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CIrculating Tumor DNA for Monitoring Response to First Line Chemotherapy in Unresectable PANcreatic Cancer

Not Applicable
Completed
Conditions
Pancreatic Cancer
KRAS Mutation-Related Tumors
Circulating Tumor DNA
Interventions
Diagnostic Test: Blood samples
Registration Number
NCT04560270
Lead Sponsor
Poitiers University Hospital
Brief Summary

With an incidence of more than 11,600 new cases per year in France and an annual number of deaths close to the incidence rate, adenocarcinoma of the pancreas is a public health problem.

The aim of this study is to assess the predictive value of response to the 1st line of chemotherapy of mutated KRAS ctDNA (circulating tumor DNA) in unresectable metastatic or locally advanced pancreatic adenocarcinomas.

Detailed Description

With an incidence of more than 11,600 new cases per year in France and an annual number of deaths close to the incidence rate, adenocarcinoma of the pancreas is a public health problem especially since there is a significant increase in its incidence. incidence (+ 417% between 1980 and 2012).

Most often diagnosed late, pancreatic adenocarcinoma is managed at a metastatic stage in 60 to 70% of cases with a very poor prognosis (8.7 to 11.1 months median survival with current chemotherapies). The first line of chemotherapy therefore represents a major issue in the management of these unresectable patients. There are few predictive markers of response to chemotherapy in pancreatic adenocarcinoma. It is conventionally evaluated by scanner every 2 to 3 months. The response to chemotherapy is associated with a good prognosis while non-response has a poor prognosis and requires a 2nd line of treatment if the patient is able to receive it.

A KRAS mutation is present in approximately 70-90% of pancreatic adenocarcinomas. Its research on tissue sampling (fine needle aspiration or anatomo-pathological specimen) is not carried out routinely because no prognostic or predictive value of KRAS mutations has been demonstrated. New high-throughput DNA sequencing techniques have been developed and now allow a blood sample to detect and quantify circulating tumor DNA (ctDNA), including KRAS mutations.

Very few studies have investigated the change in cDNA levels during 1st line chemotherapy in unresectable pancreatic adenocarcinoma.

The aim of this study is to assess the predictive value of response to the 1st line of chemotherapy of mutated KRAS cDNA in unresectable metastatic or locally advanced pancreatic adenocarcinomas.

Recruitment & Eligibility

Status
COMPLETED
Sex
All
Target Recruitment
69
Inclusion Criteria
  • Proven pancreatic adenocarcinoma (histology or cytology)
  • Metastatic or locally advanced unresectable
  • With thoraco-abdomino-pelvic scanner less than a month old
  • Chemotherapy treatment regardless of the protocol
  • Patients benefiting from a Social Security scheme or benefiting through the intermediary of a third party
  • Informed consent signed by the patient after clear and fair information about the study
Exclusion Criteria
  • Linguistic or psychological refusal or inability to understand and / or sign the informed consent
  • History of cancer in the 5 years preceding inclusion
  • Patient who has already received chemotherapy or radiotherapy for pancreatic cancer.
  • Immediately resectable tumor

Study & Design

Study Type
INTERVENTIONAL
Study Design
SINGLE_GROUP
Arm && Interventions
GroupInterventionDescription
Only armBlood samplesBlood samples to analyze ctDNA
Primary Outcome Measures
NameTimeMethod
Correlation of the ctDNA level to the response to chemotherapy3 months

Response to chemotherapy was evaluated with RECIST criteria 1.1 on the first CT scan

Secondary Outcome Measures
NameTimeMethod
Overall survival6 months after last patients inclusion

Correlation between variation of ctDNA and overall survival

Progression free survival6 months after last patients inclusion

Correlation between variation of ctDNA and progression free survival

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