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临床试验/NCT03708419
NCT03708419
已完成
不适用

Personalized Glucose Optimization Through Nutritional Intervention

Maastricht University Medical Center2 个研究点 分布在 1 个国家目标入组 242 人2018年6月4日

概览

阶段
不适用
干预措施
未指定
疾病 / 适应症
Obesity
发起方
Maastricht University Medical Center
入组人数
242
试验地点
2
主要终点
Disposition index
状态
已完成
最后更新
4年前

概览

简要总结

Maintaining well-controlled blood glucose concentrations is essential in the prevention of chronic cardiometabolic diseases. The blood glucose response to dietary and/or lifestyle patterns may vary between individuals. Insulin resistance in specific metabolic organs such as skeletal muscle, adipose tissue or the liver may underlie differential blood glucose responses.

This dietary intervention study aims to obtain insight into the metabolic and lifestyle determinants of postprandial blood glucose responses, and to establish the effect of macronutrient manipulation of a 12-week dietary intervention on blood glucose homeostasis in metabolically different subgroups an its relationship to physical and mental performance and well-being.

详细描述

Study design: this study is a double-blinded, randomised, controlled, parallel design dietary intervention study. The study will be conducted at Maastricht University and Wageningen University and Research, the Netherlands. Study population: the study population will consist of 240 men and women between 40-75 years old, with a BMI 25-40 kg/m2. Participants will be either muscle insulin resistant (MIR) or liver insulin resistant (LIR), as classified by an oral glucose tolerance test (OGTT) during the screening procedure. A subgroup of 80 participants will be selected for detailed metabolic phenotyping. Intervention: for 12 weeks, participants will receive either a diet optimal for MIR (high in mono-unsaturated fatty acids) or a diet optimal for LIR (high in protein and fiber, low in fat) with respect to changes in disposition index. Participants will be randomly allocated to one of the two diets. Detailed laboratory and daily life phenotyping will be done pre- and post intervention.

注册库
clinicaltrials.gov
开始日期
2018年6月4日
结束日期
2021年11月29日
最后更新
4年前
研究类型
Interventional
研究设计
Parallel
性别
All

研究者

责任方
Sponsor

入排标准

入选标准

  • BMI 25 to \<40 kg/m2
  • Predominantly muscle (MIR) or liver (LIR) insulin resistant
  • Weight stability for at least 3 months (+/- 3 kg)

排除标准

  • Pre-diagnosis of type 1 or type 2 diabetes mellitus
  • Renal or hepatic malfunctioning (pre-diagnosis or determined based on alanine aminotransferase (ALAT), aspartate aminotransferase (ASAT) and creatinine values)
  • Gastrointestinal diseases or abdominal surgery (allowed i.e.: appendectomy, cholecystectomy)
  • Food allergies, intolerances (including gluten/lactose intolerance) and/or dietary restrictions interfering with the study (including special diets, vegetarians and eating disorders)
  • Cardiovascular diseases (e.g. heart failure) or cancer (e.g. non-invasive skin cancer allowed)
  • High blood pressure (untreated \>160/100 mmHg, drug-regulated \>140/90 mmHg)
  • Diseases affecting glucose and/or lipid metabolism (e.g. pheochromocytoma, Cushing's syndrome, acromegaly)
  • Anemia defined as hemoglobin (Hb) men \<8.5 and women \<7.5 mmol/l
  • Diseases with a life expectation shorter than 5 years
  • Major mental disorders

结局指标

主要结局

Disposition index

时间窗: Change from baseline at week 12 dietary intervention

The primary objective of this study is to establish the effect of a metabolically targeted, optimal versus suboptimal macronutrient manipulated 12-week dietary intervention on the change in disposition index, a composite marker of first phase insulin secretion and insulin sensitivity during a 2-hour 7-points oral glucose tolerance test (OGTT). Disposition index will be calculated as follows: \[Insulin sensitivity index (ISI) \* (AUC30 min insulin / AUC30 min glucose)\], where AUC30 min is the area under the curve between 0 and 30 minutes of the OGTT for insulin (pmol/l) and glucose (mmol/l), respectively, and ISI is defined as: \[10,000 ÷ square root of (fasting plasma glucose (mmol/l) x fasting insulin (pmol/l)) x (mean glucose (mmol/l) x mean insulin (pmol/l))\]. Higher values represent a higher insulin sensitivity.

次要结局

  • Mean 24h glucose concentrations(Change from baseline at week 12 dietary intervention)
  • Self-reported fatigue(Change from baseline at week 12 dietary intervention)
  • Self-reported physical activity(Change from baseline at week 12 dietary intervention)
  • Subcutaneous adipose tissue biopsy(Change from baseline at week 12 dietary intervention)
  • Intervention effects on all above outcomes within the LIR and MIR group.(Change from baseline at week 12 dietary intervention)
  • DNA analysis(Baseline)
  • Insulin sensitivity(Change from baseline at week 12 dietary intervention)
  • Blood pressure(Change from baseline at week 12 dietary intervention)
  • Fasting blood lipid spectrum(Change from baseline at week 12 dietary intervention)
  • Substrate oxidation(Change from baseline at week 12 dietary intervention)
  • Oral microbiota composition(Change from baseline at week 12 dietary intervention)
  • Self-reported sleep quality over a 1 month period(Change from baseline at week 12 dietary intervention)
  • Self-reported intestinal health(Change from baseline at week 12 dietary intervention)
  • Glucose incremental area under the curve (iAUC)(Change from baseline at week 12 dietary intervention)
  • Glucose tolerance(Change from baseline at week 12 dietary intervention)
  • Muscle insulin sensitivity(Change from baseline at week 12 dietary intervention)
  • Hepatic insulin sensitivity(Change from baseline at week 12 dietary intervention)
  • Hip circumferences(Change from baseline at week 12 dietary intervention)
  • Fasting circulating metabolic markers(Change from baseline at week 12 dietary intervention)
  • Self-reported self efficacy in physical activity(Change from baseline at week 12 dietary intervention)
  • Self-reported daytime sleepiness(Change from baseline at week 12 dietary intervention)
  • Self-reported eating rate(Change from baseline at week 12 dietary intervention)
  • The frequency and duration of hypo- and hyperglycemia(Change from baseline at week 12 dietary intervention)
  • Body composition(Change from baseline at week 12 dietary intervention)
  • Waist circumference(Change from baseline at week 12 dietary intervention)
  • Body fat distribution(Change from baseline at week 12 dietary intervention)
  • Self-reported sleep behaviour(Change from baseline at week 12 dietary intervention)
  • Skeletal muscle biopsy(Change from baseline at week 12 dietary intervention)
  • Physical activity patterns(Change from baseline at week 12 dietary intervention)
  • Postprandial circulating metabolic markers(Change from baseline at week 12 dietary intervention)
  • Energy expenditure(Change from baseline at week 12 dietary intervention)
  • Fecal microbiota composition(Change from baseline at week 12 dietary intervention)
  • Self-reported perceived stress(Change from baseline at week 12 dietary intervention)
  • Self-reported sedentary behaviour(Change from baseline at week 12 dietary intervention)
  • Cognitive performance(Change from baseline at week 12 dietary intervention)
  • Carotid artery reactivity(Change from baseline at week 12 dietary intervention)
  • Self-reported quality of life(Change from baseline at week 12 dietary intervention)
  • Advanced glycation end-product (AGE) accumulation(Change from baseline at week 12 dietary intervention)
  • Fasting immune metabolism (PBMCs)(Change from baseline at week 12 dietary intervention)
  • Food preferences(Change from baseline at week 12 dietary intervention)

研究点 (2)

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