MedPath

Oral Metformin for Treatment of ABCA4 Retinopathy

Phase 1
Recruiting
Conditions
Stargardt Disease
ABCA4 Retinopathy
Retinal Dystrophy
Retinal Degeneration
Interventions
Registration Number
NCT04545736
Lead Sponsor
National Eye Institute (NEI)
Brief Summary

Background:

ABCA4 retinopathy is a genetic disease in which the ABCA4 protein is absent or faulty. It can cause waste material to collect in the eye and may cause cells to die. The cell death can lead to vision loss. Researchers want to see if an oral drug called metformin can help.

Objective:

To see if metformin is safe and possibly helps to slow the rate of ABCA4 retinopathy.

Eligibility:

People age 12 and older who have ABCA4 retinopathy and have problems with their vision.

Design:

Participants will be screened under a separate protocol.

Participants will have a medical and family history. They will complete a questionnaire about their vision and daily activities. They will have a physical exam. They may have blood drawn through a needle in the arm.

Participants will have an eye exam. Their pupils may be dilated with eye drops. Their retina may be photographed.

Participants will have a visual field test. They will sit in front of a large dome and press a button when they see a light within the dome.

Participants will have an electroretinogram. It examines the function of the retina. They will sit in the dark for 30 minutes. Then their eyes will be numbed with eye drops. They will wear contact lenses that can sense signals from the retinas. They will watch flashing lights.

Participants will have optical coherence tomography. This non-invasive procedure makes pictures of the retina.

Participants will have fundus autofluorescence. A bright blue light will be shone into their eye.

Participants will take metformin by mouth for 24 months.

Participants will have study visits every 6 months. Participation will last for at least 36 months....

Detailed Description

Study Description:

ABCA4 retinopathy is an autosomal recessive progressive retinal dystrophy that leads to retinal pigment epithelium (RPE) and photoreceptor degeneration, with consequent central visual loss. A treatment that either reduces the rate of lipofuscin accumulation or improves the clearance of lipofuscin in the RPE could potentially slow the degeneration associated with this disease. Metformin hydrochloride is a well-characterized, commonly prescribed oral anti-diabetic medication that acts by suppressing liver gluconeogenesis and increasing peripheral insulin sensitivity.

An additional effect of metformin is to increase macroautophagy via the Mammalian target of rapamycin complex 1 (mTORC1)/AMP-activated Kinase (AMPK) pathway; stimulation of this pathway would be predicted to enable the RPE to more efficiently handle lipofuscin. This suggests an association between metformin use and slowing of retinal degeneration. The objective of this study is to investigate the safety and potential efficacy of oral metformin in slowing the rate of change in photoreceptor degeneration in ABCA4 retinopathy.

Objectives:

The objective of this study is to investigate the safety and potential efficacy of oral metformin in slowing the rate of change in photoreceptor degeneration in ABCA4 retinopathy.

Endpoints:

Primary Endpoint: The difference in growth rate of square-root transformed area of EZ band loss (square-root AreaEZloss), from OCT, between the pre-treatment phase and 24 month metformin treatment phase.

Secondary Endpoints: Proportion of participants with a 30% reduction in growth rate of square-root AreaEZloss during the treatment phase compared to the pre-treatment phase, changes from baseline to Month 24 in Best-Corrected Visual Acuity (BCVA) total letters read, perimetry, and color fundus photography measurements and the change in rate of area of atrophy enlargement between the pre-treatment and 24-month metformin treatment phase.

Recruitment & Eligibility

Status
RECRUITING
Sex
All
Target Recruitment
56
Inclusion Criteria
  1. A growth rate of square-root AreaEZloss > 0.025 mm/year based on calculation from natural history data.

    Qualifying Eye

Exclusion Criteria
  1. Retinal degeneration has advanced beyond a point where reliable measurement of the integrity of the IS-OS on OCT is possible.

Study & Design

Study Type
INTERVENTIONAL
Study Design
SINGLE_GROUP
Arm && Interventions
GroupInterventionDescription
MetforminMetformin hydrochlorideOral administration of metformin
Primary Outcome Measures
NameTimeMethod
The difference in growth rate of square-root transformed area of EZ band loss (vAreaEZloss)Pre-treatment, Baseline, Month 24

The difference in growth rate of vAreaEZloss, from OCT, between the pre-treatment phase and treatment phase.

Secondary Outcome Measures
NameTimeMethod
Proportion of participants with a 30% reduction in growth rate of vAreaEZlossPre-treatment, Baseline, Month 24

Proportion of participants with 30% reduction in growth rate of vAreaEZloss, from OCT, during the treatment phase compared to the pre-treatment phase.

Number and severity of adverse eventsongoing throughout study

The number and severity of systemic and ocular toxicities, adverse events and infections by severity, type and assessed relatedness to the IP throughout the study period.

Changes in best corrected visual acuity (BCVA)Baseline, Month 24

The change in BCVA total letters read from baseline to Month 24.

Change in rate of area of atrophy enlargementPre-treatment, Baseline, Month 24

The difference in rate of region of atrophy measurements from FAF between the pre-treatment phase and treatment phase.

Change in perimetry and color fundus photography measurementsBaseline, Month 24

The change in perimetry and color fundus photography from baseline to Month 24.

Trial Locations

Locations (2)

National Institutes of Health Clinical Center

🇺🇸

Bethesda, Maryland, United States

University of Michigan

🇺🇸

Ann Arbor, Michigan, United States

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