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A Study to Learn How Safe and Tolerable Vonsetamig is in Adult Patients With Chronic Kidney Disease (CKD) Who Need Kidney Transplantation and Are Highly Sensitized to Human Leukocyte Antigen (HLA)

Phase 1
Recruiting
Conditions
Chronic Kidney Disease (CKD)
Interventions
Registration Number
NCT05092347
Lead Sponsor
Regeneron Pharmaceuticals
Brief Summary

The purpose of this study is to determine whether vonsetamig will safely decrease anti-HLA antibodies to allow for kidney transplantation.

Vonsetamig is being studied for treatment of patients in need of kidney transplantation who are highly sensitized to HLA.

The study is looking at several other research questions, including:

* Side effects that may be experienced from taking vonsetamig

* How vonsetamig works in the body

* How much vonsetamig is present in the blood

* If vonsetamig works to lower levels of antibodies to HLA

Detailed Description

Not available

Recruitment & Eligibility

Status
RECRUITING
Sex
All
Target Recruitment
56
Inclusion Criteria
  1. Has Chronic Kidney Disease (CKD) requiring hemodialysis, and awaiting kidney transplant on the United Network for Organ Sharing (UNOS), with a cPRA ≥99.9%, or those with a cPRA >98% (98.1% to 99.8%) who have spent 5 years or longer on the waitlist, as defined in the protocol
  2. Adequate hematologic and adequate hepatic function as defined in the protocol
  3. Willing and able to comply with clinic visits and study-related procedures

Key

Exclusion Criteria
  1. Current or active malignancy not in remission for at least 1 year
  2. Central nervous system (CNS) pathology or history of CNS neurodegenerative or movement disorders
  3. Patients who have had their spleen removed, including patients with functional asplenia
  4. Patients who have received a stem cell transplantation within 5 years
  5. Use of investigational agents within 8 weeks or 5 half-lives of study drug administration (whichever is larger)
  6. Total plasma IgG <300 mg/dL at screening
  7. Continuous systemic corticosteroid treatment with more than 10 mg per day of prednisone (or anti-inflammatory equivalent) within 72 hours of start of study drug administration
  8. Received a calcineurin inhibitor (eg, tacrolimus, cyclosporine) within 30 days of study drug administration
  9. Received cyclophosphamide, rituximab, obinutuzumab, other anti-CD20 or B cell-depleting agents, or proteasome inhibitors or anti-CD38 therapies (eg, isatuximab, daratumumab) within 12 months of study drug administration
  10. Prior treatment with any anti-BCMA antibody (including antibody drug conjugate or bsAb) or BCMA-directed CAR-T cell therapy, as described in the protocol
  11. Has received a COVID-19 vaccination, as described in the protocol

Note: Other protocol defined inclusion / exclusion criteria apply

Study & Design

Study Type
INTERVENTIONAL
Study Design
SEQUENTIAL
Arm && Interventions
GroupInterventionDescription
VonsetamigVonsetamig-
Primary Outcome Measures
NameTimeMethod
Incidence of adverse event(s) of interest (AEI) from the first dose through end of the safety observation periodUp to approximately 6 weeks
Incidence and severity of treatment-emergent adverse events (TEAE)s from the first study drug dose up to the end of the studyUp to 78 weeks

TEAEs include adverse events of special interest (AESI) and serious adverse events (SAEs)

Secondary Outcome Measures
NameTimeMethod
Percent change from baseline in the peak (immunodominant) MFIUp to 78 weeks
Time to first clinically meaningful reduction in anti-HLA alloantibody levels by SAB assayUp to 78 weeks

Defined as peak anti-HLA alloantibody MFI \<5,000 or ≥50% reduction

Duration of maximal reduction in anti-HLA alloantibody MFI by SAB assayUp to 78 weeks
Time to maximal reduction in cPRA from baselineUp to 78 weeks
Percent change from baseline in the sum of MFI of anti-HLA alloantibodies using the SAB assayUp to 78 weeks
Maximum reduction in cPRA from baselineUp to 78 weeks
Duration of a reduction in peak anti-HLA alloantibody to MFI <5,000 or by ≥50% by SAB assayUp to 78 weeks
Duration of maximal reduction in cPRA by SAB assayUp to 78 weeks
Serum concentration of Immunoglobulin (Ig) classes over timeUp to 78 weeks
Percent change from baseline of serum concentration of Ig classesUp to 78 weeks
Concentration of vonsetamig in serum over timeUp to 78 weeks
Incidence of treatment-emergent anti-drug antibodies (ADAs) to vonsetamig over timeUp to 78 weeks
Proportion of Participants with a clinically meaningful reduction in anti-HLA alloantibodiesUp to 78 weeks

Clinically meaningful reduction in anti-HLA alloantibodies are defined as either:

* Reduction in Calculated panel-reactive antibody (cPRA) from baseline, or

* Reduction in the peak (immunodominant) anti-HLA mean fluorescence intensity (MFI) to \<5,000, or by ≥50% by Single antigen bead (SAB) assay

Maximum reduction in the peak (immunodominant) MFI of anti-HLA alloantibodies from baselineUp to 78 weeks
Time to maximal reduction in anti-HLA alloantibody levels by SAB assayUp to 78 weeks

Defined as peak anti-HLA alloantibody MFI \<5,000 or ≥50% reduction

Time to first clinically meaningful reduction in cPRAUp to 78 weeks

Trial Locations

Locations (10)

Cedars-Sinai Medical Center

🇺🇸

Los Angeles, California, United States

University of California Irvine

🇺🇸

Orange, California, United States

Connie Frank Transplant Center at UCSF

🇺🇸

San Francisco, California, United States

Yale University of Medicine

🇺🇸

New Haven, Connecticut, United States

Medstar Georgetown Transplant Institute - 2-PHC

🇺🇸

Washington, District of Columbia, United States

Comprehensive Transplant Center

🇺🇸

Chicago, Illinois, United States

John Hopkins Hospital

🇺🇸

Baltimore, Maryland, United States

University of Minnesota

🇺🇸

Minneapolis, Minnesota, United States

New York University Langone Health

🇺🇸

New York, New York, United States

Penn Transplant Institute

🇺🇸

Philadelphia, Pennsylvania, United States

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