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Metoprolol and Trimetazidine for Coronary Heart Disease With Angina

Not Applicable
Completed
Conditions
Coronary Heart Disease (CHD)
Interventions
Registration Number
NCT07074834
Lead Sponsor
Shiyan City Renmin Hospital
Brief Summary

This is a prospective, randomized study to evaluate the effectiveness and safety of combining metoprolol succinate with trimetazidine compared to standard therapy for patients with coronary heart disease (CHD) and angina pectoris. The study aims to assess the effects of the combination therapy on inflammatory biomarkers, clinical efficacy, angina symptoms, and left ventricular function over a 3-month treatment-period.

Detailed Description

Coronary heart disease (CHD) with angina pectoris is a significant cause of morbidity and mortality. While standard treatments like β-blockers (e.g., metoprolol succinate) are effective, many patients continue to experience symptoms. Inflammation is known to play a crucial role in the pathophysiology of CHD. This study was designed to investigate whether the addition of trimetazidine, a myocardial anti-ischemic agent that improves cellular energy metabolism, to standard metoprolol succinate therapy could offer superior benefits. This prospective study enrolled and randomized 102 patients with CHD and angina into two groups: a control group receiving routine drug therapy and a treatment group receiving routine therapy plus metoprolol succinate and trimetazidine. The primary objective was to compare the therapeutic efficacy, changes in angina attack frequency and severity, improvements in left ventricular function, and reductions in key inflammatory markers (IL-1β, TNF-α, hs-CRP, IL-18) between the two groups after 3 months of treatment. The study aims to provide evidence for this combination therapy as a safe and effective option for managing CHD with angina pectoris.

Recruitment & Eligibility

Status
COMPLETED
Sex
All
Target Recruitment
102
Inclusion Criteria
  • Diagnosis of CHD and angina pectoris based on CAD and angina pectoris guidelines.
  • History of previous myocardial infarction, confirmed by coronary angiography or coronary computed tomography angiography, with at least one coronary artery stenosis ≥50%.
  • Disease duration over 3 months.
  • Frequency of angina attacks in the past week was ≥3 times, with a severity grade of I, II, or III.
  • All patients signed an informed consent form.
Exclusion Criteria
  • Patients with mental disorders, drug allergies, acute myocardial infarction, electrolyte imbalances, important organ dysfunction, or cardiomyopathy.
  • Patients with severe immune or infectious diseases.
  • Those with stable symptoms or no symptoms after acute coronary syndrome.
  • Patients with severe arrhythmia, uncontrolled hypertension (SBP ≥180 mmHg or DBP ≥110 mmHg), pacemaker, or aortic dissection.
  • Pregnant or lactating women.
  • Patients requiring revascularization (e.g., multi-vessel disease, left main disease, or ischemic areas >10% of the left ventricle).
  • Patients who participated in other clinical trials within the past month.

Study & Design

Study Type
INTERVENTIONAL
Study Design
PARALLEL
Arm && Interventions
GroupInterventionDescription
Experimental: Treatment GroupMetoprolol Succinate Sustained-Release TabletsPatients were randomized to receive routine drug therapy plus metoprolol succinate combined with trimetazidine for 3 months.
Experimental: Treatment GroupTrimetazidinePatients were randomized to receive routine drug therapy plus metoprolol succinate combined with trimetazidine for 3 months.
Experimental: Treatment GroupRoutine Drug TherapyPatients were randomized to receive routine drug therapy plus metoprolol succinate combined with trimetazidine for 3 months.
Active Comparator: Control GroupRoutine Drug TherapyPatients were randomized to receive routine drug therapy for 3 months.
Primary Outcome Measures
NameTimeMethod
Change in Clinical Efficacy RateAt 3 months

Clinical efficacy was assessed based on symptom changes and reduction in angina attacks. Efficacy was categorized as "Marked effect" (symptoms nearly controlled, attacks decreased \>80%), "Effective" (symptoms partially improved, attacks decreased 50-80%), or "Ineffective". The total effective rate was calculated as (Marked effect + Effective) / Total cases × 100%.

Change in Frequency of Angina AttacksFrom Baseline to 3 months

The mean number of angina attacks per week was recorded and compared between groups.

Change in Serum Inflammatory MarkersFrom Baseline to 3 months

Changes in serum levels of Interleukin-1 beta (IL-1β), Tumor Necrosis Factor-alpha (TNF-α), and high-sensitivity C-reactive protein (hs-CRP) were measured using ELISA and immune transmission nephelometry.

Secondary Outcome Measures
NameTimeMethod
Change in Angina Pain ScoreFrom Baseline to 3 months

Pain severity was assessed using the Visual Analog Scale (VAS) with a score from 0 (no pain) to 10 (most severe pain).

Change in Duration of Angina EpisodesFrom Baseline to 3 months

The mean duration of angina attacks was recorded and compared.

Change in Left Ventricular Ejection Fraction (LVEF)From Baseline to 3 months

LVEF was measured by color Doppler ultrasound to assess left ventricular systolic function.

Change in Left Ventricular End-Diastolic Diameter (LVEDD)From Baseline to 3 months

LVEDD was measured by color Doppler ultrasound to assess cardiac remodeling.

Change in Left Ventricular End-Systolic Diameter (LVESD)From Baseline to 3 months

LVESD was measured by color Doppler ultrasound to assess cardiac remodeling.

Change in Serum Interleukin-18 (IL-18) levelsFrom Baseline to 3 months

Serum IL-18 levels were measured using ELISA.

Incidence of Adverse ReactionsOver 3 months

The number and percentage of patients experiencing adverse reactions, including gastrointestinal abnormalities, arrhythmias, or hypotension, were recorded.

Trial Locations

Locations (1)

Renmin Hospital, Hubei University of Medicine

🇨🇳

Shiyan, Hubei, China

Renmin Hospital, Hubei University of Medicine
🇨🇳Shiyan, Hubei, China

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