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Clinical Trials/NCT03753334
NCT03753334
Active, not recruiting
Phase 2

Étude Pilote randomisée de Phase IIB, contrôlée Par placébo, évaluant l'Effet thérapeutique d'Une supplémentation en Omega-3 (Principalement EPA) Chez Des Patients en récidive Biochimique ou en Progression du Cancer de la Prostate.

CHU de Quebec-Universite Laval1 site in 1 country40 target enrollmentJuly 10, 2017
ConditionsProstate Cancer

Overview

Phase
Phase 2
Intervention
Not specified
Conditions
Prostate Cancer
Sponsor
CHU de Quebec-Universite Laval
Enrollment
40
Locations
1
Primary Endpoint
Prostate-specific antigen (PSA) doubling time from baseline to 12 months.
Status
Active, not recruiting
Last Updated
4 months ago

Overview

Brief Summary

Prostate cancer biochemical recurrence (BCR) occurs in 20-50% of patients following radical prostatectomy or radiotherapy. Due to significant risk of side effects and uncertainty about the benefits, physicians and patients are seeking alternatives to delay androgen deprivation therapy (ADT) for non-metastatic BCR. Long-chain omega-3 fatty acids (LCn3), mainly found in seafood and fatty fish, have beneficial effects against prostate cancer in pre-clinical experimental studies and randomized clinical trials of intermediate prostate cancer outcomes. The current observational evidence also supports testing LCn3 in prostate cancer patients. LCn3 have beneficial effects on inflammation, cardiovascular, psychological, and other outcomes, contrasting sharply with ADT-associated side effects.

Investigators propose to conduct a pilot randomized placebo-controlled trial to determine the effects over one year of an innovative LCn3 supplement (5g of omega-3-rich fish oil daily, including 4g of monoglycerides eicosapentaenoic acid (MAG-EPA)) in 40 men experiencing BCR or prostate cancer progression after a curative treatment.

This project proposes a simple intervention by dietary supplementation that could eventually help to prevent or delay ADT-related side effects and thus could contribute to diminish the heavy individual and societal burden of prostate cancer. The clinical data generated by this pilot trial will serve as basis for a larger-scale phase II clinical trial.

Registry
clinicaltrials.gov
Start Date
July 10, 2017
End Date
December 1, 2026
Last Updated
4 months ago
Study Type
Interventional
Study Design
Parallel
Sex
Male

Investigators

Sponsor
CHU de Quebec-Universite Laval
Responsible Party
Sponsor

Eligibility Criteria

Inclusion Criteria

  • Patients must have a histologically or cytologically confirmed history of adenocarcinoma of the prostate.
  • Patients must have a PSA failure defined as PSA of \>= 0.5 ng/ml that has increased above nadir following radical prostatectomy (RP); or a PSA increase of 2.0 above post-therapy nadir after radiotherapy (RT); or a PSA increase between 0.05-0.49 ng/ml that has increased above nadir following RP. The maximal PSA value at enrolment must be \<5.0 ng/mL after RP and \<6 ng/mL after RT.
  • PSA value must be increasing based on three consecutive measurements each separated by at least 4 weeks prior to enrolment to this study.
  • Patients may have received any number of local therapies (RP, external beam RT or brachytherapy).
  • Provide written informed consent.

Exclusion Criteria

  • Patients with evidence of metastatic disease.
  • Patients who have received prior cytotoxic chemotherapy for recurrent disease.
  • Patients currently receiving biological response modifiers, or corticosteroids.
  • Uncontrolled intercurrent illness including, but not limited to, ongoing active infection, symptomatic congestive heart failure, unstable angina pectoris, cardiac arrhythmia, or psychiatric illness or social situations that would limit compliance with study requirements.
  • Use of omega-3 or any other dietary supplements for the previous 3 months and during study is not allowed.
  • Known allergy to fish or shellfish or sunflower.

Outcomes

Primary Outcomes

Prostate-specific antigen (PSA) doubling time from baseline to 12 months.

Time Frame: 12 months

Efficacy of a one-year MAG-EPA supplementation versus placebo on PSA kinetics will be evaluated based on the comparison of PSA doubling time from baseline to 12 months. The investigators will measure PSA level every three months and calculate PSA doubling time at 12 months (using a linear regression approach) after randomisation using the randomisation PSA value as the starting point. PSA slope will be defined as the linear regression line of the natural log of PSA (in ng/mL) against time (in months). PSA doubling time will be defined as the natural log of 2 divided by the PSA slope.

Secondary Outcomes

  • Fatty acid profiles in red blood cells, changes relative to baseline (time 0).(3, 6, 9,12 months)
  • Modulation of the Quality of life related to Sleep, changes relative to baseline (time 0) and between arms.(3, 6, 9, 12 months)
  • Change in Inflammatory mediators levels(0, 3, 12 months)
  • Modulation of the Quality of life related to Cognitive Function, changes relative to baseline (time 0) and between arms.(3, 6, 9,12 months)
  • Modulation of the Quality of life related to Prostate Symptoms, changes relative to baseline (time 0) and between arms.(0, 3, 6, 9, 12 months)
  • Modulation of the Quality of life related to Anxiety and Depression, changes relative to baseline (time 0) and between arms.(0, 3, 6, 9, 12 months)
  • Modulation of the Quality of life related to Health, changes relative to baseline (time 0) and between arms.(0, 3, 6, 9, 12 months)

Study Sites (1)

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