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The Effect of Sedoanalgesia and General Anaesthesia on Early Neurological Recovery in Acute Ischaemic Stroke Patients Undergoing Endovascular Thrombectomy

Not Applicable
Active, not recruiting
Conditions
Anesthesia
Ischemic Stroke
Neurologic Symptoms
Interventions
Procedure: Sedoanalgesia
Procedure: general anesthesia
Registration Number
NCT06183567
Lead Sponsor
Umraniye Education and Research Hospital
Brief Summary

The hypothesis of this study is that sedoanalgesia will provide better early neurological recovery than general anaesthesia in acute ischaemic stroke patients undergoing endovascular thrombectomy and to investigate the haemodynamic data of both anaesthetic methods.

Detailed Description

Endovascular mechanical thrombectomy (EMT) is the standard emergency treatment for patients presenting with acute ischemic stroke in the anterior circulation due to urgent large vessel occlusion and suitable for interventional procedures. However, despite reperfusion of the ischemia-affected area, some patients do not recover clinically. The reason for this is not known exactly. It is known that age and baseline function, which are thought to indicate brain reserve, affect the long-term outcome of stroke. Chronic hypertension, diabetes mellitus, dyslipidemia and coronary artery disease, which are associated with low brain reserve, are quite common in acute ischemic stroke patients.

There is controversy as to whether general anesthesia (GA) or sedoanalgesia (SA) should be used during EMT for acute ischemic stroke. There are not enough randomized trials addressing this question. Benefits of GA include airway preservation, pain control and potentially improved radiographic imaging and patient immobility for intervention. Conversely, GA is time-consuming and possibly associated with longer time for groin puncture and revascularization. In addition, hypotension may occur during GA, which carries a greater risk of ischemic damage. Advantages of SA may include shorter time to revascularization, fewer hemodynamic problems and the possibility of better neurological assessment during the procedure. The main arguments against SA are that patient movement can lead to procedural complications, higher radiation dose, the need for more contrast media and lack of airway control. Simonsen et al. compared general anesthesia and conscious sedation in patients with acute ischemic stroke undergoing endovascular treatment (GOLIATH) and showed that the choice of different anesthesia method can affect infarct area growth, clinical outcomes, and important physiological and anesthetic parameters.

Again, in the SIESTA (Sedation vs Intubation for Endovascular Stroke Treatment) study comparing sedation and intubation in endovascular stroke treatment, no significant difference was shown between both groups when early neurological recovery was compared (24th hour NIHSS). In this study, no superiority of conscious sedation over general anesthesia was demonstrated.

In the ESCAPE and SWIFT study, general anesthesia and conscious sedation were compared and conscious sedation was associated with better outcome than general anesthesia.

Recruitment & Eligibility

Status
ACTIVE_NOT_RECRUITING
Sex
All
Target Recruitment
62
Inclusion Criteria
  1. Presence of cerebral ischemic embolism within the first 6 hours
  2. ASA 1-4
  3. Body mass index below 30 kg m-2
  4. NIHSS score ≥ 10
  5. Presence of isolated/combined occlusion at any level of the anterior circulation internal carotid or middle cerebral artery
Exclusion Criteria
  • Chronic renal failure
  • EF less than 40
  • Presence of intracranial hemorrhage
  • Previous known history of severe neurological disease
  • Presence of bleeding diathesis
  • Pre-procedure GCS ≤ 8 and intubated patients
  • Failure to clearly show the site of vascular occlusion on diagnostic imaging results
  • Clinical or imaging evidence of vascular occlusion that is not internal carotid artery or middle cerebral artery
  • Absence of gag reflex, loss of airway protective reflex, inadequate saliva swallowing
  • History of lung infection, advanced COPD or respiratory failure
  • Known history of aspiration due to vomiting,
  • Known history of difficult airway
  • In the presence of known intolerance or allergy to certain drugs for sedation, analgesia or both
  • Previously known carotid artery stenosis
  • Pregnant patients
  • Patients without consent

Study & Design

Study Type
INTERVENTIONAL
Study Design
PARALLEL
Arm && Interventions
GroupInterventionDescription
Sedoanalgesia (SA) groupSedoanalgesiaThe aim was to achieve a moderate level of sedoanalgesia (previously called conscious sedation) that would reduce agitation, anxiety and mobility but allow communication with the patient. Sedation was maintained at a level where cardiovascular function was preserved and no intervention was required to protect the airway in spontaneous breathing. Fentanyl iv 25-50 µg bolus was administered to each patient undergoing sedoanalgesia. For maintenance of sedoanalgesia, propofol iv infusion was started at doses of 1-2 mg kg-1 hour. It was titrated according to BIS level. In case of patient noncompliance, propofol iv 0.5 mg kg-1 was intervened.
Sedoanalgesia (SA) groupgeneral anesthesiaThe aim was to achieve a moderate level of sedoanalgesia (previously called conscious sedation) that would reduce agitation, anxiety and mobility but allow communication with the patient. Sedation was maintained at a level where cardiovascular function was preserved and no intervention was required to protect the airway in spontaneous breathing. Fentanyl iv 25-50 µg bolus was administered to each patient undergoing sedoanalgesia. For maintenance of sedoanalgesia, propofol iv infusion was started at doses of 1-2 mg kg-1 hour. It was titrated according to BIS level. In case of patient noncompliance, propofol iv 0.5 mg kg-1 was intervened.
General anesthesia (GA) groupgeneral anesthesiaAnesthesia induction was performed with lidocaine iv 0.5 mg kg-1, propofol iv 1-2 mg kg-1, fentanyl iv 25-50 µg. After providing adequate ventilation with a mask, rocuronium iv 0.45-0.6 mg kg-1 is administered and endotracheal intubation is performed. After intubation is confirmed with end-tidal CO2 monitoring, tidal volume is set to 6-8 ml kg-1 and respiratory frequency to 12/min in CMV mode. In order to maintain cerebral perfusion, PaCO2: 35-40 mmHg is aimed to be maintained. Sevoflurane MAC 0.8 and remifentanil infusion 0.03 µg kg-1 min iv were used for maintenance of anesthesia. At the end of the procedure, sugammadex iv 2mg kg-1 was administered for extubation.
General anesthesia (GA) groupSedoanalgesiaAnesthesia induction was performed with lidocaine iv 0.5 mg kg-1, propofol iv 1-2 mg kg-1, fentanyl iv 25-50 µg. After providing adequate ventilation with a mask, rocuronium iv 0.45-0.6 mg kg-1 is administered and endotracheal intubation is performed. After intubation is confirmed with end-tidal CO2 monitoring, tidal volume is set to 6-8 ml kg-1 and respiratory frequency to 12/min in CMV mode. In order to maintain cerebral perfusion, PaCO2: 35-40 mmHg is aimed to be maintained. Sevoflurane MAC 0.8 and remifentanil infusion 0.03 µg kg-1 min iv were used for maintenance of anesthesia. At the end of the procedure, sugammadex iv 2mg kg-1 was administered for extubation.
Primary Outcome Measures
NameTimeMethod
effects of both anesthesia management on hemodynamics during the procedureBefore the Endovascular Thrombectomy procedure and until the end of the recovery period (4 hours)

Mean arterial pressure, heart rate

scoring systems of neurological findingsBefore Endovascular Thrombectomy and after 48 hours

NIHSS (National Institutes of Health Stroke Scale), Glasgow coma scale (GCS) and FOUR (Full Outline of UnResponsiveness) scores

Secondary Outcome Measures
NameTimeMethod
mortality and morbidityhospitalization days

exitus,discharge to home or palliative unit

early neurological outcome findings48 hours

hemiparesis, hemiplegia, aphasia, facial paralysis

Trial Locations

Locations (1)

Umraniye Education and Research Hospital

🇹🇷

Istanbul, Umraniye, Turkey

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