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The Role of Glucocorticoids to Maintain Energy Homeostasis During Starvation (Gluco-Starve)

Early Phase 1
Completed
Conditions
Glucocorticoid Effect
Interventions
Registration Number
NCT05919992
Lead Sponsor
Eleonora Seelig
Brief Summary

In a randomized, cross-over study, 20 healthy volunteers will receive a block and replace therapy that mimics physiological GC rhythm (metyrapone plus hydrocortisone) or placebo. Participants will undergo two identical fasting periods with each treatment. With the block and replace therapy, fasting-induced GC peak will be suppressed. Metabolic and autonomic parameters will be compared to reveal whether GCs mediate the physiological adaptions to caloric restriction.

Understanding acute effects of GCs upon caloric restriction is critical, since repetitive disruptions of GC secretion may become harmful in chronic conditions.

Detailed Description

Obesity is one of the major causes of morbidity and mortality worldwide. Achieving long-term weight loss is challenging, as the body counteracts weight loss to preserve energy by increasing appetite and lowering energy expenditure. These physiological defense mechanisms are the main obstacle to successful weight reduction in obese people.

Therefore, identifying the signals that defend body weight during caloric restriction is essential for developing new antiobesity drugs. Corticosteroids mediate the physiological defense to starvation in rodents. Whether cortisol has the same impact on humans is unknown.

Therefore, we investigate whether cortisol regulates the physiological adaptions to caloric restriction in humans.

The general objective of this project is to investigate whether cortisol mediates physiological adaptions to caloric restriction.

The primary objective is to test whether cortisol mediates the increased appetite during caloric restriction.

Secondary objectives are to test whether the cortisol response to caloric restriction affects satiation, satiety, energy expenditure, substrate utilization, blood pressure, weight, body composition, secretion of neuroendocrine hormones, lipids, glucose, ketone bodies, sympathetic nervous system activity, immune cells, and inflammatory markers.

This is a double-blind, randomized, placebo-controlled crossover study.

After screening, subjects will be randomized to two crossover 7-day study periods with a wash-out period of 28 days:

A) Participants will receive hydrocortisone 19.9 mg/d subcutaneously via a pump in a pulsed fashion (eight times/day) and metyrapone capsules per os (starting with a dose of 500 mg/d on day 1 to 3000mg/d on day 5, and then will be kept constant until day 7).

B) Participants will receive a placebo (0,9% NaCl solution) subcutaneously via a pump in a pulsed fashion and identical-looking placebo capsules per os with the same regimen as for metyrapone.

During both study periods, participants will undergo two days of caloric restriction.

Recruitment & Eligibility

Status
COMPLETED
Sex
Male
Target Recruitment
24
Inclusion Criteria
  • BMI 18.5 - 27 kg/m2
  • Weight stability for 6 months prior to the trial (+/- 2kg)
Exclusion Criteria
  • Previous medical history for any chronic condition in the last three months, active disease or abnormal physical examination as verified by a qualified physician.
  • Casual smoking (>6 cigarettes per day)
  • Frequent, heavy alcohol consumption (>30g/day)
  • Frequent, heavy caffeine consumption (>4 caffeinated drinks/day)
  • Regular physical exercise (>4hrs per week)
  • Shift workers
  • Participation in an investigational drug trial within the past two months
  • Intake of any drugs (prescribed, over the counter or recreational), within 48 hours of the study initiation
  • Intake of any steroids (including topical or inhaler) six month prior to the study
  • Known allergy to metyrapone or hydrocortisone
  • Inability or unwillingness to provide informed consent

Study & Design

Study Type
INTERVENTIONAL
Study Design
CROSSOVER
Arm && Interventions
GroupInterventionDescription
Metyrapone And HydrocortisoneMetyrapone 250 mg Oral TabletsDuring one of the study periods, subjects receive hydrocortisone 19.9 mg/d subcutaneously via a pump in a pulsed fashion (eight times/day) and metyrapone per os (starting with a dose of 500 mg/d, then the dose will be increased the next days until 3000mg/d is achieved).
Metyrapone And HydrocortisoneHydrocortisone 19.9mg s.c., pulsatile with a flow rate of 10μl/sDuring one of the study periods, subjects receive hydrocortisone 19.9 mg/d subcutaneously via a pump in a pulsed fashion (eight times/day) and metyrapone per os (starting with a dose of 500 mg/d, then the dose will be increased the next days until 3000mg/d is achieved).
PlaceboPlacebo 250 mg TabletsDuring the other study period, subjects receive placebo (0,9% NaCl solution) 19.9 mg/d subcutaneously via a pump in a pulsed fashion and the same dose of placebo tablets p.o instead of metyrapone
PlaceboPlacebo (0,9% NaCl solution)During the other study period, subjects receive placebo (0,9% NaCl solution) 19.9 mg/d subcutaneously via a pump in a pulsed fashion and the same dose of placebo tablets p.o instead of metyrapone
Primary Outcome Measures
NameTimeMethod
SatiationTwo 7-day intervention periods

Amount of food intake with ad libitum buffet

Secondary Outcome Measures
NameTimeMethod
Ketone bodiesTwo 7-day intervention periods

measured via blood sample

SatietyTwo 7-day intervention periods

Appetite rating by visual analog scale, minimum value 0, maximum value 100

Food preferenceTwo 7-day intervention periods

Amount of fat/ protein/carbohydrates consumed during ad libitum buffet

Energy expenditureTwo 7-day intervention periods

Basal metabolic rate, diet-induced thermogenesis

Substrate utilizationTwo 7-day intervention periods

Respiratory quotient

Blood pressureTwo 7-day intervention periods

Blood pressure

WeightTwo 7-day intervention periods

Body weight

Body compositionTwo 7-day intervention periods

measured with DEXA-Scans and body impedance analysis

Neuroendocrine hormonesTwo 7-day intervention periods

Leptin, thyroid hormones, insulin, c-peptide, growth hormone, IGF1, catecholamines, GLP-1, GIP, glucagon, PYY, CCK, ghrelin, GDF-15, cortisol total and free, ACTH, renin, aldosterone, pregnenolone, progesterone, 11-deoxycorticosterone, corticosterone, 18-hydroxycorticosterone, 17-hydroxypregnenolone, 17-hydroxyprogesterone, 11-deoxycortisol, oxytocin, FGF-21

LipidsTwo 7-day intervention periods

Total cholesterol, LDL-cholesterol, HDL-cholesterol, and triglycerides

GlucoseTwo 7-day intervention periods

measured via blood sample

Insulin sensitivityTwo 7-day intervention periods

measured via blood sample

Sympathetic nervous system activityTwo 7-day intervention periods

measured via ECG: Heart rate, interbeat interval, high-frequency activity, low-frequency activity, root mean square of successive differences

Immune cellsTwo 7-day intervention periods

Peripheral blood mononuclear cells (PBMCs)

Inflammatory markersTwo 7-day intervention periods

IL-6, IL-1RA, IL-8, CRP

Motivation to eatTwo 7-day intervention periods

clicking speed computer test

Pleasure from eatingTwo 7-day intervention periods

Fonts rating test

Measure of behavioural approach and behavioural inhibition systemTwo 7-day intervention periods

Questionnaire

Eating behaviour typeTwo 7-day intervention periods

Questionnaire

Trial Locations

Locations (1)

University Hospital Basel

🇨🇭

Basel, Basel-Stadt, Switzerland

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