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Lifestyle Modification Programme for HIV-infected Individuals With Fatty Liver

Not Applicable
Recruiting
Conditions
Hiv
NAFLD
Interventions
Behavioral: Lifestyle modification
Registration Number
NCT03913351
Lead Sponsor
Chinese University of Hong Kong
Brief Summary

Non-alcoholic fatty liver disease (NAFLD) is rising in prevalence, and will likely become the predominant cause of chronic liver disease in HIV-infected individuals.

Metabolic factors and obesity are important risk factors for NAFLD in HIV-infected individuals. There is currently no approved effective pharmacological treatment for fatty liver disease. Therefore, lifestyle modification directing at weight loss is currently the cornerstone of treatment for fatty liver disease in the general population. Hypocaloric diets can improve fatty liver in the general population, but the most effective specific dietary interventions are yet to be elucidated.

The study aims to 1. determine the efficacy of a lifestyle modification programme in inducing resolution of NAFLD in HIV-infected individuals 2. to determine the efficacy of a lifestyle modification programme in improving insulin resistance, pro-inflammatory markers, and liver fibrosis in HIV-infected individuals with fatty liver disease 3. to determine changes in intestinal microbiome secondary to the lifestyle modification programme, and the association with resolution of NAFLD in this group of patients.

Detailed Description

Not available

Recruitment & Eligibility

Status
RECRUITING
Sex
All
Target Recruitment
96
Inclusion Criteria
  • age 18 years or above
  • positive HIV antibody, on anti-retroviral therapy
  • HIV viral load ≤50 copies/mL for ≥6 months
  • intrahepatic triglyceride content ≥5% on magnetic resonance spectroscopy
Exclusion Criteria
  • current AIDS-defining illness
  • active malignancy, or history of malignancy within the last 5 years
  • hepatitis B and/or hepatitis C co-infection, as determined by positive HBsAg and anti--HCV antibody
  • alcohol consumption >30g per week in men or 20g per week in women
  • alanine aminotransferase (ALT) above 10 times the upper limit of normal
  • liver decompensation (as evidenced by bilirubin above 50 µmol/l, platelet count below 100 × 109/l, prothrombin time above 1.3 times the upper limit of normal, albumin below 35 g/l, presence of ascites or varices).

Study & Design

Study Type
INTERVENTIONAL
Study Design
PARALLEL
Arm && Interventions
GroupInterventionDescription
Lifestyle modification programLifestyle modificationThe dietary intervention program will be scheduled for 12 months, and conducted by a dietitian.The program aims to increase energy expenditure and reduce caloric intake, with an emphasis on long-term lifestyle and behavioural change. An exercise instructor will provide advice on physical activity. A mobile tracking device to monitor calories expenditure will be provided to each participant during they study period to encourage physical activity.
Primary Outcome Measures
NameTimeMethod
Resolution of NAFLD12 months

The primary endpoint is the proportion of patients with resolution of NAFLD as determined by proton-magnetic resonance spectroscopy at month 12.

Secondary Outcome Measures
NameTimeMethod
Change in liver fibrosis12 months

The changes in liver fibrosis will be determined by transient elastography by Fibroscan

Biomarkers of inflammation and monocyte activation12 months

Changes from baseline in marker of monocyte activation (sCD163)

Metabolic endpoints12 months

The proportion of patients with metabolic syndrome will be determined

Partial resolution of NAFLD12 months

Partial resolution of NAFLD is defined as absolute reduction of hepatic triglyceride content by 30% or more.

Changes in adiposity12 months

The changes in visceral fat will be determined by magnetic resonance imaging at the same session

Trial Locations

Locations (1)

Prince of Wales Hospital

🇭🇰

Shatin, Hong Kong

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