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Clinical Trials/NCT00799396
NCT00799396
Completed
Phase 4

Pharmacogenomics of Anti-Platelet Interventions (The PAPI Study)

University of Maryland, Baltimore1 site in 1 country682 target enrollmentJuly 2006

Overview

Phase
Phase 4
Intervention
Clopidogrel
Conditions
Platelet Aggregation Inhibitors
Sponsor
University of Maryland, Baltimore
Enrollment
682
Locations
1
Primary Endpoint
Changes in Platelet Function in Response to Clopidogrel
Status
Completed
Last Updated
4 years ago

Overview

Brief Summary

One of the most common ways for preventing coronary heart disease (CHD) is to take aspirin or clopidogrel. However, studies have shown that not all people respond to these medications. The variance in treatment response may be linked to genetics. This study will examine the effects of aspirin and clopidogrel in a population whose genes are well known in order to determine the role that genes play in treatment responses.

Detailed Description

CHD is the leading cause of death in the United States. Anti-platelet agents lessen platelet aggregation and are used commonly to prevent recurrent CHD events. Two of the most common anti-platelet agents are aspirin and clopidogrel. However, up to 25% to 30% of people do not respond to these medications. Evidence indicates that treatment response may be related to genetics. The purpose of this study is to determine specific gene variants that predict response to aspirin and clopidogrel therapy. This study is part of a larger group of studies called the Pharmacogenomics Research Network (PGRN). Participants will include the Old Order Amish of Lancaster, Pennsylvania. They are well suited for genetic studies because they are a homogenous, closed, founder population. Participants will receive 300 mg of clopidogrel on the first day, then 75 mg of clopidogrel per day for the next 6 days. On the last day of clopidogrel treatment, participants will take a single dose of 324 mg aspirin. Participants will undergo platelet function tests before and after clopidogrel alone, and then again after taking clopidogrel plus aspirin. Using the gene variation profiles across the genome, researchers will analyze which genes correspond to treatment response.

Registry
clinicaltrials.gov
Start Date
July 2006
End Date
February 2012
Last Updated
4 years ago
Study Type
Interventional
Study Design
Single Group
Sex
All

Investigators

Responsible Party
Principal Investigator
Principal Investigator

Alan Shuldiner

Associate Dean for Personalized Medicine; Director, Program in Personalized and Genomic Medicine; Head, Division of Endocrinology, Diabetes and Nutrition

University of Maryland, Baltimore

Eligibility Criteria

Inclusion Criteria

  • Of Old Order Amish descent

Exclusion Criteria

  • Currently pregnant or less than 6 months have passed since delivery
  • Has a history of a bleeding disorder or major spontaneous bleed, such as peptic ulcer, epistasis, or intracranial bleed
  • Has severe hypertension, defined by a blood pressure above 160/95 mm Hg, making it unethical not to recommend prompt treatment
  • Takes medications that would affect the outcome(s) to be measured and cannot willingly and safely, in the opinion of the treating physician and study physician, discontinue these medications for 1 week prior to protocol initiation
  • Is taking vitamins or other supplements and is unwilling to discontinue their use for at least 1 week prior to study
  • Has a coexisting malignancy
  • Has a creatinine level greater than 2.0 mg/dl, aspartate transaminase (AST) or alanine transaminase (ALT) greater than two times the upper limit of normal, hematocrit less than 32%, or a thyroid-stimulating hormone (TSH) less than 0.4 or greater than 5.5 mIU/L
  • Has a bleeding disorder or history of gastrointestinal bleeding or other major bleeding episode
  • Is currently taking aspirin, clopidogrel, or other anti-coagulant, such as warfarin, heparin, or GPIIb/IIIa antagonists, and have conditions that might place them at increased risk from withdrawal of these medications 14 days prior to protocol initiation, including history of unstable angina, heart attack, angioplasty (including stent placement), coronary artery bypass surgery, atrial fibrillation, stroke or transient ischemic attacks, diabetes, or deep vein thrombosis or other thrombosis
  • Has polycythemia, or thrombocytosis, defined by a platelet count greater than 500,000

Arms & Interventions

Overall Study

Participants will receive clopidogrel treatment alone, followed by clopidogrel plus aspirin treatment on the last day of treatment.

Intervention: Clopidogrel

Overall Study

Participants will receive clopidogrel treatment alone, followed by clopidogrel plus aspirin treatment on the last day of treatment.

Intervention: Aspirin

Outcomes

Primary Outcomes

Changes in Platelet Function in Response to Clopidogrel

Time Frame: Measured at baseline, and after clopidogrel treatment

Baseline minus post clopidogrel/pre-aspirin platelet rich plasma (PRP) maximum aggregation.

Changes in Platelet Function in Response to Clopidogrel Plus Aspirin

Time Frame: Measured at baseline, and after clopidogrel plus aspirin treatment

Baseline minus post clopidogrel/post-aspirin platelet rich plasma (PRP) maximum aggregation

Study Sites (1)

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