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Chronotype-adapted Diet and Weight Loss

Not Applicable
Recruiting
Conditions
Overweight and Obesity
Interventions
Other: Dietary intervention - control group
Other: Dietary intervention - intervention group
Registration Number
NCT05941871
Lead Sponsor
Azienda Ospedaliero-Universitaria Careggi
Brief Summary

In humans, prolonged alterations in the circadian rhythm have been linked to cognitive impairments, premature ageing, and oncological and metabolic disorders such as diabetes and obesity. Obesity, in particular, is an ever-increasing condition with innumerable deleterious effects on human health. In recent years, studies have shown a relationship between a person's chronotype (morning or evening) and eating habits, as well as the importance of adapting these habits to physiological rhythms. Furthermore, it has been suggested that customising the caloric distribution of meals according to personal circadian rhythms may influence body weight and be one of the strategies to control overweight and obesity. In spite of the strong interest in this topic and the increasing number of observational studies conducted, there is currently a lack of intervention studies evaluating whether a low-calorie diet that takes into account the individual chronotype may be more effective than a standard low-calorie diet in the treatment of overweight and/or obesity.

Detailed Description

Background

Society has changed enormously in recent decades and this has had a strong impact on the processes regulating circadian rhythms, in particular the sleep-wake and fasting-eating cycles. The 'normalisation' of the environment, favoured by technological progress, has in fact caused light pollution, noise pollution, excessive thermoregulation, continuous work shifts and disordered eating, leading to an uncoordinated circadian cycle with consequences on physical and mental balance. In humans, prolonged alterations of the biological clock have been linked to cognitive disorders, premature ageing, and oncological and metabolic diseases such as diabetes and obesity. Obesity, in particular, is a condition with innumerable negative effects on human health.

In recent years, a new branch of nutritional research has aroused growing interest in the scientific community: this is chrono-nutrition, which combines elements of nutritional research with elements of chronobiology and studies the impact of eating times on health. The first to use the term "chrono-dystrophy" as a chronic desynchronisation of circadian rhythms were Erren and colleagues, who in their work reported how a loss of synchronisation between environmental signals and physiological processes can lead to alterations in the communication between the central nervous system and peripheral clocks and a change in the subject's metabolism. Subsequently, numerous studies have evaluated the impact of the thirteen dimensions of eating behaviour - timing, frequency and regularity - on health, hypothesising a possible role of the individual circadian rhythm, or chronotype, on the risk of developing overweight and/or obesity. Recent data have demonstrated a relationship between a person's chronotype (morning or evening) and eating habits, as well as the importance of adapting these habits to physiological rhythms. Furthermore, it has been suggested that customising the caloric distribution of meals according to personal circadian rhythms may influence body weight and be one of the strategies to control overweight and obesity. Indeed, recent research has shown that calories ingested at different times of the day have different effects on energy utilisation, leading to differential weight loss, even in the presence of isocaloric quantities.

Despite the strong interest in this topic and the increasing number of observational studies conducted, there is currently a lack of intervention studies evaluating whether a dietary regimen can be used to control body weight. Evidence to date suggests that in order to increase the effectiveness of low-calorie diets, it may be of great interest to consider not only patients' daily energy expenditure but also their circadian preferences. Overall, chrono-nutrition could mediate the effects between sleep, diet and urbanisation, but further research is needed to elucidate the precise physiological and metabolic mechanisms underlying this phenomenon, the importance of chronotype for metabolic health and its impact on public health.

Objectives of the study

The objectives of the study are to compare the effects of a diet with a daily calorie distribution adapted to the individual chronotype with a control diet with a conventional daily calorie distribution. The primary outcome is weight change from baseline. Secondary outcomes are changes in body mass index (BMI), percentage of fat mass, biochemical parameters and gut microbiota profile.

Recruitment & Eligibility

Status
RECRUITING
Sex
All
Target Recruitment
150
Inclusion Criteria
  • overweight or obese condition (BMI≥25 kg/m2)
  • age between 18 and 65 years
  • willing to give informes consent
Exclusion Criteria
  • chronic illnesses or unstable conditions (e.g. cancer, kidney or liver disease, inflammatory-intestinal disease, cognitive decline, psychiatric disease)
  • drug therapies (use of corticosteroids, antidiabetic drugs)
  • pregnancy or intention to become pregnant in the next 12 months
  • breastfeeding
  • current or recent (last 3 months) adoption of a low-calorie diet

Study & Design

Study Type
INTERVENTIONAL
Study Design
PARALLEL
Arm && Interventions
GroupInterventionDescription
ControlDietary intervention - control groupGroup that will follow a low-calorie diet with a standard daily energy distribution for 4 months
InterventionDietary intervention - intervention groupGroup following a low-calorie diet with a different daily energy distribution according to their chronotype for 4 months
Primary Outcome Measures
NameTimeMethod
Weight change4 months

Measurement of body weight change from baseline in kg

Secondary Outcome Measures
NameTimeMethod
LDL-cholesterol changes4 months

Measurement of LDL cholesterol change from baseline in mg/dL

Body mass index (BMI) changes4 months

Measurement of BMI change from baseline. Weight and height will be combined to report BMI in kg/m\^2

Aspartate transaminase changes4 months

Measurement of aspartate transaminase change from baseline in U/l

Urea changes4 months

Measurement of urea change from baseline in mg/dL

Uric acid changes4 months

Measurement of uric acid change from baseline in mg/dL

HDL-cholesterol changes4 months

Measurement of HDL cholesterol change from baseline in mg/dL

Triglycerides changes4 months

Measurement of triglycerides change from baseline in mg/dL

Alanine transaminase changes4 months

Measurement of alanine transaminase change from baseline in U/L

Gamma gamma-glutamyl transferase changes4 months

Measurement of gamma-glutamyl transferase change from baseline in U/L

Total cholesterol changes4 months

Measurement of total cholesterol change from baseline in mg/dL

Fat mass changes4 months

Measurement of fat mass change from baseline. Percentage of fat mass will be assessed using the Akern bioelectrical impedance analyser (model SE 101)

Fasting Blood Glucose changes4 months

Measurement of blood glucose concentration change from baseline in mg/dL

Creatinine changes4 months

Measurement of creatinin change from baseline in mg/dL

Gut microbiota changes4 months

Measurement of gut microbiota profile change from baseline. Each subject will be asked for a stool sample at the start of the study and at the end after 4 months in order to analyse the composition of the gut microbiota and short-chain fatty acids production

Trial Locations

Locations (1)

Unit of Clinical Nutrition, University Hospital of Careggi

🇮🇹

Florence, Italy

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