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Clinical Trials/NCT04489160
NCT04489160
Unknown
Phase 2

A Phase II Trial on the Safety and Efficacy of C1 Inhibitor for the Acute Management of Severe Traumatic Brain Injury

Leiden University Medical Center3 sites in 1 country106 target enrollmentFebruary 25, 2021

Overview

Phase
Phase 2
Intervention
C1 Inhibitor, Human
Conditions
Traumatic Brain Injury
Sponsor
Leiden University Medical Center
Enrollment
106
Locations
3
Primary Endpoint
Therapy Intensity Level (TIL) Scale
Last Updated
4 years ago

Overview

Brief Summary

Severe Traumatic Brain Injury (s-TBI) is a major cause of death and disability across all ages. Besides the primary impact, the pathophysiologic process of major secondary brain damage consists of a neuroinflammation response that critically leads to irreversible brain damage in the first days after the trauma. A key catalyst in this inflammatory process is the complement system. Inhibiting the complement system is therefore considered to be a potentially important new treatment for TBI, as has been shown in animal studies. This trial aims to study the safety and efficacy of C1-inhibitor compared to placebo in TBI patients. By temporarily blocking the complement system we hypothesize limitation of secondary brain injury and more favourable clinical outcome for TBI patients due to a decrease in the posttraumatic neuroinflammatory response.

Registry
clinicaltrials.gov
Start Date
February 25, 2021
End Date
July 2024
Last Updated
4 years ago
Study Type
Interventional
Study Design
Parallel
Sex
All

Investigators

Sponsor
Leiden University Medical Center
Responsible Party
Principal Investigator
Principal Investigator

wcpeul

Principal Investigator

Leiden University Medical Center

Eligibility Criteria

Inclusion Criteria

  • Age at admission ≥ 18 years and \< 65 years;
  • Clinical diagnosis of traumatic brain injury with GCS \< 13 (with intracranial deviations);
  • Catheter placement for monitoring and management of increased ICP for at least 24 hours;

Exclusion Criteria

  • A clear, non-traumatic cause of low GCS (e.g. toxic, cardial) on admission;
  • Not expected to survive more than 24 hours after admission;
  • Brain death on arrival in the participating centers;
  • Severe pre-trauma disability, defined as being dependent on other people;
  • Known prior history of sensibility to blood products or Cinryze;
  • Patients with a history of hereditary angioedema;
  • Patients with a history of thrombosis;
  • Pregnant women.

Arms & Interventions

C1-inhibitor

One dose 6000 IU C1-inhibitor intravenously

Intervention: C1 Inhibitor, Human

Placebo

0.9% saline

Intervention: Placebo

Outcomes

Primary Outcomes

Therapy Intensity Level (TIL) Scale

Time Frame: First four ICU days

TIL differentiated for various treatment modalities aimed at prevention or control of raised Intracranial Pressure (ICP) and/or for CPP management (0 to 38 points)

Glasgow Outcome Scale Extended (GOSE)

Time Frame: At 6 months after trauma

Functional outcome (minimum score = 1, maximum score = 8)

Complication rate

Time Frame: Up to 1 year

Adverse and serious adverse events related possibly related to study medication

Secondary Outcomes

  • Intracranial pressure (ICP) burden(First four ICU days)
  • CT scan midline shift(Up to 1 year)
  • Mortality(Up to 1 year after trauma)
  • Glasgow Outcome Scale Extended (GOSE)(At discharge (an average of 14 days), 3 and 12 months after trauma)
  • QoLiBri(At 3, 6 and 12 months after trauma)
  • SF-36(At 3, 6 and 12 months after trauma)
  • EQ-5D-5L(At 6 and 12 months after trauma)
  • ICU length of stay(Up to 1 year)
  • Ventilator days(Up to 1 year)
  • Hospital length of stay(Up to 1 year)
  • Hospital disposition(Up to 1 year)
  • UCH-L1 and GFAP biomarkers(Baseline (Before adminstration of investigational product ) and 6, 12, 24, 48, 72 and 96 hours after adminstration of investigational product)
  • Complement activation(Baseline (Before adminstration of investigational product ) and 6, 12, 24, 48, 72 and 96 hours after adminstration of investigational product)
  • Coagulation cascade activation(Baseline (Before adminstration of investigational product ) and 6, 12, 24, 48, 72 and 96 hours after adminstration of investigational product)
  • Inflammatory markers(Baseline (Before adminstration of investigational product ) and 6, 12, 24, 48, 72 and 96 hours after adminstration of investigational product)

Study Sites (3)

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