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Clinical Trials/NCT06336655
NCT06336655
Recruiting
Phase 2

Physiology of Unloading VA ECMO Trial

University of Utah1 site in 1 country104 target enrollmentOctober 2, 2024

Overview

Phase
Phase 2
Intervention
Not specified
Conditions
Cardiogenic Shock
Sponsor
University of Utah
Enrollment
104
Locations
1
Primary Endpoint
Change in pulmonary capillary wedge pressure
Status
Recruiting
Last Updated
last year

Overview

Brief Summary

The goal of this clinical trial is to compare the use of veno-arterial extracorporeal membrane oxygenation (VA ECMO) with and without left ventricular (LV) unloading in patients being treated for cardiogenic shock (CS). The main aims of the study are:

  1. To determine the physiologic effects on cardiopulmonary congestion of adding LV unloading to VA ECMO
  2. To determine the effects on myocardial function of adding LV unloading to ECMO
  3. To test the effects on myocardial recovery of adding LV unloading to VA ECMO

Participants who are being treated with VA ECMO will be randomized to receive or not receive LV unloading in the form of an intra-aortic balloon pump (IABP). Over the course of the study, the investigators will obtain measurements via lab work, echocardiography, and pulmonary artery catheter that will allow comparison of the two groups.

Detailed Description

Although extracorporeal membrane oxygenation (ECMO) for cardiogenic shock (CS) is used in over 3,000 patients per year, the best management strategies are not known. Identifying and improving treatment of CS is critically important, as CS occurs in 160,000 patients per year in the US with a 50% mortality rate. VA ECMO is an increasingly used method of mechanical circulatory support (MCS) for patients with CS refractory to medical therapy. Despite the benefit of full cardiopulmonary support ECMO is also thought to increase after load in the failing heart- which paradoxically reduces cardiac output and may lead to myocardial injury and cardiac congestion. A potential solution is to add a device to VA ECMO that decreases after-load - known as left ventricular (LV) unloading. LV unloading can be achieved with different approaches, directly with transvalvular pumps (known as a peripheral ventricular assist device (pVAD)), or indirectly with an intra-aortic balloon pump (IABP) Preliminary data suggests that unloading the LV is associated with improved survival. Results from a cohort of VA ECMO patients with medical CS, showed a hospital survival benefit LV unloading (aOR 0.87 (0.79, 094); p=0.001). Data has also shown that the survival benefit of LV unloading was much larger with pVAD (HR 0.6), but with higher complications, including limb ischemia - a potentially catastrophic complication. However, results also show that different unloading approaches have different physiologic effects on the myocardium and on peripheral perfusion - highlighting the uncomfortable observation that it is not known how (physiologically) these unloading devices lead to changes in survival. There are two potential pathways whereby LV unloading could influence survival, including myocardial effects (distension, injury, ejection fraction ) and peripheral effects (peripheral pulse pressure, lactate clearance, CO2 gap). Determining the physiologic effects from LV unloading according to device type and patient etiology will allow us to match the intervention with the patient's physiology. Data suggests that ECMO patients with acute myocardial infarction (AMI) have different mortality and different physiologic changes than patients with decompensated chronic heart failure (CHF) when unloaded. The ultimate goal is to reduce morbidity and mortality in cardiogenic shock. This study will define the physiologic benefit of LV unloading during CS.

Registry
clinicaltrials.gov
Start Date
October 2, 2024
End Date
February 1, 2029
Last Updated
last year
Study Type
Interventional
Study Design
Parallel
Sex
All

Investigators

Responsible Party
Principal Investigator
Principal Investigator

Joseph Tonna

Associate Professor, Division of Cardiothoracic Surgery

University of Utah

Eligibility Criteria

Inclusion Criteria

  • Adult patients (age 18 years or older)
  • Diagnosis of acute cardiogenic shock (CS)
  • Patients failing medical therapy, defined as 1 or more of the following:
  • Society for Coronary Angiography and Interventions (SCAI) Stage C or greater
  • 2 or more inotropic medications and not improving
  • IABP in place and clinically worsening
  • Placed on VA ECMO for CS
  • In the opinion of the attending physician, patient has worsening CS and could require VA ECMO support in the near-term

Exclusion Criteria

  • Metastatic or stage 4 cancer
  • Atrial septostomy
  • Planned LV unloading on ECMO
  • Anticipated death \<72 hours
  • Existing durable left ventricular assist device (dLVAD)
  • Unwillingness to randomize
  • Patients who are pregnant

Outcomes

Primary Outcomes

Change in pulmonary capillary wedge pressure

Time Frame: ECMO start, ECMO day 5, ECMO decannulation up to 3 months, day of ICU discharge up to 6 months

Change in pulmonary capillary wedge pressure (PCWP) from ECMO start to ECMO day 5 (Day 5 - Baseline) with and without LV unloading

Secondary Outcomes

  • Change in pulmonary artery diastolic pressure(ECMO start, ECMO days 1-5, ECMO decannulation up to 3 months, day of ICU discharge up to 6 months)
  • Change in left ventricular end diastolic diameter(ECMO start, ECMO day 5, ECMO decannulation up to 3 months, day of ICU discharge up to 6 months)
  • Change in N-terminal pro b-type natriuretic peptide(ECMO start, ECMO days 1-5, ECMO decannulation up to 3 months, day of ICU discharge up to 6 months)
  • Difference in partial pressure of carbon dioxide (pCO2)(ECMO start, q12 hours ECMO days 1-3, ECMO decannulation up to 3 months, day of ICU discharge up to 6 months)
  • Hemodynamic stability(ECMO start, ECMO day 5)
  • Global cardiovascular function(ECMO start, ECMO days 1-5)
  • Lactate(ECMO start, ECMO days 1-5, ECMO decannulation up to 3 months, day of ICU discharge up to 6 months)
  • Cardiac BIN1(ECMO start, ECMO day 5, day of ICU discharge up to 6 months)
  • Troponin I(ECMO start, ECMO days 1-5, ECMO decannulation up to 3 months, day of ICU discharge up to 6 months)
  • Tumor necrosis factor alpha(ECMO start, ECMO day 5, ECMO decannulation up to 3 months, day of ICU discharge up to 6 months)
  • Ejection fraction percentage(ECMO start, ECMO day 5, ECMO decannulation up to 3 months, day of ICU discharge up to 6 months)
  • Mortality(Hospital discharge up to 6 months)
  • Interferon gamma(ECMO start, ECMO day 5, ECMO decannulation up to 3 months, day of ICU discharge up to 6 months)
  • Limb ischemia(Randomization, 48 hours post-device removal)

Study Sites (1)

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