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Anti-BK Virus Immune Response and Kidney Transplantation

Completed
Conditions
Kidney Transplantation
Interventions
Other: biological parameters
Registration Number
NCT01109186
Lead Sponsor
Nantes University Hospital
Brief Summary

BK virus infections are very frequent during months following a kidney transplantation: a viral reactivation is observed for almost 50% of patients during first year. This reactivation leads to a viremia for 10 to 15% of patient during this same period. The most frequent complication is interstitial nephritis for 2 to 8% of patients (27 patients representing 2.7% during 6 years in Nantes).

An intensive et persisting viral replication, assessed by detection of high blood viral load, could evolved to a viral nephropathy which lead to a very pejorative functional issue for the graft.

Biological follow-up of these infections lay on the measures of viral load. Their positivity must alert the physician and lead him to modulate immunosuppressive treatment.

Actually, there is no real consensus about the modalities of pharmacological immunosuppression decrease (decrease dose or change of molecule).

Specific lymphocytic anti-BKv evaluated on several cohorts of patients permit to prove:

* weakness of immune cellular response for patient with high viremia

* increase of this response when viral load decrease These studies laid on detection of INFg synthesis by Elispot after stimulation with viral antigens and in vitro cellular expansion.

New prospective and longitudinal data comparing the immune cellular response (systematic and early) after graft between patients controlling or not BKv infection are necessary to improve the comprehension of illness natural history.

The investigators propose to enlarge the investigation of anti-BKv immune cellular response to other functions than IFNg synthesis in the aim of detecting the eventual role of polyfunctional lymphocytes for infection control. Furthermore, the investigators propose to identify better diagnostic and prognostic makers.

Detailed Description

Not available

Recruitment & Eligibility

Status
COMPLETED
Sex
All
Target Recruitment
158
Inclusion Criteria
  • Kidney-transplanted patient, since less than 30 days
  • Older than 18 years old
  • Treatment with tacrolimus and mycophénolate mofetil
Exclusion Criteria
  • No informed consent
  • Pregnant women
  • Patient under legal guardianship
  • Treated by ciclosporin or mTOR-inhibitor

Study & Design

Study Type
OBSERVATIONAL
Study Design
Not specified
Arm && Interventions
GroupInterventionDescription
kidney-transplanted patientsbiological parameters-
Primary Outcome Measures
NameTimeMethod
Comparison of the level of response between patient with a "non-controlled infection" and patients for who the blood viral load is under 103 copBKv/ml.at 1month, 2months, 3months, 4, 5, 6, 7, 8 and 9 months after kidney transplantation

Analyse the role of T-lymphocytes response in the control of BKv infection

Secondary Outcome Measures
NameTimeMethod
Measurement of histological consequences of a BKv non-controlled infection during the first year post-graftat 12 months after kidney transplantation

Determine the frequence of occurence of nephropathy due to BKv for the population with a non-controlled infection

Analyse of the other causes that could influence the occurence of a viremia higher than 103 copBKv/mlat 1month, 2months, 3months, 4, 5, 6, 7, 8 and 9 months after kidney transplantation

Other causes for abnormal viremia

Measurement of increase of immune response due to modifications of immunosuppressive treatment for patient with a non-controlled infectionat 1, 3 et 6 months after modification of immunosuppressive treatment

Comparision between the level of immune response and the time when the first viremia is higher than 103 copBKv/ml and 1, 3 et 6 months after modification of immunosuppressive treatment.

Trial Locations

Locations (1)

Nantes' Univeristy hospital

🇫🇷

Nantes, France

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