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Combined Aerobic Exercise and Cognitive Training in Seniors at Increased Risk for Alzheimer's Disease

Not Applicable
Not yet recruiting
Conditions
Alzheimer Disease
Interventions
Behavioral: Combined aerobic exercise and cognitive training program
Registration Number
NCT05163626
Lead Sponsor
Xuanwu Hospital, Beijing
Brief Summary

The study aims to investigate the effect of a long-term combined aerobic exercise and cognitive training program on cognitive function and blood exosomal synaptic protein levels in seniors at increased risk for Alzheimer's Disease.

Detailed Description

Alzheimer's disease (AD) is the most common cause of dementia in people older than 65 years worldwide. The neuropathological changes of AD occur decades before the onset of cognitive impairment, suggesting that early identification and timely intervention may postpone the clinical progress. In addition to its characteristic amyloid β and tau pathology, AD is also marked by synaptic dysfunction. Abnormal synaptic protein levels, such as growth associated protein 43 (GAP43), neurogranin, synaptotagmins, and synaptosome associated protein 25 (SNAP25) have been observed in the brain tissue and cerebrospinal fluid (CSF). Blood neuro-exosomal synaptic proteins have emerged as promising predictors for AD and cognitive decline. Particularly, the investigators previously reported a combination of blood neuro-exosomal protein (GAP43, neurogranin, SNAP25, and synaptotagmin 1) can predict AD 5 to 7 years before the clinical onset.

Both physical exercise and cognitive training have been demonstrated to improve cognitive function in AD and to exert a protective effect against developing dementia in the normal aging population. Furthermore, cognitive stimulation is an established modulator of synaptic plasticity and physical exercise might regulate synapse functional and structural change. However, whether cognitive training and physical exercise can alter exosomal synaptic protein levels and the relationship of biomarker changes to cognitive function in those seniors at increased risk for AD remain unclear.

In this study, the investigators aim to

1. assess the effects of a long-term combined aerobic exercise and cognitive training program on cognitive function and the predictive biomarkers (blood neuro-exosomal synaptic proteins: GAP43, neurogranin, SNAP25, and synaptotagmin 1) in seniors at increased risk of AD with abnormally decreased levels of the biomarkers.

2. determine the relationship of biomarker changes with cognitive function in these people.

3. confirm the predictive value of the blood neuro-exosomal synaptic proteins for AD in a longitudinal setting.

Recruitment & Eligibility

Status
NOT_YET_RECRUITING
Sex
All
Target Recruitment
200
Inclusion Criteria
  • Mandarin-speaking subjects.
  • Not clinically demented.
  • Meeting the cutoff values of MMSE and CDR.
  • With low levels of blood neuro-exosomal synaptic proteins (GAP43<1983pg/ml, synaptotagmin 1<431pg/ml, neurogranin<1433pg/ml, SNAP25<448pg/ml)

Exclusion criteria:

  • Had major neurologic diagnosis (e.g., Alzheimer's disease, Parkinson's disease, stroke, encephalitis, and epilepsy) or other condition that might impair cognition or confound assessments.
  • Had a history of psychotic episodes or had major depression (Hamilton Depression Rating Scale score > 24 points).
  • Had severe systemic diseases, such as tumors, cardiovascular or orthopedic disorders that can affect the ability to perform the proposed intervention tasks.
Exclusion Criteria

Not provided

Study & Design

Study Type
INTERVENTIONAL
Study Design
PARALLEL
Arm && Interventions
GroupInterventionDescription
Combined aerobic exercise and cognitive training programCombined aerobic exercise and cognitive training program-
Primary Outcome Measures
NameTimeMethod
Change in cognitive function over time as assessed by Clinical Dementia Rating (CDR)baseline time, year 1, year 3, year 5, year 7

CDR will be performed to evaluate the cognition of participants at the enrollment and year 1, year 3, year 5, year 7. The score ranges from 0 to 18, with higher values indicating worse cognition.

Change in cognitive function over time as assessed by Trail-Making Test Parts A and B (TMT-A and TMT-B)baseline time, year 1, year 3, year 5, year 7

TMT-A and TMT-B will be performed to evaluate the executive function of participants at the enrollment and year 1, year 3, year 5, year 7. Scoring is based on time taken to complete the test (e.g., 35 seconds yielding a score of 35), with lower scores indicating better cognition.

Change in cognitive function over time as assessed by the Rey-Osterrieth Complex Figure Test (ROCF)baseline time, year 1, year 3, year 5, year 7

ROCF will be performed to evaluate the visuospatial function and other cognition domains of participants at the enrollment and year 1, year 3, year 5, year 7. Participants are asked to produce a complicated line drawing, with higher scores indicating better cognition.

Change in cognitive function over time as assessed by Boston Naming Test (BNT)baseline time, year 1, year 3, year 5, year 7

BNT will be performed to evaluate the language function of participants at the enrollment and year 1, year 3, year 5, year 7. The score ranges from 0 to 30, with higher values indicating better cognition.

Change in cognitive function over time as assessed by California Verbal Learning Test (CVLT)baseline time, year 1, year 3, year 5, year 7

CVLT will be performed to evaluate the memory function of participants at the enrollment and year 1, year 3, year 5, year 7. Participants are asked to finish immediate recall, delayed recall, and delayed recognition tasks in the test, with higher scores indicating better cognition.

Change in cognitive function over time as assessed by the Montreal Cognitive Assessment (MoCA)baseline time, year 1, year 3, year 5, year 7

MoCA will be performed to evaluate the cognition of participants at the enrollment and year 1, year 3, year 5, year 7. The score ranges from 0 to 30, with higher values indicating better cognition.

Change in cognitive function over time as assessed by Verbal Fluency Testbaseline time, year 1, year 3, year 5, year 7

Verbal Fluency Test will be performed to evaluate the semantic memory function of participants at the enrollment and year 1, year 3, year 5, year 7. Participants are asked to produce as many animals as possible within 1 minute. The score is the number of animals, with higher scores indicating better cognition.

Change in cognitive function over time as assessed by Mini Mental State Examination (MMSE)baseline time, year 1, year 3, year 5, year 7

MMSE will be performed to evaluate the cognition of participants at the enrollment and year 1, year 3, year 5, year 7. The score ranges from 0 to 30, with higher values indicating better cognition.

Change in cognitive function over time as assessed by Digit Span Test-Forward and Backwardbaseline time, year 1, year 3, year 5, year 7

Digit Span Tests will be performed to evaluate the working memory of participants at the enrollment and year 1, year 3, year 5, year 7. The total scores are twelve for each test, with higher values indicating better cognition.

Secondary Outcome Measures
NameTimeMethod
Changes in concentrations of blood neuro-exosomal synaptotagmin1 over timebaseline time, year 1, year 3, year 5, year 7

Concentrations of blood neuro-exosomal synaptotagmin1 will be evaluated at the enrollment and year 1, year 3, year 5, year 7, and measured in pg/ml.

Changes in concentrations of blood neuro-exosomal GAP43 over timebaseline time, year 1, year 3, year 5, year 7

Concentrations of blood neuro-exosomal GAP43 will be evaluated at the enrollment and year 1, year 3, year 5, year 7, and measured in pg/ml.

The area under curve of the blood neuro-exosomal synaptic proteins (GAP43, neurogranin, SNAP25, and synaptotagmin1) for the accurate diagnosis of ADup to 7 years

The area under curve is used to show the ability of the blood neuro-exosomal synaptic proteins (GAP43, neurogranin, SNAP25, and synaptotagmin1) to diagnose AD. The value of area under curve is higher, then the ability of the blood neuro-exosomal synaptic proteins to diagnose AD is stronger.

Changes in concentrations of blood neuro-exosomal neurogranin over timebaseline time, year 1, year 3, year 5, year 7

Concentrations of blood neuro-exosomal neurogranin will be evaluated at the enrollment and year 1, year 3, year 5, year 7, and measured in pg/ml.

Changes in concentrations of blood neuro-exosomal SNAP25 over timebaseline time, year 1, year 3, year 5, year 7

Concentrations of blood neuro-exosomal SNAP25 1 will be evaluated at the enrollment and year 1, year 3, year 5, year 7, and measured in pg/ml.

Trial Locations

Locations (1)

Xuanwu Hospital

🇨🇳

Beijing, China

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