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Food Supplementation With Diuripres for Blood Pressure Modulation in Subjects With High-Normal Blood Pressure

Not Applicable
Completed
Conditions
High-Normal Blood Pressure
Interventions
Dietary Supplement: Dietary Supplement
Other: Placebo
Registration Number
NCT05167747
Lead Sponsor
University of Bologna
Brief Summary

Cardiovascular diseases (CVDs) are the leading cause of mortality worldwide, reaching the 31% of deaths in 2012. CVDs represent also the major cause of disability in developed countries and has been estimated that their growing burden could lead to a global increase in loss of disability-adjusted life years (DALY), from a loss of 85 million DALYs of 1990 to a loss of \~ 150 million DALYs in 2020, becoming a major cause of no psychic responsible for lost productivity.

Several risk factors contribute to the aetiology and development of CVD. These factors have been traditionally stratified into modifiable risk factors through the lifestyle changes or by taking a pharmacologic treatment (e.g. hypertension, smoking, diabetes mellitus, hypercholesterolemia) and not modifiable risk factors (age, male sex and family history). Essential hypertension is the most common modifiable risk factor in the general population, with a prevalence in Western Countries -including Italy- ranging between about 25-45%.

Given the large prevalence of the disease of the general population, hypertension is responsible for the vast majority of CVD in individuals with different CV risk profiles, despite the availability of effective and well tolerated antihypertensive therapies. In this regard, several reports have shown that hypertensive patients often present additional CV and metabolic risk factors, mostly hypercholesterolemia, hypertriglyceridemia, metabolic syndrome and diabetes, which further contribute to increase the individual risk of developing hypertension-related complications, including stroke, end-stage renal disease, congestive heart failure, and CVD death. The concomitant presence of hypertension and dyslipidaemia is also responsible for the objective difficulty in achieving the recommended therapeutic targets for BP and cholesterol levels in a setting of clinical practice.

Several pharmacological and non-pharmacological interventions have been proposed for ameliorating the relatively low rates of control of hypertension. Among these, an extensive use of nutrients and food supplements has been shown to provide favourable effects in the management and control of high-normal blood pressure (BP) (or pre-hypertension), that increases the risk of developing hypertension, cardiovascular diseases, and renal failure.

Detailed Description

Among food supplements largely used in this field, Orthosiphon stamineus Benth leaf extract has been recognized for its well-known diuretic properties; extract of hawthorn (Crataegus curvisepala Lind.) has been shown to exert a renal-protective effect in high salt-induced hypertension and extract of hibiscus (Hibiscus sabdariffa L.) is well known for its antihypertensive and vasodilator effect in human. Moreover, supplementation with magnesium has been recently found to play a critical role in BP regulation, through directly stimulating prostacyclin and nitric oxide formation, modulating endothelium-dependent and endothelium-independent vasodilation, reducing vascular tone and reactivity, and preventing vascular injury via its antioxidant and anti-inflammatory functions.

The objective of this study is to evaluate the effect of dietary supplementation of a combined food supplement (NUT) containing magnesium, standardized extract of orthosiphon (Orthosiphon stamineus Benth), standardized extract of hawthorn (Crataegus curvisepala Lind.) and standardized extract of hibiscus (Hibiscus sabdariffa L.) compared to placebo, on blood pressure and other markers of vascular aging in subjects with high-normal blood pressure.

Recruitment & Eligibility

Status
COMPLETED
Sex
All
Target Recruitment
60
Inclusion Criteria
  • Subjects agree to participate in the study and having dated and signed the informed consent form;
  • Subjects who have the capability to communicate, to make themselves understood, and to comply with the study's requirements;
  • Subjects with high-normal blood pressure (SBP= 130-139 mmHg and/or DBP= 85-89 mmHg);
  • Subjects who, according to the SCORE charts, have a low or moderate cardiovascular risk (defined as a total cardiovascular risk < 5%) and for whom, according to 2018 ESC/ESH guidelines, the intervention strategy does not require a pharmacological intervention.
Exclusion Criteria
  • Subjects already affected by cardiovascular diseases (secondary prevention) or with an estimated 10 years cardiovascular disease risk> 5%;
  • Obesity (Body mass index>30 kg/m2);
  • Type 1 or type 2 diabetes;
  • Assumption of anti-hypertensive drugs or food supplements, or drugs potentially affecting blood pressure;
  • Lipid-lowering treatment not stabilized since at least 2 months;
  • Anticoagulants therapy;
  • Known current thyroid, gastrointestinal or hepatobiliary diseases;
  • Any medical or surgical condition that would limit the patient adhesion to the study protocol;
  • Abuse of alcohol or drugs (current or previous);
  • History of malignant neoplasia in the 5 years prior to enrolment in the study;
  • History or clinical evidence of inflammatory disease such as severe arthritis, systemic lupus erythematosus or chronic inflammatory diseases or current therapy with immunosuppressive agents or long-term glucocorticoids;
  • History or clinical evidence of any significant concomitant disease that could compromise the safety of the subject or the possibility of completing the study;
  • Known previous intolerance to one component of the tested nutraceuticals;
  • Women in fertile age not using consolidated contraceptive methods;
  • Pregnancy and Breastfeeding.

Study & Design

Study Type
INTERVENTIONAL
Study Design
PARALLEL
Arm && Interventions
GroupInterventionDescription
Active ComparatorDietary SupplementDietary supplement
Placebo comparatorPlaceboPlacebo
Primary Outcome Measures
NameTimeMethod
Systolic blood pressure absolute reduction from baseline and between groups8 weeks

Absolute reduction of systolic blood pressure after 8 weeks of treatment

Diastolic blood pressure absolute reduction from baseline and between groups8 weeks

Absolute reduction of diastolic blood pressure after 8 weeks of treatment

Secondary Outcome Measures
NameTimeMethod
Absolute change from baseline and between groups in body mass index8 weeks

Absolute change of body mass index after 8 weeks of treatment

Absolute change from baseline and between groups in waist/hip circumference ratio8 weeks

Absolute change waist/hip circumference ratio after 8 weeks of treatment

Diastolic blood pressure absolute reduction from baseline and between groups4 weeks

Absolute reduction of diastolic blood pressure after 4 weeks of treatment

Absolute change from baseline and between groups in fat and lean mass8 weeks

Absolute change of fat and lean mass after 8 weeks of treatment

Absolute change from baseline and between groups in serum lipids8 weeks

Absolute change of serum concentrations of total cholesterol, triglycerides - TG, high-density lipoprotein cholesterol - HDL-C, non-high-density lipoprotein cholesterol - non HDL-C, low-density lipoprotein cholesterol - LDL-C after 8 weeks of treatment

Absolute change from baseline and between groups in high sensitivity C reactive protein8 weeks

Absolute change of high sensitivity C reactive protein (hsCRP) after 8 weeks of treatment

Absolute change from baseline and between groups in the visceral adiposity index8 weeks

Absolute change of the visceral adiposity index after 8 weeks of treatment

Absolute change from baseline and between groups in waist circumference8 weeks

Absolute change of waist circumference after 8 weeks of treatment

Systolic blood pressure absolute reduction from baseline and between groups4 weeks

Absolute reduction of systolic blood pressure after 4 weeks of treatment

Absolute change from baseline and between groups in body weight8 weeks

Absolute change of body weight after 8 weeks of treatment

Absolute change from baseline and between groups in hip circumference8 weeks

Absolute change of hip circumference after 8 weeks of treatment

Absolute change from baseline and between groups in serum uric acid concentrations8 weeks

Absolute change of serum uric acid after 8 weeks of treatment

Absolute change from baseline and between groups in fasting plasma glucose concentrations8 weeks

Absolute change of fasting plasma glucose after 8 weeks of treatment

Percent reduction from baseline and between groups in vascular reactivity8 weeks

Percent reduction of flow-mediated dilation (FMD) after 8 weeks of treatment

Absolute change from baseline and between groups in serum lipids ratios8 weeks

Absolute change of lipids ratios after 8 weeks of treatment

Treatment tolerability8 weeks

Evaluation of treatment's tolerability assessed by patients with a Visual Analogue Scale (VAS) scale (with two end points representing 0 -no pain- and 10 -pain as bad as it could possibly be-) after 8 weeks of treatment

Absolute change from baseline and between groups in augumentation pressure8 weeks

Absolute change of augumentation pressure after 8 weeks of treatment assessed by Vicorder software

Absolute change from baseline and between groups in the index of central obesity8 weeks

Absolute change of the index of central obesity after 8 weeks of treatment

Absolute change from baseline and between groups in renal function8 weeks

Absolute change of creatinine and the estimated glomerular filtration rate (eGFR) after 8 weeks of treatment

Absolute change from baseline and between groups in lipids ratios4 weeks

Absolute change of lipids ratios after 4 weeks of treatment

Absolute change from baseline and between groups in liver transaminases and gamma glutamil transferase8 weeks

Absolute reduction of alanine aminotransferase, aspartate aminotransferase and gamma glutamil transferase after 8 weeks of treatment

Absolute change from baseline and between groups in augumentation index8 weeks

Absolute change of augumentation index after 8 weeks of treatment assessed by Vicorder software

Treatment acceptability8 weeks

Evaluation of patients' satisfaction assessed by a Visual Analogue Scale (VAS) scale (with two end points representing 0 -low acceptability- and 10 -high acceptability-) after 8 weeks of treatment

Percent change from baseline and between groups in the estimated body water content8 weeks

Percent change of the body water content after 8 weeks of treatment, assessed by bioelectrical impedance analysis (BIA)

Absolute change from baseline and between groups in estimated risk of cardiovascular disease8 weeks

Absolute change of the percent estimated risk of cardiovascular disease after 8 weeks of treatment

Trial Locations

Locations (1)

S. Orsola-Malpighi University Hospital

🇮🇹

Bologna, Italy

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