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Clinical Trials/NCT02823184
NCT02823184
Completed
Not Applicable

Endoplasmic Reticulum Stress and Resistance to Treatments in Ph-negative Myeloproliferative Neoplasms

University Hospital, Bordeaux7 sites in 1 country148 target enrollmentApril 27, 2017

Overview

Phase
Not Applicable
Intervention
Not specified
Conditions
Polycythemia Vera
Sponsor
University Hospital, Bordeaux
Enrollment
148
Locations
7
Primary Endpoint
To correlate endoplasmic reticulum stress (defined as splicing of XBP1 above 30%) to the rate of complete response after 6 months according to the 2009 ELN criteria
Status
Completed
Last Updated
5 years ago

Overview

Brief Summary

The aim of this study is to evaluate the endoplasmic reticulum stress markers as predictive for response to hydroxyurea in polycythemia vera (PV) and essential thrombocythemia (ET).

Detailed Description

The recent discovery of calreticulin mutations in myeloproliferative neoplasms point to the unexpected role of the endoplasmic reticulum biology in the pathophysiology in these diseases. Otherwise, the association of endoplasmic reticulum stress with solid cancers, in particular in resistance to chemotherapy, is well documented, contrary to hematological malignancies. The study aims to evaluate endoplasmic reticulum stress markers as predictors for the response to hydroxyurea in polycythemia vera and essential thrombocythemia patients. The main objective is to correlate endoplasmic reticulum stress (defined as splicing of XBP1 above 30%) to the rate of complete response after 6 months according to the 2009 ELN criteria. This is an observational retrospective study.

Registry
clinicaltrials.gov
Start Date
April 27, 2017
End Date
April 27, 2019
Last Updated
5 years ago
Study Type
Observational
Sex
All

Investigators

Sponsor
University Hospital, Bordeaux
Responsible Party
Sponsor

Eligibility Criteria

Inclusion Criteria

  • ET or PV patients diagnosed before acceleration phase and treated by hydroxyurea with a follow up period of at least 6 months following treatment start.
  • Diagnosis criteria of PV :
  • WHO criteria of PV with :
  • Acquired JAK2V617F mutation \> 5%
  • Absence of evident cause of secondary polycythemia
  • Diagnosis criteria of ET :
  • Platelet count \> 450 G/L
  • Absence of PV or Chronic Myeloid Leukemia
  • Bone marrow biopsy preferred but not necessary in absence of reactional causes (CRP and ferritin levels normal) and/or presence of acquired JAK2V617F, CALR exon 9 or MPL exon 10
  • Availability of RNA sample of total leukocytes before start of treatment.

Exclusion Criteria

  • In absence of clonality marker, presence of secondary cause of :
  • Thrombocytosis :
  • Inflammatory syndrom (CRP or SV increased)
  • Iron deficiency (decreased ferritin level or increased soluble transferrin receptor level)
  • Polycythemia :
  • Increased or normal level of EPO in context of :
  • Hypoxia, respiratory insufficiency
  • Sleep apnea syndrome
  • Hyperaffin hemoglobin
  • Absence of treatment by hydroxyurea

Outcomes

Primary Outcomes

To correlate endoplasmic reticulum stress (defined as splicing of XBP1 above 30%) to the rate of complete response after 6 months according to the 2009 ELN criteria

Time Frame: After 6 months of treatment

Study Sites (7)

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