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Dissection of the Gastrointestinal-mediated Glucose Disposal and Incretin Defect in Patients With Type 2 Diabetes

Not Applicable
Completed
Conditions
Type 2 Diabetes
Interventions
Drug: LY2409021 placebo
Procedure: IIGI
Procedure: OGTT
Procedure: Standardised liquid meal
Registration Number
NCT02669524
Lead Sponsor
University Hospital, Gentofte, Copenhagen
Brief Summary

In patients with type 2 diabetes, the incretin effect is markedly reduced contributing to the relative insulin deficiency that characterizes these patients. This defect is believed to be due to a decreased effect of GLP-1 and an almost ceased effect of GIP. Nevertheless, the impact of the defect on glucose tolerance is not fully understood. The so-called gastrointestinal-mediated glucose disposal (GIGD) is a measure of glucose handling, which includes the incretin effect, but also other factors affecting glucose disposal (e.g. glucagon secretion). Interestingly, patients with type 2 diabetes exhibit elevated plasma glucagon levels in the fasting state, and glucagon concentrations fail to decrease appropriately and may even increase in response to ingestion of glucose and show exaggerated increases after a mixed meal. With the current project the investigators wish to elucidate how this paradoxical glucagon response observed in patients with type 2 diabetes affects the GIGD, the incretin effect and postprandial glucose excursions.

Ten patients with type 2 diabetes and 10 healthy matched control subjects will be enrolled in this randomised, placebo-controlled, double-blinded study. The aim is to examine the effect of a glucagon receptor antagonist (GRA) on gastrointestinal-mediated glucose disposal (GIGD), incretin effect and postprandial glucose excursions in patients with type 2 diabetes and healthy controls. Participants will attend two oral glucose tolerance tests (OGTT), two isoglycaemic iv glucose infusion (IIGI) and two standardised liquid meals.

Detailed Description

Not available

Recruitment & Eligibility

Status
COMPLETED
Sex
All
Target Recruitment
20
Inclusion Criteria

Patients with type 2 diabetes

  • Caucasians above 35 years of age with diet or metformin treated type 2 diabetes for at least 3 month (diagnosed according to the criteria of the World Health Organization (WHO)
  • Normal haemoglobin
  • Informed consent

Healthy subjects

  • Normal fasting plasma glucose (FPG) <6.1 mmol/l and HbA1c <42 mmol/mol (6.0%)
  • Normal haemoglobin
  • Age above 35 years
  • Informed consent
Exclusion Criteria

Patients with type 2 diabetes

  • Inflammatory bowel disease
  • Intestinal resections
  • Nephropathy (serum creatinine above normal range and/or albuminuria)
  • Liver disease (serum alanine aminotransferase (ALAT) and/or serum aspartate aminotransferase (ASAT) >2×normal values)
  • Treatment with medicine that cannot be paused for 12 hours
  • Pregnancy and/or breastfeeding
  • Family history of pancreatic islet tumours
  • Age above 80 years

Healthy subjects

  • Diabetes or prediabetes with reduced glucose tolerance: FPG >6.0 mmol/l and/or HbA1c >42 mmol/mol
  • First degree relatives with type 2 diabetes
  • Inflammatory bowel disease
  • Intestinal resections
  • Treatment with medicine that cannot be paused for 12 hours
  • Pregnancy and/or breastfeeding
  • Age above 80 years

Study & Design

Study Type
INTERVENTIONAL
Study Design
CROSSOVER
Arm && Interventions
GroupInterventionDescription
CTRL + OGTT + LY2409021LY2409021Healthy controls + 50 oral glucose tolerance test 4 hours + the human antagonist of the glucagon receptor.
CTRL + IIGI + LY2409021IIGIHealthy controls + isoglycaemic iv glucose infusion + the human antagonist of the glucagon receptor.
T2D + IIGI + LY2409021IIGIType 2 diabetes patients + isoglycaemic iv glucose infusion + the human antagonist of the glucagon receptor.
T2D + OGTT + LY2409021OGTTType 2 diabetes patients + 50 oral glucose tolerance test 4 hours + the human antagonist of the glucagon receptor.
T2D + OGTT + placeboLY2409021 placeboType 2 diabetes patients + 50 oral glucose tolerance test 4 hours + placebo comparator to the human antagonist of the glucagon receptor.
T2D + MEAL + LY2409021Standardised liquid mealType 2 diabetes patients + Standardised liquid meal + the human antagonist of the glucagon receptor.
CTRL + MEAL + placeboStandardised liquid mealHealthy controls + Standardised liquid meal + placebo comparator of the human antagonist of the glucagon receptor.
T2D + OGTT + LY2409021LY2409021Type 2 diabetes patients + 50 oral glucose tolerance test 4 hours + the human antagonist of the glucagon receptor.
CTRL + OGTT + placeboOGTTHealthy controls + 50 oral glucose tolerance test 4 hours + placebo comparator of the human antagonist of the glucagon receptor.
CTRL + IIGI + placeboLY2409021 placeboHealthy controls + isoglycaemic iv glucose infusion + placebo comparator the human antagonist of the glucagon receptor.
CTRL + MEAL + placeboLY2409021 placeboHealthy controls + Standardised liquid meal + placebo comparator of the human antagonist of the glucagon receptor.
T2D + OGTT + placeboOGTTType 2 diabetes patients + 50 oral glucose tolerance test 4 hours + placebo comparator to the human antagonist of the glucagon receptor.
T2D + IIGI + placeboLY2409021 placeboType 2 diabetes patients + isoglycaemic iv glucose infusion + placebo comparator to the human antagonist of the glucagon receptor.
T2D + IIGI + placeboIIGIType 2 diabetes patients + isoglycaemic iv glucose infusion + placebo comparator to the human antagonist of the glucagon receptor.
T2D + MEAL + placeboLY2409021 placeboType 2 diabetes patients + Standardised liquid meal + placebo comparator to the human antagonist of the glucagon receptor.
T2D + MEAL + placeboStandardised liquid mealType 2 diabetes patients + Standardised liquid meal + placebo comparator to the human antagonist of the glucagon receptor.
CTRL + OGTT + LY2409021OGTTHealthy controls + 50 oral glucose tolerance test 4 hours + the human antagonist of the glucagon receptor.
CTRL + OGTT + placeboLY2409021 placeboHealthy controls + 50 oral glucose tolerance test 4 hours + placebo comparator of the human antagonist of the glucagon receptor.
CTRL + IIGI + LY2409021LY2409021Healthy controls + isoglycaemic iv glucose infusion + the human antagonist of the glucagon receptor.
CTRL + IIGI + placeboIIGIHealthy controls + isoglycaemic iv glucose infusion + placebo comparator the human antagonist of the glucagon receptor.
CTRL + MEAL + LY2409021Standardised liquid mealHealthy controls + Standardised liquid meal + the human antagonist of the glucagon receptor.
T2D + IIGI + LY2409021LY2409021Type 2 diabetes patients + isoglycaemic iv glucose infusion + the human antagonist of the glucagon receptor.
T2D + MEAL + LY2409021LY2409021Type 2 diabetes patients + Standardised liquid meal + the human antagonist of the glucagon receptor.
CTRL + MEAL + LY2409021LY2409021Healthy controls + Standardised liquid meal + the human antagonist of the glucagon receptor.
Primary Outcome Measures
NameTimeMethod
Differences in GIGD (%)Comparison between experimental days with and without the glucagon receptor antagonist . The glucose disposal at time 240 minutes will be used.

GIGD = Gastrointestinal glucose disposal. GIGD (%) = 100% × (glucoseOGTT-glucoseIIGI)/glucoseOGTT.

Difference in postprandial glucose excursionsArea under the curve (AUC) time frame: 0, 5, 10, 15, 20, 25, 30, 40, 50, 60, 70, 80, 90, 105, 120, 150, 180, 210, 240 minutes. Comparison between experimental days with and without the glucagon receptor antagonist.

Difference in postprandial glucose excursions (measured as incremental (baseline substracted) area under the curve (AUC) values).

Secondary Outcome Measures
NameTimeMethod
Incretin effectInsulin AUC time frame: 0,10, 20, 30, 50, 60, 70, 90, 105, 120, 150, 240 minutes. Comparison between experimental days with and without the glucagon receptor antagonist

The incretin effect (100% × \[β-cell secretory response to oral glucose tolerance test - intravenous β-cell secretory response\]/β-cell secretory response to oral glucose tolerance test)

Endogenous glucose productionPlasma concentration of 6,6^2 H2-glucose and U-13C^6-glucose at times: 0,10, 20, 30, 50, 60, 70, 90, 105, 120, 150, 240 minutes.

Glucose rate of appearance will be calculated by the non-steady state equation using double tracer technique.

LipolysisPlasma concentration of 1,1,2,3,3-^2-H5 - glycerol measured at times: 0,10, 20, 30, 50, 60, 70, 90, 105, 120, 150, 240 minutes.

Glycerol disappearance will be calculated by the non-steady state equation using double tracer technique.

Serum/plasma concentrations of insulin, C-peptide, glucagon, GIP and GLP-1.Time frame: 0,10, 20, 30, 50, 60, 70, 90, 105, 120, 150, 240 minutes.

Insulin, C-peptide, glucagon, GIP and GLP-1 serum/plasma concentrations will be measured in pM.

Energy intake (kcal/kJ)At time 240 to 270, the participants will eat an ad libitum meal. Comparison between experimental days with and without the glucagon receptor antagonist

At the end of the clamp experiment food intake will be examined with an ad libitum meal. The weight of the food will be measured i grams and calculated to the energy intake in kcal/kJ.

Changes in blood pressure (mmHg)Measured at time 0 and time 210 minutes. Comparison between experimental days with and without the glucagon receptor antagonist
Free fatty acids-30,-15, 0, 10, 20, 30, 50, 70, 90, 105, 120, 150, 240 minutes

serum values of free fatty acids

Fibroblast growth factor-21-30,-15, 0, 10, 20, 30, 50, 70, 90, 105, 120, 150, 240 minutes

plasma values of FGF-21

AppetiteVAS scales will be handed out at time 0, 30, 60, 90, 120, 150, 180 and 240 minutes.

Appetite will be evaluated with a visual analogue scale (VAS).

Changes in pulse rate (beat per minute)Measured at time 0 and at time 210 minutes. Comparison between experimental days with and without the glucagon receptor antagonist
Differences in gastric emptying-30,-15, 0, 10, 20, 30, 50, 70, 90, 105, 120, 150, 240 minutes

Measurement of p-paracetamol. Measurement of time to peak and incremental area under the curve (iAUC)

Trial Locations

Locations (1)

Center for Diabetes Research, Gentofte Hospital, Copenhagen University

🇩🇰

Hellerup, Denmark

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