expressionAt the start and end of surgery

Histology and HIF1alpha mRNA expression of a peritoneal biopsy

Trial Locations

Locations (1)

Canisius Wilhelmina Hospital

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Nijmegen, Gelderland, Netherlands

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