Skip to main content
Clinical Trials/NCT03492658
NCT03492658
Completed
Phase 4

Abatacept to Silence Anti-citrullinated Protein Antibody-expressing B Cells in Rheumatoid Arthritis (ASCARA).

Leiden University Medical Center1 site in 1 country46 target enrollmentMay 17, 2018

Overview

Phase
Phase 4
Intervention
Abatacept
Conditions
Rheumatoid Arthritis
Sponsor
Leiden University Medical Center
Enrollment
46
Locations
1
Primary Endpoint
Percentage of ACPA-expressing B cells that express the marker Ki-67
Status
Completed
Last Updated
3 years ago

Overview

Brief Summary

To investigate the effect of CTLA4-Ig (abatacept) on phenotype, transcriptional profile, B cell receptor usage and functional parameters of circulating B cells expressing anticitrullinated protein antibodies (ACPA) in patients with early, methotrexate-naïve, ACPA positive rheumatoid arthritis.

Detailed Description

B cells expressing anti citrullinated protein antibodies (ACPA) in patients with rheumatoid arthritis (RA) display an activated, proliferative phenotype. Experimental data indicate that ACPA and ACPA-expressing B cells are actively involved in driving the disease process in RA. The present study is based on the hypothesis that targeted intervention with CTLA4-Ig (abatacept) as a means to interfere with T cell help for B cells in early, active, ACPA-positive rheumatoid arthritis can reverse the activated, proliferative phenotype of citrullinated antigen-specific B cells.

Registry
clinicaltrials.gov
Start Date
May 17, 2018
End Date
April 6, 2022
Last Updated
3 years ago
Study Type
Interventional
Study Design
Parallel
Sex
All

Investigators

Sponsor
Leiden University Medical Center
Responsible Party
Principal Investigator
Principal Investigator

Hans Ulrich Scherer

Principal Investigator

Leiden University Medical Center

Eligibility Criteria

Inclusion Criteria

  • Each patient must:
  • have a diagnosis of rheumatoid arthritis according to the revised 2010 EULAR/ACR criteria for classification of RA
  • have a positive test for the presence of anti-citrullinated protein antibodies (ACPA) in serum as determined by routine clinical assay.
  • have adequate hematologic function (ANC ≥ 4000 cells/μL, platelet count ≥ 150000/μL, and hemoglobin ≥ 10 g/dL (corresponding to 6.2 mmol/L)
  • have serum creatinine concentrations \< 1.5 mg/dl and/or a normal creatinine clearance
  • if a female patient is of childbearing potential, agree to: comply with effective contraceptive measures, use adequate contraception since the last menses, use adequate contraception during the study, have a negative pregnancy test within one week of study entry
  • be willing to receive a booster vaccination against tetanus toxoid three to four weeks prior to randomization
  • be able and willing to give written informed consent prior to entry in the study

Exclusion Criteria

  • Any patient who has:
  • been previously treated with either abatacept and/or methotrexate or another csDMARD
  • been previously treated with a kinase inhibitor
  • been previously treated with rituximab or another B-cell depleting agent
  • been previously treated with a biological DMARD
  • received intra-articular or systemic glucocorticoid injections or has required treatment for acute RA flare (not being part of a regular therapeutic regimen) within four weeks prior to randomization or requires narcotic analgesics other than those accepted by the investigator for analgesia (e.g. paracetamol, codeine, tramadol)
  • been tested negative for anti citrullinated protein antibodies
  • contraindications for a booster vaccination against tetanus toxoid prior to randomization to the treatment arms; if a patient refuses booster vaccination but has detectable numbers of tetanus toxoid-specific B cells circulating in peripheral blood prior to the baseline visit, the patient can still be allowed to participate in the study at the judgement of the investigator.
  • evidence of any other major chronic inflammatory disease (i.e. psoriasis, psoriatic arthritis, spondyloarthritis or inflammatory bowel disease)
  • evidence of poorly controlled diabetes, history of clinically significant pulmonary disease including interstitial lung disease or methotrexate-induced lung disease, poorly controlled asthma or a history of severe life-threatening asthma attacks, history of active tuberculosis, history of latent tuberculosis without adequate medical treatment, liver cirrhosis or fibrosis, significant active infection or any underlying diseases that could predispose the subject to infections

Arms & Interventions

Combination therapy (MTX/abatacept)

Treatment with a combination of methotrexate (10 - 25 mg once weekly) and abatacept (125 mg subcutaneously once weekly) for 6 months, followed by methotrexate monotherapy (10 - 25 mg once weekly) for another 6 months.

Intervention: Abatacept

Combination therapy (MTX/abatacept)

Treatment with a combination of methotrexate (10 - 25 mg once weekly) and abatacept (125 mg subcutaneously once weekly) for 6 months, followed by methotrexate monotherapy (10 - 25 mg once weekly) for another 6 months.

Intervention: Methotrexate

Methotrexate (MTX) monotherapy

Treatment with methotrexate monotherapy (10 - 25 mg once weekly) for 12 months.

Intervention: Methotrexate

Outcomes

Primary Outcomes

Percentage of ACPA-expressing B cells that express the marker Ki-67

Time Frame: 24 weeks

Flow cytometry-based determination of the percentage of ACPA-expressing B cells that stain positive for Ki-67, circulating in peripheral blood of patients with early, ACPA-positive rheumatoid arthritis.

Secondary Outcomes

  • Change in disease activity(24 weeks)

Study Sites (1)

Loading locations...

Similar Trials