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Clinical Trials/NCT03416270
NCT03416270
Completed
Phase 2

ERtugliflozin triAl in DIabetes With Preserved or Reduced ejeCtion FrAcTion mEchanistic Evaluation in Heart Failure: "ERADICATE-HF"

University Health Network, Toronto3 sites in 2 countries34 target enrollmentJune 28, 2018

Overview

Phase
Phase 2
Intervention
Ertugliflozin
Conditions
Type 2 Diabetes Mellitus
Sponsor
University Health Network, Toronto
Enrollment
34
Locations
3
Primary Endpoint
Fractional Excretion of Lithium (FELi)
Status
Completed
Last Updated
12 months ago

Overview

Brief Summary

This study aims to elucidate the mechanisms whereby the SGLT2i "ertugliflozin" modifies cardiorenal interactions that regulate fluid volume and neurohormonal activation in patients with type 2 diabetes and heart failure (T2D-HF).

Detailed Description

Newer agents called sodium glucose co-transporter-2 inhibitors (SGLT2i) have been developed to improve glycemic control and lower hemoglobin A1c by increasing glycosuria. SGLT2i also reduce blood pressure and albuminuria in T2D - possibly through natriuresis. Importantly, a landmark trial "EMPA-REG OUTCOME" demonstrated that the SGLT2i "empagliflozin" is the first anti- hyperglycemic agent to reduce mortality and HF risk, and also to decrease the risk of progressive diabetic nephropathy. Similar benefits were also recently reported in the CANVAS Program trial with canagliflozin. Despite the benefits observed in these two pivotal trials, the mechanisms responsible for beneficial effects of SGLT2i in patients with T2D with respect to the development and/or worsening of HF are not currently known. In light of the results of EMPA-REG OUTCOME, the investigators aim to elucidate the mechanisms whereby the SGLT2i "ertugliflozin" modifies cardiorenal interactions that regulate fluid volume and neurohormonal activation in patients with T2D and HF (T2D-HF). The investigators will test the hypothesis that ertugliflozin increases proximal tubular natriuresis, thereby reducing plasma volume, without inducing significant renal vasoconstriction or activation of the sympathetic nervous system (SNS) (see below, Figure 1). The systematic understanding of the effects of SGLT2i in the setting of HF will enable the design of rational physiology based strategies to decrease the burden of HF, which could have major clinical and research implications internationally.

Registry
clinicaltrials.gov
Start Date
June 28, 2018
End Date
April 14, 2021
Last Updated
12 months ago
Study Type
Interventional
Study Design
Parallel
Sex
All

Investigators

Responsible Party
Principal Investigator
Principal Investigator

David Z.I. Cherney

Associate Professor of Medicine, Clinician Scientist

University Health Network, Toronto

Eligibility Criteria

Inclusion Criteria

  • Male or female subjects diagnosed with T2D ≥12 months prior to informed consent;
  • eGFR ≥30 ml/min/1.73m2;
  • Age \>18 years;
  • HbA1c 6.5%-10.5%;
  • Body Mass Index (BMI) 18.5-45.0 kg/m2;
  • Blood pressure ≤160/110 and ≥90/60 at screening,
  • Heart failure with New York Heart Association (NYHA) class 2-3 symptoms and ejection fraction ≥20%
  • Stable dose of maximally tolerated ACE inhibitor, angiotensin receptor blocker or renin inhibitor for at least 30 days
  • Stable diuretic dose for at least 30 days at the time of baseline physiological assessment
  • BNP levels at baseline ≥100 pg/ml (no atrial fibrillation), ≥200 pg/ml if in atrial fibrillation

Exclusion Criteria

  • Type 1 Diabetes;
  • Leukocyte and/or nitrite positive urinalysis that is untreated;
  • Severe hypoglycaemia within 2 months prior to screening;
  • History of brittle diabetes or hypoglycaemia unawareness based on investigator judgement;
  • Unstable coronary artery disease with acute coronary syndrome, percutaneous intervention or bypass surgery within 3 months;
  • Clinically significant valvular disease;
  • Congestive heart failure secondary to an infiltrative cardiomyopathic process (for example amyloid) or pericardial constriction;
  • Uncontrolled systemic hypertension (systolic blood pressure \>160 mmHg and/or diastolic blood pressure \>110) or systemic hypotension (systolic blood pressure \< 90/60 mmHg);
  • Bariatric surgery or other surgeries that induce chronic malabsorption;
  • Anti-obesity drugs or diet regimen and unstable body weight three months prior to screening;

Arms & Interventions

Ertuglifozin Treatment Arm

Ertugliflozin Tablets Total Dose 15mg (10mg + 5 mg) for 12 weeks

Intervention: Ertugliflozin

Placebo Arm

Placebo Matching Ertugliflozin Tablet for 12 weeks

Intervention: Placebo

Outcomes

Primary Outcomes

Fractional Excretion of Lithium (FELi)

Time Frame: Change in outcomes was measured acute (1 week minus baseline values) and chronic (12 weeks minus baseline values)

The difference in fractional excretion of lithium (FELi) with ertugliflozin vs. placebo. This was measured using exogenous lithium administration 12 hours before lithium excretion was measured in urine and blood.

Fractional Excretion of Sodium (FENa)

Time Frame: Change in outcomes was measured acute (1 week minus baseline values) and chronic (12 weeks minus baseline values)

The difference in fractional excretion of sodium (FENa) with ertugliflozin vs. placebo. This was measured using exogenous lithium administration 12 hours before sodium excretion was measured in urine and blood.

Change in Absolute Fractional Distal Sodium Reabsorption From Baseline (FELi-FENa)

Time Frame: Change in outcomes was measured acute (1 week minus baseline values) and chronic (12 weeks minus baseline values)

The difference in fractional excretion of lithium and fractional excretion of sodium (calculated by the difference between FELi and FENa) with ertugliflozin vs. placebo. This was measured using exogenous lithium administration 12 hours before sodium excretion was measured in urine and blood.

Secondary Outcomes

  • Heart Rate (HR)(chronic (12 weeks))
  • LV Ejection Fraction(chronic (12 weeks))
  • Carotid-femoral Pulse Wave Velocity(chronic (12 weeks))
  • Plasma Volume(chronic (12 weeks))
  • Extracellular Water(chronic (12 weeks))
  • Cardiac Output(chronic (12 weeks))
  • Systemic Vascular Resistance(chronic (12 weeks))
  • Blood Angiotensin II(chronic (12 weeks))
  • BNP(chronic (12 weeks))
  • Norepinephrine(chronic (12 weeks))
  • Urinary Adenosine(chronic (12 weeks))
  • Glomerular Filtration Rate (GFR)(Glomerular Filtration Rate (GFR, based on plasma iohexol clearance) will be measured at 12 weeks)
  • Effective Renal Plasma Flow (ERPF)(Effective Renal Plasma Flow (ERPF, based on paraaminohippurate plasma clearance) will be measured at 12 weeks)
  • Systolic Blood Pressure (SBP)(chronic (12 weeks))
  • Diastolic Blood Pressure (DBP)(chronic (12 weeks))

Study Sites (3)

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