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Clinical Trials/NCT05642273
NCT05642273
Completed
N/A

The Effects of extraCorporeal bLood Purification (oXiris ®) in patiEnts With cArdiogeNic Shock Requiring VA-ECMO: A Prospective, Open-label, Randomized Controlled Pilot Study

Samsung Medical Center1 site in 1 country40 target enrollmentNovember 25, 2022

Overview

Phase
N/A
Intervention
Not specified
Conditions
Cardiogenic Shock
Sponsor
Samsung Medical Center
Enrollment
40
Locations
1
Primary Endpoint
Endotoxin level at 48 h
Status
Completed
Last Updated
2 years ago

Overview

Brief Summary

Cardiogenic shock (CS) defines a state of systemic hypo-perfusion leading to end-organ dysfunction related to cardiac pump failure and with mortality rates in the range of 27-50% according to recent reviews. Patients with CS often received mechanical circulatory support, and venoarterial extracorporeal membrane oxygenator (V-A ECMO) is an effective tool to support refractory CS while ensuring continuous organ perfusion. Patients with CS present clinical signs of systemic inflammation and elevated plasma levels of prototypical inflammatory and vasoactive mediators, including C-reactive protein (CRP), Interleukin-6 (IL-6) and tumor necrosis factor alpha (TNFα). As data is scarce in this field, we decided to perform a prospective randomized controlled pilot study to investigate the efficacy of extracorporeal cytokine and lipopolysaccharide adsorption using Oxiris on humoral inflammation parameters, hemodynamics, and clinical outcomes in patients with CS requiring VA ECMO.

Registry
clinicaltrials.gov
Start Date
November 25, 2022
End Date
January 8, 2024
Last Updated
2 years ago
Study Type
Interventional
Study Design
Parallel
Sex
All

Investigators

Responsible Party
Principal Investigator
Principal Investigator

Jeong Hoon Yang

Professor

Samsung Medical Center

Eligibility Criteria

Inclusion Criteria

  • Patients with more than 18 years old
  • CS is defined as the presence of the following:
  • 2-1) Systolic blood pressure is less than 90 mmHg for more than 30 minutes despite the fluid therapy, or the use of pressure boosting agents to maintain the systolic blood pressure more than 90 mmHg.
  • 2-2) Peripheral hypoperfusion (cold skin, urine less than 30 cc per hour, impaired consciousness, lactate ≥2.0 mmol/l) or a person with pulmonary edema.
  • 2-3) Causes of CS include ischemic (acute myocardial infarction or ischemic cardiomyopathy, shock during percutaneous coronary intervention), myocardial (end-stage heart failure, myocarditis), post-cardiotomy shock, cardiac tamponade, or pulmonary thromboembolism.
  • Patients receiving VA ECMO owing related to the causes listed in 2-1, 2-2, 2-
  • Written informed consent from patient or legal surrogates

Exclusion Criteria

  • Other causes except for CS: septic shock, cardiac arrest by serious ventricular arrhythmia mot related to the myocardial ischemia or heart failure.
  • Shock with unwitnessed cardiac arrest outside the hospital
  • Severe non-cardiac morbidity with expected survival less than 6 months (malignancy, respiratory failure)
  • Suspicious of brain death
  • Those who refused active treatment
  • Body weight under 30 kg
  • Heparin allergy

Outcomes

Primary Outcomes

Endotoxin level at 48 h

Time Frame: 48 hours after enrollment

Plasma endotoxin level

Secondary Outcomes

  • Neurologic outcome(at hospital discharge, through study completion, an average 2 months)
  • Plasma cytokines(baseline, 24 hours, 48 hours, Day 7)
  • Vasoactive-Inotrope score(baseline, 24 hours, 48 hours, 72 hours, 96 hours, Day 7)
  • Sequential Organ Failure Assessment score(baseline, 24 hours, 48 hours, 72 hours, 96 hours, Day 7)
  • GDF-15, angiopoietin-2(baseline, 24 hours, 48 hours, Day 7)
  • Hospital mortality(at hospital discharge, through study completion, an average 2 months)

Study Sites (1)

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