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Clinical Trials/NCT01167257
NCT01167257
Completed
Phase 2

Comparative Study of the Efficacy and Safety of Liposome Encapsulated Botulinum Toxin-A (Lipotoxin) Versus Normal Saline Instillation in Treatment of Patients With Refractory Overactive Bladder Syndrome

Buddhist Tzu Chi General Hospital1 site in 1 country62 target enrollmentMay 2010

Overview

Phase
Phase 2
Intervention
Liposome encapsulated botulinum toxin A
Conditions
Overactive Bladder
Sponsor
Buddhist Tzu Chi General Hospital
Enrollment
62
Locations
1
Primary Endpoint
Mean Change of the Total Frequency Per 3 Days
Status
Completed
Last Updated
11 years ago

Overview

Brief Summary

Overactive bladder (OAB) is a bothered symptom syndrome. Traditional medication for OAB is antimuscarinic agent. However, adverse events such as dry mouth, constipation, blurred vision, and dizziness may prohibit patient to take this drug for OAB. Intravesical botulinum toxin A (BoNT-A) is a novel treatment however, BoNT-A can cause acute urinary retention and large postvoid residual. In this grant we will evaluate liquid liposome delivery of BoNT-A (Liposome encapsulated BoNT-A) into the bladder without the need for cystoscopic-guided needle injection for refractory OAB.

Detailed Description

Overactive bladder (OAB) is a symptom syndrome characterized by urgency frequency with or without urgency incontinence, usually no metabolic or anatomical disorders can be found and it may have great impact on quality of life. Traditional medication for OAB is antimuscarinic agent which targets at the muscarinic receptors. There are several adverse events such as dry mouth, constipation, blurred vision, and dizziness related to antimuscarinics, therefore, some patients cannot tolerated this treatment. Intravesical botulinum toxin A (BoNT-A) has recently emerged as novel treatment for OAB refractory to antimuscarinics, however, BoNT-A injection can cause acute urinary retention and large postvoid residual. Urinary tract infection usually occurred following large postvoid residual and urinary retention. If we can deliver BoNT-A through the urothelium to the suburothelial space, but not into the detrusor layer, we might have therapeutic effects on the urothelial sensory nerves without compromising the detrusor contractility. This treatment will enable us to prevent the undesired detrusor underactivity after BoNT-A injection, especially in the elderly patients who had impaired detrusor contractility and OAB. Liposomes are vesicles, composed of concentric phospholipid bilayers separated by aqueous compartments. Because liposomes adsorb to cell surfaces and fuse with cells, they are being used as vehicles for drug delivery and gene therapy. In this grant we will evaluate liquid liposome delivery of BoNT-A (Liposome encapsulated BoNT-A) into the bladder without the need for cystoscopic-guided needle injection for refractory OAB, and study the mechanism of action of intravesical liposomal drug delivery. If successful, we will leverage our technology transfer expertise and bring the science from the bench top to the bed side to apply for a physician sponsored Investigational New Drug (IND) trial using liposome-BoNT in patients with OAB or DO.

Registry
clinicaltrials.gov
Start Date
May 2010
End Date
December 2013
Last Updated
11 years ago
Study Type
Interventional
Study Design
Parallel
Sex
All

Investigators

Sponsor
Buddhist Tzu Chi General Hospital
Responsible Party
Principal Investigator
Principal Investigator

Hann-Chorng Kuo

Department of Urology

Buddhist Tzu Chi General Hospital

Eligibility Criteria

Inclusion Criteria

  • Adults with age of 20 years old or above
  • Patients with symptoms of urgency frequency and/or urge incontinence and a urgency severity scale (USS) of at least 2, with or without urodynamically proven detrusor overactivity (DO) (defined by the International Continence Society (ICS) recommendation as: spontaneous detrusor contraction occurring during bladder filling phase or occurring before uninhibited detrusor contraction voiding at bladder capacity in the urodynamic study)
  • Free of active urinary tract infection
  • Free of bladder outlet obstruction on enrollment
  • Free of overt neurogenic bladder dysfunction
  • Having been treated with antimuscarinic agents for at least 4 weeks without effect or with intolerable adverse effects
  • Patient has not been treated with bladder surgery for OAB, such as enterocystoplasty, that might affect the therapeutic effect of test drug
  • Patient can record voiding diary for the urinary frequency and urgency
  • Patient or his/her legally acceptable representative has signed the written informed consent form

Exclusion Criteria

  • Use of antimuscarinic agent and effective in treatment of lower urinary tract symptoms
  • Patients with severe cardiopulmonary disease and such as congestive heart failure, arrhythmia, poorly controlled hypertension, not able to receive regular follow-up
  • Patients with bladder outlet obstruction on enrollment
  • Patients with postvoid residual \>150 mL
  • Patients with uncontrolled confirmed diagnosis of acute urinary tract infection
  • Patients have laboratory abnormalities at screening including:
  • Alanine aminotransferase (ALT) \>3 x upper limit of normal range Aspartate aminotransferase (AST) \>3 x upper limit of normal range Patients have abnormal serum creatinine level \>2 x upper limit of normal range
  • Patients with any contraindication to be urethral catheterization during treatment
  • Female patients who is pregnant, lactating, or with child-bearing potential without contraception.
  • Myasthenia gravis, Eaton Lambert syndrome.

Arms & Interventions

Experimental arm

Liposome encapsulated BoNT-A ( mixed BOTOX 200 U/10 mL in Liposome 80 mg/40 mL) in single intravesical instillation Liposome encapsulated botulinum toxin A'

Intervention: Liposome encapsulated botulinum toxin A

Control arm

Normal saline 50 mL in single intravesical instillation Normal saline instillation'

Intervention: Normal saline instillation

Outcomes

Primary Outcomes

Mean Change of the Total Frequency Per 3 Days

Time Frame: Baseline to 4 weeks after initial treatment

Efficacy: Mean change of the total frequency per 3 days from baseline to 4 weeks after the treatment day based on the 3-day voiding diary. Change = Week 4 minus Baseline value

Secondary Outcomes

  • Mean Change of the Urgency Episodes Per 3 Days(Baseline to 4 weeks after initial treatment)
  • Mean Change of the Urgency Urinary Incontinence (UUI) Per 3 Days(Baseline to 4 weeks after initial treatment)
  • Net Change of the Overactive Bladder Symptom Score (OABSS)(Baseline to 4 weeks after initial treatment)
  • Net Change of the Functional Bladder Capacity (FBC)(Baseline to 4 weeks after initial treatment)
  • Net Change of the Maximum Flow Rate (Qmax)(Baseline to 4 weeks after initial treatment)
  • Net Change of the Postvoid Residual Volume (PVR)(Baseline and 1 month after initial treatment)
  • Net Change of the Urgency Severity Score (USS) Within 3 Days(Baseline to 4 weeks after initial treatment)
  • Net Change of the Global Response Assessment (GRA)(Baseline to 4 weeks after initial treatment)

Study Sites (1)

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