Cerebral Oxymetry in Traumatic Brain Injury

• Conditions: Traumatic Brain Injury

• Registration Number: NCT01940861
• Lead Sponsor: Oslo University Hospital

Brief Summary

Vasopressor for current treatment protocols for acute traumatic brain injury can lead to vasoconstriction and thus cerebral hypoperfusion that can be detected with cerebral oxymetry as a drop in SCO2.

Detailed Description

Not available

Study Timeline

• Study First Submitted: 2013-09-09
• Study First Submitted QC: 2013-09-09
• Study First Posted: 2013-09-12
• Status Verified: 2014-11
• Study Start: 2014-11
• Last Update Submitted: 2014-11-21
• Last Update Posted: 2014-11-24
• Primary Completion: 2016-12
• Study Completion: 2016-12

Recruitment & Eligibility

• Status: UNKNOWN
• Sex: All
• Target Recruitment: 15

Inclusion Criteria

• Patients with acute traumatic severe brain injury requiring neuro intensive care.
• The patient admitted must be at least 18 years of age and under 60 years.
• Intoxication is not an obstacle for inclusion as we believe this will not affect the recording in relation to our problem.

Exclusion Criteria

• Traumatic injury in the frontal lobes corresponding to the area where ScO2 monitored.
• Patients in pharmacological studies.
• Patients with severe heart / lung / blood vessel diseases.

Study & Design

• Study Type: OBSERVATIONAL
• Study Design: Not specified

Primary Outcome Measures

Name	Time	Method
Dosage of vasopressor	24 hours

Trial Locations

Locations (1)

Oslo University Hospital; 🇳🇴 Oslo, Norway