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Clinical Trials/NCT03466385
NCT03466385
Unknown
Not Applicable

Nasal High Flow Versus Non-Invasive Ventilation in Patients With Acute Exacerbation of Chronic Obstructive Pulmonary Disease

Evangelismos Hospital3 sites in 1 country498 target enrollmentApril 15, 2018

Overview

Phase
Not Applicable
Intervention
Not specified
Conditions
Chronic Obstructive Pulmonary Disease
Sponsor
Evangelismos Hospital
Enrollment
498
Locations
3
Primary Endpoint
Frequency of treatment failure
Last Updated
6 years ago

Overview

Brief Summary

Although non-invasive ventilation (NIV) usage has increased significantly over time in COPD exacerbation, a great percentage of patients (~30%) present contraindications to NIV or cannot tolerate it. Nasal high flow (NHF) has been introduced for the management of hypoxemic respiratory failure in adults with favorable effects on ventilation and respiratory mechanics. The above mentioned NHF positive effects has been observed also in stable COPD patients with or without chronic hypercapnia.

In this study, the investigators hypothesize that NHF is not inferior to NIV for respiratory support in patients with COPD exacerbation and acute or acute on chronic hypercapnic respiratory failure.

Detailed Description

The use of non-invasive ventilation (NIV) in COPD exacerbation has increased significantly over time since it has been shown to improve acute respiratory acidosis (increases pH and decreases PaCO2), decrease respiratory rate, work of breathing, severity of breathlessness and therefore reduce mortality and intubation rates. Despite all these favorable effect, a great percentage of patients (\~30%) present contraindications to NIV or cannot tolerate it increasing thus the possibility of NIV failure and consequently intubation rates. Over the past decade, nasal high flow (NHF) oxygen therapy has been introduced for the management of hypoxemic respiratory failure in adults. NHF can generate high flow rates up to 60 L·min-1 and through this mechanism exerts its positive effects on respiratory mechanics, carbon dioxide washout, patient's respiratory rate and work of breathing. Although the above mentioned NHF positive effects has been observed also in stable COPD patients with or without chronic hypercapnia, NHF use in COPD exacerbation is questionable and only a few case reports studies have been published showing favorable effects of NHF on COPD exacerbation. In this study, the investigators hypothesize that NHF is not inferior to NIV for respiratory support in patients with COPD exacerbation and acute or acute on chronic hypercapnic respiratory failure.

Registry
clinicaltrials.gov
Start Date
April 15, 2018
End Date
December 31, 2021
Last Updated
6 years ago
Study Type
Interventional
Study Design
Parallel
Sex
All

Investigators

Sponsor
Evangelismos Hospital
Responsible Party
Principal Investigator
Principal Investigator

ISCHAKI ELENI

Consultant, ICU Department

Evangelismos Hospital

Eligibility Criteria

Inclusion Criteria

  • Patient with mild to moderate COPD exacerbation and the following characteristics persisting after initial medical therapy with bronchodilators and controlled oxygen therapy
  • 7,25\<pH\<7,35
  • PaCO2\>45mmHg
  • Ability to obtain written informed consent by the patient or patient's next of kin

Exclusion Criteria

  • severe facial deformity
  • Facial burns
  • Fixed upper airway obstruction
  • Criteria for imminent intubation and invasive mechanical ventilation (any of the following)
  • respiratory or cardiac arrest
  • gasping respiration
  • pH \<7.15
  • depressed consciousness (Glasgow Coma Score \<8)
  • psychomotor agitation inadequately controlled by sedation
  • massive aspiration

Outcomes

Primary Outcomes

Frequency of treatment failure

Time Frame: The first assessment will be held at 2 hours

As treatment failure is defined any need to switch to other treatment group because of discomfort, intolerance or failure to improve physiologic parameters (especially respiratory rate and ABG) despite optimum settings

Secondary Outcomes

  • Changes on respiratory rate(They will be assessed on baseline, before NIV or NHF initiation, and on 1, 2, 4, 6, 12, 24, 48 hours and once daily after day 3.)
  • Changes on arterial partial pressure of oxygen(They will be assessed on baseline, before NIV or NHF initiation, and on 1, 2, 4, 6, 12, 24, 48 hours and once daily after day 3.)
  • Changes on arterial partial pressure of carbon dioxide(They will be assessed on baseline, before NIV or NHF initiation, and on 1, 2, 4, 6, 12, 24, 48 hours and once daily after day 3.)
  • Changes on arterial pH(They will be assessed on baseline, before NIV or NHF initiation, and on 1, 2, 4, 6, 12, 24, 48 hours and once daily after day 3.)
  • Changes on respiratory accessory muscle use(They will be assessed on baseline, before NIV or NHF initiation, and on 1, 2, 4, 6, 12, 24, 48 hours and once daily after day 3.)
  • Patient's dyspnea(They will be assessed on baseline, before NIV or NHF initiation, and on 1, 2, 4, 6, 12, 24, 48 hours and once daily after day 3 (in those subjects whose neurological status allowed them to complete the evaluation))
  • Pulmonary complications(They will be assessed on baseline, before NIV or NHF initiation, and on 1, 2, 4, 6, 12, 24, 48 hours and once daily after day 3.)
  • Extrapulmonary complications(They will be assessed on baseline, before NIV or NHF initiation, and on 1, 2, 4, 6, 12, 24, 48 hours and once daily after day 3)

Study Sites (3)

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