Effect of Spironolactone on Treatment of Central Serous Chorioretinopathy
- Conditions
- Condition 1: Central Serous Chorioretinopathy. Condition 2: Central Serous Chorioretinopathy.Separation of Retinal LayersSerous retinal detachment
- Registration Number
- IRCT2017101536801N1
- Lead Sponsor
- Zahedan University of Medical Sciences
- Brief Summary
Not available
- Detailed Description
Not available
Recruitment & Eligibility
- Status
- Recruiting
- Sex
- All
- Target Recruitment
- 30
All patients with central serous chorioretinopathy, whose condition has just been diagnosed and who have not received any treatment.
Exclusion criteria: Patients whohave been received any treatment for central serous chorioretinopathy, including intravitreal injection of bevasizumab, laser therapy, photodynamic therapy, and drug treatment; Failure to refer patients for follow-up treatment; Drug-related complications requiring drug discontinuation; Disagreement with the proposed treatment
Not provided
Study & Design
- Study Type
- interventional
- Study Design
- Not specified
- Primary Outcome Measures
Name Time Method Macular Central Thickness. Timepoint: At the beginning - One month later - Three months later - Six months later. Method of measurement: Based on OCT, Measured as the Thickness of the Macular Center.;Choroidal Vasculai Index. Timepoint: At the beginning - One month later - Three months later - Six months later. Method of measurement: The ratio of the luminal area to the total choroid tissue in the subfovea area using the image J software.;Visual Acuity. Timepoint: At the beginning - One month later - Three months later - Six months later. Method of measurement: Snellen Chart.;Recovery. Timepoint: Six months later. Method of measurement: Increase in visual acuity or a decrease in the thickness of the macula to less than 252 micrometers, or a rise in the choroid vascular index.
- Secondary Outcome Measures
Name Time Method Recurrence. Timepoint: At 1 year. Method of measurement: Based on OCT measurements, which, with the increase in the thickness of the macular center, precipitates as fluid re-accumulation in the sub-retinal substrate after the fluid has completely disappeared.