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Prevalence in Iron deficiency Acute Heart Faliure

Recruiting
Conditions
heart failure, iron deficiency
Registration Number
NL-OMON28189
Lead Sponsor
Maastricht University Medical Centre ( MUMC)Prof Dr Brunner
Brief Summary

Not available

Detailed Description

Not available

Recruitment & Eligibility

Status
Recruiting
Sex
Not specified
Target Recruitment
1000
Inclusion Criteria

Age> 18 years

Admission with an episode of acute heart failure

Exclusion Criteria

-History of erythropoietin stimulating agent, IV iron therapy, and/or blood transfusion within 3 months prior to hospitalisation.

-Oral iron therapy at any doses in 4 weeks prior to hospitalization or iron containing multivitamins irrespectively of the dose of iron.

Study & Design

Study Type
Observational non invasive
Study Design
Not specified
Primary Outcome Measures
NameTimeMethod
This study aims to analyse the prevalence of iron deficiency (ID) in patients with an episode of acute heart failure at different time points (t0 = within 24(+12h) hours of admission due to acute decompensated heart failure, t1 = after stabilisation and within 0-2 days prior to discharge, t2 = 6 weeks after discharge (+- 2 weeks, but never earlier than 4 weeks after discharge).<br /><br><br /><br>Iron deficiency is defined as serum ferritin <100µg/l or serum ferritin =100-299µg/l and transferrin saturation <20%.<br>
Secondary Outcome Measures
NameTimeMethod
To determine at what time point screening for ID is reliable after a patient is hospitalized due to an acute episode (guidance for cardiologists).<br /><br>To analyse the prevalence of ID in subgroups (female, LVEF, NYHA, co-morbidities (hypertension, diabetes mellitus, COPD/asthma, CVA/TIA, cancer, anaemia, systemic inflammatory disorders, valve disease, atrial fibrillation), eGFR and CRP and anemia).<br /><br><br /><br>For possible later analyses, left-over of already drawn serum may be stored (at discretion of the investigators, until further notice in MUMC Maastricht, Zuyderland Hospital Heerlen and Amphia hospital Breda only). Such analyses might include measurement of hepcidine or soluble transferrin-receptor, among other biomarkers, to better define the role of iron deficiency in the acute setting of HF. This may also give insight if measurements of ferritin provide reliable results or if definition of ID should be different in the acute setting.<br>
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