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Clinical Trials/NCT00648674
NCT00648674
Withdrawn
Phase 4

A Prospective, Single-Site Study to Identify and Characterize the Molecular Mechanisms of Apligraf in Non-healing Wounds of Subjects With Venous Leg Ulcers

Organogenesis0 sitesJuly 2008
ConditionsVaricose Ulcer

Overview

Phase
Phase 4
Intervention
Not specified
Conditions
Varicose Ulcer
Sponsor
Organogenesis
Primary Endpoint
To use the novel technology of microarrays to identify and characterize the molecular mechanisms through which Apligraf promotes healing of venous leg ulcers.
Status
Withdrawn
Last Updated
10 years ago

Overview

Brief Summary

Single site clinical study utilizing the novel technology of microarrays to identify and characterize the molecular mechanisms through which Apligraf promotes healing of venous leg ulcers.

Registry
clinicaltrials.gov
Start Date
July 2008
End Date
March 2010
Last Updated
10 years ago
Study Type
Interventional
Study Design
Single Group
Sex
Female

Investigators

Eligibility Criteria

Inclusion Criteria

  • Subject has non-infected partial or full-thickness venous leg ulcer for a duration of four weeks or greater which has not adequately responded to conventional therapy
  • Stage II or III ulcer as defined by the IAET/WOCN ulcer classification guide listed in Appendix IV.
  • Subject is female and 18 years of age or older.
  • Subject with venous leg ulcer (target ulcer) between 5 - 40 cm2 in size.
  • Sexually active females must be practicing a medically proven form of contraception during the course of the study period.
  • Subject or legal guardian must have read, understood and signed an institutional review board (IRB) approved Informed Consent Form.
  • Subject and/or legal guardian must be able and willing to follow study procedures and instructions.

Exclusion Criteria

  • Subject whose target ulcer has healed 60% or greater in area from post debridement (if applicable) Baseline Screen (Visit 1) to post debridement Day 0 (Visit 2) as determined by wound tracings.
  • Subjects who are being treated with VAC® (Vacuum Assisted Closure™) Therapy.
  • Subject has arterial disease as determined by an Ankle Brachial Index (ABI ) of \<0.
  • Subject with any systemic condition like uncontrolled diabetes mellitus (glycosylated HbA1C \> 12%), cancer (biopsy confirmed active malignancy), positive HIV test, or any disorder(s) that may compromise wound healing.
  • Subjects who are currently receiving, or have received at any time within one month prior to entry into the study, corticosteroids (\>15 mg/day), immunosuppressive agents, radiation therapy, hemodialysis or chemotherapy. Anticipated use of the above agents will exclude subjects from entry into the study.
  • Clinical vasculitis, severe rheumatoid arthritis, and other collagen vascular diseases.
  • Signs and symptoms of cellulitis or osteomyelitis.
  • Necrotic or avascular ulcer beds.
  • Venous leg ulcer with exposed bone, tendon or fascia.
  • Subject with the presence of recent infections in the area intended for treatment.

Outcomes

Primary Outcomes

To use the novel technology of microarrays to identify and characterize the molecular mechanisms through which Apligraf promotes healing of venous leg ulcers.

Time Frame: 2 weeks

Secondary Outcomes

  • Determine growth factors induced by Apligraf in the wound(2 weeks)
  • Safety endpoints assessed by monitoring adverse events(5 months)
  • Determine how venous leg ulcer (VLU) wounds transition from chronic to acute wound healing(2 weeks)
  • Examine how Apligraf rescues deficiencies of chronic wounds (VLU) and promotes transition from chronic to acute wound(2 weeks)
  • Determine growth factors suppressed by Apligraf in the wound(2 weeks)

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