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Clinical Trials/NCT03122652
NCT03122652
Completed
Phase 3

Multi-center, Randomized, Double-blinded Study of Teriflunomide® in Radiologically Isolated Syndrome (RIS) The TERIS Study

Centre Hospitalier Universitaire de Nice23 sites in 3 countries125 target enrollmentSeptember 25, 2017

Overview

Phase
Phase 3
Intervention
Teriflunomide 14 MG Oral Tablet [Aubagio]
Conditions
Multiple Sclerosis
Sponsor
Centre Hospitalier Universitaire de Nice
Enrollment
125
Locations
23
Primary Endpoint
Time to the first acute or progressive neurological event resulting from CNS demyelination.
Status
Completed
Last Updated
3 years ago

Overview

Brief Summary

Multiple sclerosis (MS) is a common cause of severe neurological disability in young adults, resulting from an autoimmune interruption of both myelin and axons within the central nervous system (CNS). The diagnosis is made by fulfilling both spatial criteria, by meeting the requisite number of lesions within the brain or spinal cord, along with criteria for time, by demonstrating a history of at least a second clinical attack or the development of a new MS lesion on MRI after the seminal neurological event. In the case of MS, healthy individuals who do not exhibit signs of neurological dysfunction commonly have brain MRI studies performed for a reason other than an evaluation for MS that reveal unexpected anomalies highly suggestive of demyelinating plaques given their size, location, and morphology. These healthy subjects lack symptomatology suggestive of MS and fulfill formal criteria for radiologically isolated syndrome (RIS), a recently described MS subtype that expands upon the phenotype of at-risk individuals for future demyelinating events. The discovery of such anomalies creates intersecting neuro-ethical, legal, social, and practical medical management quandaries and is, therefore, of both immediate and long-term clinical significance. Despite advancements in the characterization of RIS subjects, and in our understanding of risk factors for initial symptom development, the effect of treatment on such cases remain unclear.

The purpose of this investigation is to systematically study the efficacy of Teriflunomide in those individuals who possess incidental white matter anomalies within the brain and following a MRI study that is performed for a reason other than for the evaluation of MS.

RIS subjects are frequently exposed to disease modifying therapies despite the lack of scientific literature supporting the use of such treatments. Earlier treatment intervention may extend the time to the first acute or progressive clinical event resulting from CNS demyelination and reduce radiological progression. In addition, early treatment may result in more profound effects on reducing disability progression long-term.

The primary outcome measure for this trial is the time to the first acute or progressive neurological event resulting from CNS demyelination.

This study will include RIS subjects from the Europe who fulfill 2009 RIS Criteria.

Registry
clinicaltrials.gov
Start Date
September 25, 2017
End Date
October 4, 2022
Last Updated
3 years ago
Study Type
Interventional
Study Design
Parallel
Sex
All

Investigators

Responsible Party
Sponsor

Eligibility Criteria

Inclusion Criteria

  • Males and females of all ages(\>18 years and \<65 years) meeting 2009 RIS criteria:
  • A. The presence of incidentally identified CNS white matter anomalies meeting the following MRI criteria:
  • Ovoid, well-circumscribed, and homogeneous foci observed with or without involvement of the corpus callosum
  • T2 hyperintensities measuring ≥3 mm and fulfilling Barkhof criteria (at least three out of four) for dissemination in space
  • Anomalies not following a clear vascular pattern
  • Structural neuroimaging abnormalities identified not explained by another disease process B. No historical accounts of remitting clinical symptoms consistent with neurological dysfunction C. The MRI anomalies do not account for clinically apparent impairments in social, occupational, or generalized area of functioning D. The MRI anomalies are not due to the direct physiological effects of substances (recreational drug use, toxic exposure) or a medical condition E. Exclusion of individuals with MRI phenotypes suggestive of leukoaraiosis or extensive white matter changes lacking clear involvement of the corpus callosum F. The CNS MRI anomalies are not better accounted for by another disease process
  • Identified RIS cases with the initial MRI demonstrating anomalies suggestive of demyelinating disease dated ≥ 2009
  • Incidental anomalies identified on MRI of the brain or spinal cord with the primary reason for the acquired MRI resulting from an evaluation of a process other than MS
  • Affiliation to the social security system
  • Subjects of reproductive potential are eligible only if the following applies:

Exclusion Criteria

  • Hypersensitivity to the active substance or to any of the excipients listed in section 6.1 of summary of product characteristics (SmPC).
  • Patients with severe hepatic impairment (Child-Pugh class C).
  • Patients with severe immunodeficiency states, e.g. AIDS.
  • Patients with significantly impaired bone marrow function or significant anaemia, leucopenia, neutropenia or thrombocytopenia.
  • Patients with severe active infection until resolution.
  • Patients with severe renal impairment undergoing dialysis.
  • Patients with severe hypoproteinaemia, e.g. in nephrotic syndrome.
  • Lactating or pregnant women
  • Subjects wishing to parent a child during the study
  • Incomplete medical history or radiological data

Arms & Interventions

Terifunomide

Intervention: Teriflunomide 14 MG Oral Tablet [Aubagio]

Placebo

Intervention: Placebo Oral Tablet

Outcomes

Primary Outcomes

Time to the first acute or progressive neurological event resulting from CNS demyelination.

Time Frame: Week 96

Acute neurological event: The development of an acute neurological episode localized to the optic nerve, brainstem, cerebellum, spinal cord, or long sensory or motor tracts, lasting \> 24 hours followed by a period of symptom improvement. Progressive event: The onset of a clinical symptom (e.g. leg weakness) with the temporal profile revealing at least a 12-month progression of neurological deficits.

Secondary Outcomes

  • New or enlarging T2 lesions(Week 96)
  • New contrast enhancing lesions(Week 96)
  • New T2-lesion volumes(Week 96)
  • Brain atrophy(Week 96)

Study Sites (23)

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