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Clinical Trials/NCT04616872
NCT04616872
Unknown
Phase 2

Treatment of Patients With Coronary and Aortic Atherosclerotic Disease With Methotrexate-associated to LDL Like Nanoparticles. A Randomized, Double-blind, Placebo-control Trial

University of Sao Paulo General Hospital2 sites in 1 country40 target enrollmentOctober 10, 2020

Overview

Phase
Phase 2
Intervention
Methotrexate-LDE
Conditions
Atherosclerosis
Sponsor
University of Sao Paulo General Hospital
Enrollment
40
Locations
2
Primary Endpoint
Low Attenuation Plaque Volume (LAPV) coronary
Last Updated
5 years ago

Overview

Brief Summary

The investigators propose a prospective, randomized, double-blind, placebo-controlled study. The purpose of the study is to evaluate the safety and efficacy of an anti-inflammatory agent methotrexate in a cholesterol-rich non-protein nanoparticle (MTX-LDE) in patients with stable coronary disease.

Patients with multi-vessels stable coronary disease will be randomized to receive MTX-LDE IV or placebo-LDE IV each 7 days for 12 weeks. The primary and main secondary endpoints will be analyzed by coronary and aortic CT angiography, that will be performed before the first treatment cycle, four weeks after the last drug infusion and 12 months after randomization. Patients will undergo clinical and laboratory safety evaluations before each treatment cycle, four weeks after the last cycle and 12 months after randomization.

An algorithm for drug suspension based on clinical and laboratory finding will be followed.

Detailed Description

Atherosclerosis is a life-threatening condition, as long as cardiovascular disease is responsible for one-third of all global mortality. Inflammation is extremely important in atherosclerosis pathophysiology. The use of inflammatory biomarkers to predict risk, monitor treatments and guide therapy, has shown substantial potential for clinical applicability. Many studies in primary and secondary prevention of cardiovascular disease showed that individuals with lower high sensitive C-reactive protein (hsCRP) have better clinical outcomes than those with higher levels. The potential benefit of anti-inflammatory therapy in atherosclerosis has been previously demonstrated in studies in patients with chronic inflammatory diseases (rheumatoid arthritis, psoriasis). The use of methotrexate has been associated with a reduction in cardiovascular events in these patients. In this setting, the use of non-invasive treatments to reduce lesion size and inflammation is essential for the prevention of sub-sequent cardiovascular events. The systemic use of methotrexate at high doses for the treatment of atherosclerotic cardiovascular diseases is unlikely due to their significant, often life-threatening toxicity. Nonetheless, the toxicity of such agents can be strongly diminished by the use of optimized drug-delivery systems. In a pioneer study performed on patients with acute leukemia, was reported the potential of a cholesterol-rich non-protein nanoparticle (LDE) as a drug targeting agent. LDE particles have lipid compositions and structures that resemble low-density lipoprotein (LDL) and can be injected directly into the bloodstream. When LDE particles come into contact with plasma, the particles acquire exchangeable apolipoproteins from native lipoproteins, such as apolipoprotein (apo) E, which binds the particles to LDL receptors. In neoplastic cells, lipoprotein receptors are overexpressed, such that uptake of native LDL and of LDE particles is increased relative to that in normal tissues. In aortas of cholesterol-fed rabbits the uptake of LDE particles is increased in comparison to normal aortas and in rabbit-grafted hearts take up the nanoemulsion at amounts fourfold greater than native hearts. LDE-methotrexate treatment of rabbits induced to exhibit atherosclerosis via high cholesterol intake resulted in a 65% reduction in lesion size. The aim of this study is to investigate whether patients with aortic and coronary atherosclerotic disease showed good tolerability to LDE-methotrexate treatment and whether this formulation could achieve reduction in plaque volume and characteristics by coronary and aortic CT angiography.

Registry
clinicaltrials.gov
Start Date
October 10, 2020
End Date
October 12, 2023
Last Updated
5 years ago
Study Type
Interventional
Study Design
Parallel
Sex
All

Investigators

Responsible Party
Principal Investigator
Principal Investigator

Raul Cavalcante Maranhão

MD; PHD. Director Lipid Metabolism Laboratory, Heart Institute

University of Sao Paulo General Hospital

Eligibility Criteria

Inclusion Criteria

  • Multi-vessels coronary artery disease diagnosis by coronary CT or invasive angiography
  • Aortic atherosclerosis diagnosis by multidetector computed tomography (MDCT) angiography.
  • High-sensitivity C reactive protein (hs-CRP) levels \> 2mg/L
  • Signing the study informed consent.

Exclusion Criteria

  • History of Acute myocardial infarction in the last 30 days
  • Heart failure with ejection fraction \<40%
  • Estimated glomerular filtration rate \< 40 mL/min/1.73 m
  • Prior history of chronic infectious disease, including tuberculosis, severe fungal disease, or known HIV positive.
  • Chronic hepatitis B or C infection.
  • Prior history of nonbasal cell malignancy or myeloproliferative or lymphoproliferative disease within the past 5 years.
  • White blood cell count \<4000/mm3, hematocrit \<32%, or platelet count \<75000/mm
  • Alanine aminotransferase levels (ALT) greater than 3-fold the upper limit of normal.
  • History of actual alcohol abuse or unwillingness to limit alcohol consumption to \< 4 drinks per week.
  • Pregnancy or breastfeeding.

Arms & Interventions

Methotrexate-LDE

Methotrexate carried by a lipid nanoparticle (MTX-LDE)

Intervention: Methotrexate-LDE

Placebo-LDE

Lipid nanoparticle (LDE)

Intervention: Placebo-LDE

Outcomes

Primary Outcomes

Low Attenuation Plaque Volume (LAPV) coronary

Time Frame: Baseline and change from baseline to 12 months

Compare Low attenuation Plaque Volume( LAPV) measured by coronary CT angiography between groups.

Low Attenuation Plaque Volume (LAPV) aortic

Time Frame: Baseline and change from baseline to 12 months

Compare Low attenuation Plaque Volume( LAPV) measured by aortic CT angiography between groups.

Secondary Outcomes

  • Remodeling index (RI)(Baseline and change from baseline, 4 months and 12 months)
  • Noncalcified plaque volume (NCPV)(Baseline and change from baseline to 4 months)
  • Dense calcified plaque volume (DCPV)(Baseline and change from baseline to 12 months)
  • Total lumen value (TLV)(Baseline and change from baseline, 4 months and 12 months)
  • Total atheroma volume (TAV)(Baseline and change from baseline, 4 months and 12 months)
  • Total atheroma volume (TAV) aortic(Baseline and change from baseline, 4 months and 12 months)
  • Other adverse events(1; 2; 3; 4; 5; 6; 7; 8; 9; 10; 11; 12; 16 and 52 weeks after randomization)
  • Perivascular fat attenuation index (FAI)(Baseline and change from baseline, 4 months and 12 months)
  • Clinical significant symptoms(1; 2; 3; 4; 5; 6; 7; 8; 9; 10; 11; 12; 16 and 52 weeks after randomization)

Study Sites (2)

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