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Clinical Trials/NCT03953989
NCT03953989
Completed
Phase 2

A Pilot Study Assessing the Effects of Ranolazine on Coronary Microvascular Dysfunction in Patients With Hypertrophic Cardiomyopathy

IRCCS San Raffaele1 site in 1 country26 target enrollmentOctober 2016

Overview

Phase
Phase 2
Intervention
Ranolazine PR (prolonged-release) 500 mg 1 tablet bis in die and 750 mg 1 tablet bis in die
Conditions
HCM - Hypertrophic Non-Obstructive Cardiomyopathy
Sponsor
IRCCS San Raffaele
Enrollment
26
Locations
1
Primary Endpoint
Myocardial Blood Flow during hyperemia ml/min/g
Status
Completed
Last Updated
5 years ago

Overview

Brief Summary

To demonstrate the efficacy of ranolazine in improving coronary microvascular and diastolic dysfunction in patients affected by HCM evaluating changes in maximum (i.e. during dipyridamole-induced coronary vasodilatation) myocardial blood flow (MBF) measured by PET at baseline and after 4 months of treatment with ranolazine in patients with non obstructive HCM.

Detailed Description

This pilot study aimed at assessing the effects of treatment with ranolazine, on top of optimal standard medical therapy (according to international guidelines), on microvascular dysfunction using PET in patients with HCM. Thirty months of enrolment are foreseen. Patients enrolled in the study, at the end of the titration phase will be treated with ranolazine 750 mg or 500 mg, 1 oral tablet twice a day for 4 months on top of standard of care treatment. Visit 0,1,2,3 may be performed 3 days before or 3 days after the planned date. Visit 4 and final PET scan may be performed 15 days before or 15 days after the planned date. This is a superiority study. The sample size calculation is based on the ANOVA model, but the statistical analysis will be performed applying the ANCOVA model for the absence of data on the correlation between basal and end of study values of the primary variable studied. As previously reported (Camici et al. J Am Coll Cardiol 1991; 17:879-86) maximum (i.e. during dipyridamole stress) MBF is severely blunted in HCM (1.63±0.58 mL/min/g) compared to control subjects (2.99±1.06 mL/min/g). For the primary end-point of the study using a two-sided test at 5% significance level, with a power of 90% and a SD of ± 0.7 mL/min/gr, in order to detect a change of 0.5 mL/min/gr in maximum MBF before and after treatment, 24 patients valid per protocol are required.

Registry
clinicaltrials.gov
Start Date
October 2016
End Date
March 6, 2020
Last Updated
5 years ago
Study Type
Interventional
Study Design
Single Group
Sex
All

Investigators

Responsible Party
Principal Investigator
Principal Investigator

Prof. Paolo G Camici MD FACC

Professor of Cardiology

IRCCS San Raffaele

Eligibility Criteria

Inclusion Criteria

  • Male and female gender (females of childbearing potential must be using highly effective contraceptive precautions such as implants, injectables, combined oral contraceptives, intrauterine devices, sexual abstinence or vasectomised partner);
  • Females of childbearing potential or within two years from the menopause must have a negative urine pregnancy test;
  • Patients which fulfill conventional echocardiographic criteria for the diagnosis of HCM: maximum LV wall thickness ≥ 15 mm;
  • Patients aged \> 18 years and \< 80 years;
  • Sinus rhythm accepted isolated Supraventricular and Ventricular Premature Beats (VPB);
  • Absence of severe resting LV outflow tract obstruction (peak gradient ≤ 50 mmHg);
  • Written informed consent prior to enrolment into the study;

Exclusion Criteria

  • Females of childbearing potential not using highly effective contraceptive precautions;
  • Presence of known coronary artery disease (CAD);
  • Presence of Chronic Obstructive Airways Disease;
  • Other causes of microvascular dysfunction including long-standing history of arterial hypertension, diabetes, uncontrolled dyslipidemia;
  • Body mass index \>32 kg/m2; \< 17 kg/m2
  • Overt LV systolic dysfunction with end-stage progression (LV-EF \<50%);
  • Concomitant administration of potent CYP3A4 inhibitors (e.g. itraconazole, ketoconazole, voriconazol, posaconazol, HIV protease inhibitors, clarithromycin, telithromycin, nefazodone);
  • Patients treated with sotalol, dronedarone, class I antiarrhythmics (see appendix 4) or other QT-prolonging drugs; stable treatment with amiodarone is permitted;
  • Patients with QTc (Bazett's formula) at baseline ≥ 450 ms males; ≥470 msec females;
  • Any clinically relevant haematological or biochemical abnormality on routine screening, according to Investigator's judgment;

Arms & Interventions

Patients with hypertrophic cardiomyopathy

Thestudy is articulated into Screening visit plus other 3 visits. One visit (V2 ) for dose titration. V0 screening eligibility, consent, Medical History, physical examination, BP, ECG, Echocardiography, Biochemistry (haematology, electrolytes, glycaemia, ALS and bilirubin, creatinine and urine-analysis) V1 Baseline PET scan, physical examination, vital signs, and drug supply (500 mg bid). V2 physical examination, safety check, biochemistry, drug compliance and up-titration (750 mg bid), drug supply. V3 (2 months)safety check ,drug supply, drug compliance V4 (4 months, end of treatment) repeat PET scan, physical examination, BP, ECG, drug compliance, safety. Drug of the study Ranolazine PR (prolonged-release) 500 mg 1 tablet bis in die and 750 mg 1 tablet bis in die

Intervention: Ranolazine PR (prolonged-release) 500 mg 1 tablet bis in die and 750 mg 1 tablet bis in die

Outcomes

Primary Outcomes

Myocardial Blood Flow during hyperemia ml/min/g

Time Frame: At baseline and after 4 months of treatment

Change of near maximal hyperemic myocardial blood flow (MBF ml/min/g) after treatment with ranolazine of at least 0.5 ml/min/g. Myocardial perfusion measured with 13N-ammonia and positron emission tomography at rest and during hyperemia; Hyperaemic Myocardial Blood Flow (MBF) measured following i.v. dipyridamole (0.56 mg/Kg in 4 mins)

Coronary Flow Reserve (hyperaemic MBF /resting MBF = CFR).

Time Frame: At baseline and after 4 months of treatment

Change of CFR after treatment with ranolazine for four months

Coronary resistance

Time Frame: At baseline and after 4 months of treatment

Change of Resting coronary resistance (Mean Arterial Pressure rest/ MBF rest) and minimal (during hyperemia) coronary resistance (Mean arterial Pressure hyperemia/ MBF hyperemia)

Secondary Outcomes

  • Symptoms(through 4 month of treatment)

Study Sites (1)

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